Research Areas
Links
Biography and Research Information
OverviewAI-generated summary
Ravi W. Sun's research focuses on the cellular and molecular mechanisms underlying vascular malformations. His work investigates endothelial cell proliferation, dysregulated signaling pathways, and the role of specific molecules in the development of conditions such as arteriovenous malformations (AVMs) and lymphatic malformations.
Sun has published research examining the hyperproliferative nature of venous malformation vessels and the involvement of TGF-β/BMP signaling in extracranial AVMs. His investigations also include the impact of microRNAs, such as microRNA-21, on lymphatic malformation endothelial cell proliferation by targeting the PDCD4 gene. Further studies explore the initiation of endothelial-to-mesenchymal transition by mural cells in AVMs and the potential of inhibiting angiopoietin-2 to control lymphatic malformation cell growth.
His scholarly output includes 16 publications with 121 citations and an h-index of 7. Sun collaborates with several researchers at the University of Arkansas for Medical Sciences, including Graham M. Strub, Gresham T. Richter, Haihong Zhang, and Syed J. Mehdi.
Metrics
- h-index: 7
- Publications: 16
- Citations: 126
Selected Publications
-
Rescue of Angiopoietin‐2 Inhibits Proliferation of Lymphatic Malformation Endothelial Cells (2025)
-
Mural Cells Initiate Endothelial-to-Mesenchymal Transition in Adjacent Endothelial Cells in Extracranial AVMs (2024)
-
Upregulated MicroRNA-21 Drives the Proliferation of Lymphatic Malformation Endothelial Cells by Inhibiting PDCD4 (2023)
-
Extracranial arteriovenous malformations demonstrate dysregulated TGF-β/BMP signaling and increased circulating TGF-β1 (2022)
-
Bronchogenic cyst of the posterior neck mimicking lymphatic malformation (2022)
-
Venous malformation vessels are improperly specified and hyperproliferative (2021)
Collaboration Network
Top Collaborators
- Extracranial arteriovenous malformations demonstrate dysregulated TGF-β/BMP signaling and increased circulating TGF-β1
- Upregulated MicroRNA-21 Drives the Proliferation of Lymphatic Malformation Endothelial Cells by Inhibiting PDCD4
- Bronchogenic cyst of the posterior neck mimicking lymphatic malformation
- Mural Cells Initiate Endothelial-to-Mesenchymal Transition in Adjacent Endothelial Cells in Extracranial AVMs
- Bronchogenic cyst of the posterior neck mimicking lymphatic malformation
Showing 5 of 6 shared publications
- Venous malformation vessels are improperly specified and hyperproliferative
- Extracranial arteriovenous malformations demonstrate dysregulated TGF-β/BMP signaling and increased circulating TGF-β1
- Upregulated MicroRNA-21 Drives the Proliferation of Lymphatic Malformation Endothelial Cells by Inhibiting PDCD4
- Mural Cells Initiate Endothelial-to-Mesenchymal Transition in Adjacent Endothelial Cells in Extracranial AVMs
- Rescue of Angiopoietin‐2 Inhibits Proliferation of Lymphatic Malformation Endothelial Cells
- Extracranial arteriovenous malformations demonstrate dysregulated TGF-β/BMP signaling and increased circulating TGF-β1
- Upregulated MicroRNA-21 Drives the Proliferation of Lymphatic Malformation Endothelial Cells by Inhibiting PDCD4
- Mural Cells Initiate Endothelial-to-Mesenchymal Transition in Adjacent Endothelial Cells in Extracranial AVMs
- Rescue of Angiopoietin‐2 Inhibits Proliferation of Lymphatic Malformation Endothelial Cells
- Venous malformation vessels are improperly specified and hyperproliferative
- Upregulated MicroRNA-21 Drives the Proliferation of Lymphatic Malformation Endothelial Cells by Inhibiting PDCD4
- Rescue of Angiopoietin‐2 Inhibits Proliferation of Lymphatic Malformation Endothelial Cells
- Venous malformation vessels are improperly specified and hyperproliferative
- Upregulated MicroRNA-21 Drives the Proliferation of Lymphatic Malformation Endothelial Cells by Inhibiting PDCD4
- Rescue of Angiopoietin‐2 Inhibits Proliferation of Lymphatic Malformation Endothelial Cells
- Upregulated MicroRNA-21 Drives the Proliferation of Lymphatic Malformation Endothelial Cells by Inhibiting PDCD4
- Mural Cells Initiate Endothelial-to-Mesenchymal Transition in Adjacent Endothelial Cells in Extracranial AVMs
- Rescue of Angiopoietin‐2 Inhibits Proliferation of Lymphatic Malformation Endothelial Cells
- Bronchogenic cyst of the posterior neck mimicking lymphatic malformation
- Bronchogenic cyst of the posterior neck mimicking lymphatic malformation
- Bronchogenic cyst of the posterior neck mimicking lymphatic malformation
- Bronchogenic cyst of the posterior neck mimicking lymphatic malformation
- Bronchogenic cyst of the posterior neck mimicking lymphatic malformation
- Bronchogenic cyst of the posterior neck mimicking lymphatic malformation
- Bronchogenic cyst of the posterior neck mimicking lymphatic malformation
- Bronchogenic cyst of the posterior neck mimicking lymphatic malformation
- Upregulated MicroRNA-21 Drives the Proliferation of Lymphatic Malformation Endothelial Cells by Inhibiting PDCD4
- Rescue of Angiopoietin‐2 Inhibits Proliferation of Lymphatic Malformation Endothelial Cells
- Upregulated MicroRNA-21 Drives the Proliferation of Lymphatic Malformation Endothelial Cells by Inhibiting PDCD4
- Rescue of Angiopoietin‐2 Inhibits Proliferation of Lymphatic Malformation Endothelial Cells
- Upregulated MicroRNA-21 Drives the Proliferation of Lymphatic Malformation Endothelial Cells by Inhibiting PDCD4
- Rescue of Angiopoietin‐2 Inhibits Proliferation of Lymphatic Malformation Endothelial Cells
- Venous malformation vessels are improperly specified and hyperproliferative
- Venous malformation vessels are improperly specified and hyperproliferative
Similar Researchers
Based on overlapping research topics