Preeti Nagar
Post Doctoral Fellow
Also affiliated: South Asian University (2017–2026)
Biochemistry & Molecular Biology, College of Medicine
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Preeti Nagar's research investigates molecular mechanisms underlying plant stress tolerance and human disease. Her work on plants focuses on the role of specific genes and proteins in responding to abiotic stresses such as drought and salinity. Publications include studies on phytosulfokine receptors in rice enhancing drought tolerance and the involvement of glyoxalase I and D-lactate dehydrogenase in methylglyoxal detoxification and stress tolerance in plants. She has also explored the potential of aquaporins in inducing stress tolerance.
In parallel, Nagar's research extends to human health, with a publication examining the role of nonsense-mediated mRNA decay in tumorigenesis. Her scholarship metrics include an h-index of 11 with 28 publications and 484 citations. She has collaborated with researchers Mohammad Alinoor Rahman and Md. Rafikul Islam at the University of Arkansas for Medical Sciences on multiple projects.
Metrics
- h-index: 11
- Publications: 27
- Citations: 485
Positions
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Post Doctoral Fellow publications 2023–2026University of Arkansas for Medical Sciences Biochemistry & Molecular Biology, College of Medicine Institutional directory
Selected Publications
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Targeting EZH2 oncogenic splicing: decoding the regulatory network and antisense correction (2026)
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CRISPR-mediated Correction of Oncogenic AS-NMD in Splicing Factor Mutant Cancer (2026)
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Mitochondrial antioxidant enzyme (SOD2) at the crossroads of redox signaling and cancer progression (2026)
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Genetics behind aflatoxin biosynthesis and tolerance in crops (2026)
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Targeting EZH2 Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction (2026)
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SRSF6 and SRSF1 coordinately enhance the inclusion of human MUSK exon 10 to generate a Wnt-sensitive MuSK isoform (2025)
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Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing (2024)
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RNA Splicing in Cancer and Targeted Therapies (2023)
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Nonsense-Mediated mRNA Decay as a Mediator of Tumorigenesis (2023)
Grants & Funding
As listed on this researcher's institutional profile.
- Center for Molecular Interactions in Cancer (CMIC) NIH Co-Investigator
Collaboration Network
Top Collaborators
- Nonsense-Mediated mRNA Decay as a Mediator of Tumorigenesis
- SRSF6 and SRSF1 coordinately enhance the inclusion of human MUSK exon 10 to generate a Wnt-sensitive MuSK isoform
- Mitochondrial antioxidant enzyme (SOD2) at the crossroads of redox signaling and cancer progression
- RNA Splicing in Cancer and Targeted Therapies
- Targeting EZH2 Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
Showing 5 of 6 shared publications
- Nonsense-Mediated mRNA Decay as a Mediator of Tumorigenesis
- SRSF6 and SRSF1 coordinately enhance the inclusion of human MUSK exon 10 to generate a Wnt-sensitive MuSK isoform
- RNA Splicing in Cancer and Targeted Therapies
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- SRSF6 and SRSF1 coordinately enhance the inclusion of human MUSK exon 10 to generate a Wnt-sensitive MuSK isoform
- Targeting EZH2 Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- CRISPR-mediated Correction of Oncogenic AS-NMD in Splicing Factor Mutant Cancer
- Targeting EZH2 oncogenic splicing: decoding the regulatory network and antisense correction
- Targeting EZH2 Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- CRISPR-mediated Correction of Oncogenic AS-NMD in Splicing Factor Mutant Cancer
- Targeting EZH2 oncogenic splicing: decoding the regulatory network and antisense correction
- SRSF6 and SRSF1 coordinately enhance the inclusion of human MUSK exon 10 to generate a Wnt-sensitive MuSK isoform
- Targeting EZH2 Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- Targeting EZH2 Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- Targeting EZH2 oncogenic splicing: decoding the regulatory network and antisense correction
- Targeting EZH2 Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- Targeting EZH2 oncogenic splicing: decoding the regulatory network and antisense correction
- Targeting EZH2 Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- Targeting EZH2 oncogenic splicing: decoding the regulatory network and antisense correction
- Targeting EZH2 Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- Targeting EZH2 oncogenic splicing: decoding the regulatory network and antisense correction
- CRISPR-mediated Correction of Oncogenic AS-NMD in Splicing Factor Mutant Cancer
- Targeting EZH2 oncogenic splicing: decoding the regulatory network and antisense correction
- CRISPR-mediated Correction of Oncogenic AS-NMD in Splicing Factor Mutant Cancer
- Targeting EZH2 oncogenic splicing: decoding the regulatory network and antisense correction
- RNA Splicing in Cancer and Targeted Therapies
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
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