Preeti Nagar
Post Doctoral Fellow
Also affiliated: Winthrop Rockefeller Foundation (2023–2026); South Asian University (2017–2025)
Postdoc Researcher
Biochemistry & Molecular Biology, College of Medicine
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Preeti Nagar's research investigates molecular mechanisms underlying plant stress responses and their applications in agriculture, as well as the role of molecular pathways in human cancer development. Her work on plants includes studying the function of phytosulfokine receptors in enhancing abscisic acid response and drought stress tolerance in rice, and their role in leaf senescence and reactive oxygen species signaling in *Arabidopsis thaliana*. Nagar has also examined transcript profiling of Polycomb gene families in *Oryza sativa* to understand abiotic stress responses. In the realm of human health, her research focuses on the involvement of nonsense-mediated mRNA decay in tumorigenesis and the impact of the NONO protein on breast cancer cell pathways. She has also explored RNA splicing in cancer and its potential for targeted therapies, including the role of SRSF6 and SRSF1 in human gene regulation relevant to cancer.
Metrics
- h-index: 11
- Publications: 24
- Citations: 473
Selected Publications
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Targeting <i>EZH2</i> oncogenic splicing: decoding the regulatory network and antisense correction (2026)
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CRISPR-mediated Correction of Oncogenic AS-NMD in Splicing Factor Mutant Cancer (2026)
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Mitochondrial antioxidant enzyme (SOD2) at the crossroads of redox signaling and cancer progression (2026)
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Genetics behind aflatoxin biosynthesis and tolerance in crops (2026)
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Targeting <i>EZH2</i> Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction (2026)
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SRSF6 and SRSF1 coordinately enhance the inclusion of human <i>MUSK</i> exon 10 to generate a Wnt-sensitive MuSK isoform (2025)
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Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing (2024)
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RNA Splicing in Cancer and Targeted Therapies (2023)
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Nonsense-Mediated mRNA Decay as a Mediator of Tumorigenesis (2023)
Grants & Funding
As listed on this researcher's institutional profile.
- Center for Molecular Interactions in Cancer (CMIC) NIH Co-Investigator
Collaboration Network
Top Collaborators
- Nonsense-Mediated mRNA Decay as a Mediator of Tumorigenesis
- SRSF6 and SRSF1 coordinately enhance the inclusion of human <i>MUSK</i> exon 10 to generate a Wnt-sensitive MuSK isoform
- RNA Splicing in Cancer and Targeted Therapies
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Targeting <i>EZH2</i> Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
Showing 5 of 6 shared publications
- Nonsense-Mediated mRNA Decay as a Mediator of Tumorigenesis
- SRSF6 and SRSF1 coordinately enhance the inclusion of human <i>MUSK</i> exon 10 to generate a Wnt-sensitive MuSK isoform
- RNA Splicing in Cancer and Targeted Therapies
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- SRSF6 and SRSF1 coordinately enhance the inclusion of human <i>MUSK</i> exon 10 to generate a Wnt-sensitive MuSK isoform
- Targeting <i>EZH2</i> Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- SRSF6 and SRSF1 coordinately enhance the inclusion of human <i>MUSK</i> exon 10 to generate a Wnt-sensitive MuSK isoform
- Targeting <i>EZH2</i> Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- RNA Splicing in Cancer and Targeted Therapies
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
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