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Presence Current · Arkansas
Last published 2026
Sources OpenAlex · ORCID
Refreshed 2026-08-15

Rami Ahmad Shahror

Researcher

Also affiliated: University of Maryland, Baltimore (2020–2023); National Health Research Institutes (2019–2020); Örebro University (2015–2018); University of Arkansas Medical Center (2025); Taipei Medical University (2019–2020)

Faculty Researcher

Pharmacology & Toxicology, College of Medicine

10 h-index 25 pubs 456 cited

  • Animals
  • Mice, Inbred C57BL
  • Mice
  • Mesenchymal Stem Cell Transplantation
  • Brain Injuries, Traumatic
  • Humans
  • Retinal Diseases
  • Reperfusion Injury
  • Mesenchymal Stem Cells
  • Retina
  • Histone Deacetylases
  • Magnetite Nanoparticles
  • Brain
  • Histone Deacetylase 3
  • Fibroblast Growth Factors

Biography and Research Information

OverviewAI-generated summary

Rami Ahmad Shahror's research focuses on the role of myeloid cells and innate immune responses in central nervous system and ocular diseases. His work investigates mechanisms underlying neuroinflammation and its contribution to conditions such as traumatic brain injury and ischemic retinopathies. Shahror's publications explore the modulation of inflammatory pathways, including the arginase 1/ornithine decarboxylase pathway and the role of HDAC3 in myeloid cell function, to ameliorate injury in retinal ischemic models. He also examines the potential of nanosystems for ocular drug delivery and employs techniques like multi-color flow cytometry to characterize myeloid cell populations in mouse retina research. Shahror has 25 publications with 424 citations and an h-index of 10. He collaborates with researchers at the University of Arkansas for Medical Sciences, including Abdelrahman Y. Fouda, Carol Morris, Esraa Shosha, and Melissa Wild.

Metrics

  • h-index: 10
  • Publications: 25
  • Citations: 456

Selected Publications

  • CD5L promotes efferocytosis and resolution of retinal ischemic injury (2026)
    Cell Death and Disease DOI OpenAlex
  • Myeloid HDAC3 deletion protects against traumatic optic injury (2026)
    Cell Death Discovery DOI OpenAlex
  • Recent advances and future challenges in nanosystems for ocular drug delivery (2025)
    Journal of Pharmacology and Experimental Therapeutics 7 citations DOI OpenAlex
  • Myeloid HDAC3 Deletion Protects Against Traumatic Optic Injury (2025)
    Research Square DOI OpenAlex
  • Disrupting the CD47/SIRPα Axis Protects Against Traumatic Optic Neuropathy (2025)
    Research Square DOI OpenAlex
  • Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology (2024)
    Translational Vision Science & Technology 2 citations DOI OpenAlex
  • Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury (2024)
    Journal of Neuroinflammation 16 citations DOI OpenAlex
  • Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions (2024)
    Journal of Neuroinflammation 43 citations DOI OpenAlex
  • The arginase 1/ornithine decarboxylase pathway suppresses HDAC3 to ameliorate the myeloid cell inflammatory response: implications for retinal ischemic injury (2023)
    Cell Death and Disease 27 citations DOI OpenAlex
  • Multi-color Flow Cytometry Protocol to Characterize Myeloid Cells in Mouse Retina Research (2023)
    BIO-PROTOCOL 6 citations DOI OpenAlex

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Collaboration Network

22 Collaborators 6 Institutions 2 Countries

Top Collaborators

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