Desislava A. Marinkova
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Also affiliated: University of Kentucky (2022–2023)
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Desislava A. Marinkova studies the role of TRPA1 cation channels in regulating cochlear sensitivity following acoustic trauma. Her research investigates the involvement of these channels in non-sensory supporting cells within the cochlea. Marinkova's work also includes investigations into the relationship between obesity, histone acetylation, and insulin resistance. She has published four papers and has a total of 19 citations, with an h-index of 1. Her collaborators include Rushita A. Bagchi and Ashim K. Bagchi from the University of Arkansas for Medical Sciences, and Chris Presley from Hendrix College.
Metrics
- h-index: 1
- Publications: 4
- Citations: 19
Selected Publications
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Obesity, histone acetylation, and insulin resistance (2024)
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TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma (2023)
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TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma (2022)
Collaboration Network
Top Collaborators
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- Obesity, histone acetylation, and insulin resistance
- Obesity, histone acetylation, and insulin resistance
- Obesity, histone acetylation, and insulin resistance
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