Match tier
Listed
Presence
Current · Arkansas
Last published
2025
Sources
OpenAlex · ORCID
Refreshed
2026-10-07
D
Debopam Ghosh
Role not yet determined Is this you? Add your title
Also affiliated: Indian Institute of Chemical Biology (2012); Institute of Post Graduate Medical Education and Research (2012); Stanford Medicine (2019–2022); Stanford University (2019–2022)
10 h-index
28 pubs
558 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Debopam Ghosh's research investigates the intricate mechanisms of host-pathogen interactions, particularly focusing on immune responses to parasitic infections like Leishmania donovani. His work has explored how factors such as hyperlipidemia and membrane cholesterol influence host susceptibility and protection against Leishmania infection, with a publication dating back to 1997 on lipid peroxidation in visceral leishmaniasis. Ghosh also studies the role of specific immune cells and signaling pathways in host defense, examining B cells as antigen-presenting cells and the regulation of T cell differentiation by molecules like ICOS in response to infections such as Plasmodium chabaudi chabaudi AS. His research extends to understanding immune suppression mediated by tumor-associated macrophages and investigating hypersensitivity reactions to therapeutic inhibitors, linking them to specific HLA alleles. Ghosh's scholarship includes 39 publications and an h-index of 11, with collaborations at the University of Arkansas for Medical Sciences involving Jason S. Stumhofer, J. Craig Forrest, Mark Manzano, and Shana M. Owens.
Metrics
- h-index: 10
- Publications: 28
- Citations: 558
Positions
-
Postdoctoral Researcher 2017–2022Stanford University Pediatrics ORCID
Selected Publications
-
Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment (2025)
-
p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated IgH/c-Myc Translocations (2020)
-
IL-17 Promotes Differentiation of Splenic LSK− Lymphoid Progenitors into B Cells following Plasmodium yoelii Infection (2017)
-
An Atypical Splenic B Cell Progenitor Population Supports Antibody Production during Plasmodium Infection in Mice (2016)
-
The Costimulatory Molecule ICOS Regulates Host Th1 and Follicular Th Cell Differentiation in Response to Plasmodium chabaudi chabaudi AS Infection (2015)
-
Signaling Circuits and Regulation of Immune Suppression by Ovarian Tumor-Associated Macrophages (2015)
-
Do You See What I See: Recognition of Protozoan Parasites by Toll-Like Receptors (2014)
Collaboration Network
Top Collaborators
Jason S. Stumhofer
University of Arkansas for Medical Sciences
6 shared publications
In database
- The Costimulatory Molecule ICOS Regulates Host Th1 and Follicular Th Cell Differentiation in Response to Plasmodium chabaudi chabaudi AS Infection
- Do You See What I See: Recognition of Protozoan Parasites by Toll-Like Receptors
- An Atypical Splenic B Cell Progenitor Population Supports Antibody Production during Plasmodium Infection in Mice
- IL-17 Promotes Differentiation of Splenic LSK− Lymphoid Progenitors into B Cells following Plasmodium yoelii Infection
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
Showing 5 of 6 shared publications
Daniel J. Wikenheiser
University of Arkansas for Medical Sciences (US)
2 shared publications
- The Costimulatory Molecule ICOS Regulates Host Th1 and Follicular Th Cell Differentiation in Response to Plasmodium chabaudi chabaudi AS Infection
- An Atypical Splenic B Cell Progenitor Population Supports Antibody Production during Plasmodium Infection in Mice
B. W. Kennedy
University of Arkansas for Medical Sciences (US)
2 shared publications
- The Costimulatory Molecule ICOS Regulates Host Th1 and Follicular Th Cell Differentiation in Response to Plasmodium chabaudi chabaudi AS Infection
- An Atypical Splenic B Cell Progenitor Population Supports Antibody Production during Plasmodium Infection in Mice
Shana M. Owens
University of Arkansas for Medical Sciences
2 shared publications
In database
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated IgH/c-Myc Translocations
Jeffrey M. Sifford
University of Arkansas for Medical Sciences
2 shared publications
In database
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated IgH/c-Myc Translocations
Eduardo Salinas
University of Arkansas for Medical Sciences
2 shared publications
In database
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated IgH/c-Myc Translocations
J. Craig Forrest
Winthrop Rockefeller Foundation
2 shared publications
In database
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated IgH/c-Myc Translocations
Martin J. Cannon
University of Arkansas for Medical Sciences
1 shared publication
In database
- Signaling Circuits and Regulation of Immune Suppression by Ovarian Tumor-Associated Macrophages
Swetha Gujja
University of Arkansas for Medical Sciences (US)
1 shared publication
- Signaling Circuits and Regulation of Immune Suppression by Ovarian Tumor-Associated Macrophages
Kathryn E. McGovern
University of California, Riverside (US)
1 shared publication
- An Atypical Splenic B Cell Progenitor Population Supports Antibody Production during Plasmodium Infection in Mice
Johnasha D. Stuart
University of Arkansas for Medical Sciences (US)
1 shared publication
- An Atypical Splenic B Cell Progenitor Population Supports Antibody Production during Plasmodium Infection in Mice
Emma H. Wilson
University of California, Riverside (US)
1 shared publication
- An Atypical Splenic B Cell Progenitor Population Supports Antibody Production during Plasmodium Infection in Mice
Susie L. Brown
University of Arkansas for Medical Sciences
1 shared publication
In database
- IL-17 Promotes Differentiation of Splenic LSK− Lymphoid Progenitors into B Cells following Plasmodium yoelii Infection
Gang Li
University of Arkansas for Medical Sciences (US)
1 shared publication
- p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated IgH/c-Myc Translocations
Andrew D. Miller
Cornell University (US)
1 shared publication
- p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated IgH/c-Myc Translocations
Similar Researchers
Based on overlapping research topics
Manjunath Bettadapura
University of Arkansas for Medical Sciences
Host-Pathogen Interactions
Immune Response and Inflammation
Disease Mechanisms Research
Desislava A. Marinkova
University of Arkansas for Medical Sciences
Disease Mechanisms Research
Metabolism and Genetic Disorders
Diet and metabolism studies
Ji Li
University of Arkansas for Medical Sciences
Immune Response and Inflammation
Disease Mechanisms Research
Metabolism and Genetic Disorders
Adriana Lelis Carvalho
University of Arkansas for Medical Sciences
Disease Mechanisms Research
Metabolism and Genetic Disorders
Diet and metabolism studies
Sofia Shulga
University of Arkansas at Fayetteville
Immune Response and Inflammation
Disease Mechanisms Research
Metabolism and Genetic Disorders
Weston Perrine
University of Arkansas at Fayetteville
Host-Pathogen Interactions
Metabolism and Genetic Disorders
Diet and metabolism studies