Hong Wang
Researcher
Also affiliated: Hebei Medical University (2009); Union Hospital (2010–2024); Xuzhou Medical College (2025); Shenzhen University (2023); Capital Medical University (2024); Dalian Medical University (2022); Anhui Medical University (2018); Peking University (2016–2022); Sichuan University (2025); Beijing Jishuitan Hospital (2022); University of Alabama at Birmingham (2020); Third Affiliated Hospital of Southern Medical University (2021); National Clinical Research Center for Digestive Diseases (2019); West China Hospital of Sichuan University (2025); Laboratory of Molecular Genetics (2020); Shenyang Center for Disease Control and Prevention (2015); First Affiliated Hospital of Jiangxi Medical College (2023–2024); Huaian First People’s Hospital (2025); Second Hospital of Hebei Medical University (2009); Dalian Municipal Central Hospital (2022); Jiangxi Provincial People's Hospital (2012–2024); Affiliated Hospital of Chengde Medical College (2021); Aerospace Center Hospital (2022); Hubei University of Arts and Science (2022); Harbin Electric Corporation (China) (2021); Beijing Anzhen Hospital (2024); Xiangyang Central Hospital (2022); Second People’s Hospital of Huai’an (2025); Peking University First Hospital (2019); First Hospital of China Medical University (2009–2011); National Institute for Viral Disease Control and Prevention (2020); Peking University Third Hospital (2016); Wuhan Children's Hospital (2023); Union Hospital (2024); Huazhong University of Science and Technology Hospital (2024); Tongji Hospital (2014–2023); Huazhong University of Science and Technology (2007–2024); Hubei University (2023); China Medical University (2009–2011); The University of Texas Health Science Center at Houston (2009)
Faculty Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Hong Wang's research centers on understanding disease mechanisms and identifying biomarkers for various conditions, with a focus on cardiovascular health, bone metabolism, and infectious diseases. Wang has investigated myocardial dysfunction in COVID-19 patients using strain imaging and explored the role of ferroptosis-associated biomarkers in postmenopausal osteoporosis. Additionally, research has examined the effects of traditional Chinese medicine on COVID-19 patients and investigated genetic factors, such as RBM20 and ALPK3, associated with hypertrophic cardiomyopathy. Wang also studies the mechanisms underlying bone loss, including the impact of the WNT/β-catenin signaling pathway and filamin A, and has contributed to the development of a prognostic classification system for bone metastasis in cancer patients. Wang's work includes collaborations with Xiaolun Sun, Tahrir Alenezi, Rohana Liyanage, and Janashrit Shrestha at the University of Arkansas at Fayetteville.
Metrics
- h-index: 19
- Publications: 89
- Citations: 1,247
Selected Publications
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The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid (2025)
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Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence (2024)
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Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence (2023)
Collaboration Network
Top Collaborators
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
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