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Biography and Research Information
OverviewAI-generated summary
Jillian Hackney's research investigates mechanisms related to bone health and inflammatory conditions. Her work includes studying the healing of bone lesions and the restoration of bone health in multiple myeloma through sclerostin inhibition, as detailed in a 2025 publication. Hackney has also contributed to research interrogating the epithelium in active ulcerative colitis, identifying dysregulated myeloid cells associated with non-response to anti-Tumour Necrosis Factor therapy, as published in 2023. Her scholarly output includes two publications with one citation and an h-index of 1. She collaborates with researchers at the University of Arkansas for Medical Sciences, including Hayley M. Sabol, Japneet Kaur, Elena Ambrogini, and Cecile Bustamante-Gomez, with whom she shares publications.
Metrics
- h-index: 1
- Publications: 2
- Citations: 1
Selected Publications
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Healing of lytic lesions and restoration of bone health in multiple myeloma through sclerostin inhibition (2025)
Collaboration Network
Top Collaborators
Divya Jha
Icahn School of Medicine at Mount Sinai (US)
1 shared publication
- OP06 Multiomic interrogation of the epithelium in active Ulcerative Colitis reveals dysregulated myeloid cells associated with non-response to anti-Tumour Necrosis Factor therapy.
Z. Al-Taie
Icahn School of Medicine at Mount Sinai (US)
1 shared publication
- OP06 Multiomic interrogation of the epithelium in active Ulcerative Colitis reveals dysregulated myeloid cells associated with non-response to anti-Tumour Necrosis Factor therapy.
Azra Krek
Icahn School of Medicine at Mount Sinai (US)
1 shared publication
- OP06 Multiomic interrogation of the epithelium in active Ulcerative Colitis reveals dysregulated myeloid cells associated with non-response to anti-Tumour Necrosis Factor therapy.
Shirin Eshghi
1 shared publication
- OP06 Multiomic interrogation of the epithelium in active Ulcerative Colitis reveals dysregulated myeloid cells associated with non-response to anti-Tumour Necrosis Factor therapy.
Michael Tankelevich
Icahn School of Medicine at Mount Sinai (US)
1 shared publication
- OP06 Multiomic interrogation of the epithelium in active Ulcerative Colitis reveals dysregulated myeloid cells associated with non-response to anti-Tumour Necrosis Factor therapy.
H Meringer
Assuta Medical Center (IL)
1 shared publication
- OP06 Multiomic interrogation of the epithelium in active Ulcerative Colitis reveals dysregulated myeloid cells associated with non-response to anti-Tumour Necrosis Factor therapy.
F Cossarini
Icahn School of Medicine at Mount Sinai (US)
1 shared publication
- OP06 Multiomic interrogation of the epithelium in active Ulcerative Colitis reveals dysregulated myeloid cells associated with non-response to anti-Tumour Necrosis Factor therapy.
Pablo Canales-Herrerías
Icahn School of Medicine at Mount Sinai (US)
1 shared publication
- OP06 Multiomic interrogation of the epithelium in active Ulcerative Colitis reveals dysregulated myeloid cells associated with non-response to anti-Tumour Necrosis Factor therapy.
Alexandra E. Livanos
Icahn School of Medicine at Mount Sinai (US)
1 shared publication
- OP06 Multiomic interrogation of the epithelium in active Ulcerative Colitis reveals dysregulated myeloid cells associated with non-response to anti-Tumour Necrosis Factor therapy.
Alexandros D. Polydorides
Icahn School of Medicine at Mount Sinai (US)
1 shared publication
- OP06 Multiomic interrogation of the epithelium in active Ulcerative Colitis reveals dysregulated myeloid cells associated with non-response to anti-Tumour Necrosis Factor therapy.
Jérôme C. Martin
Nantes Université (FR)
1 shared publication
- OP06 Multiomic interrogation of the epithelium in active Ulcerative Colitis reveals dysregulated myeloid cells associated with non-response to anti-Tumour Necrosis Factor therapy.
H M Ko
City University of Seattle (US)
1 shared publication
- OP06 Multiomic interrogation of the epithelium in active Ulcerative Colitis reveals dysregulated myeloid cells associated with non-response to anti-Tumour Necrosis Factor therapy.
Jacqueline McBride
1 shared publication
- OP06 Multiomic interrogation of the epithelium in active Ulcerative Colitis reveals dysregulated myeloid cells associated with non-response to anti-Tumour Necrosis Factor therapy.
J F Colombel
Icahn School of Medicine at Mount Sinai (US)
1 shared publication
- OP06 Multiomic interrogation of the epithelium in active Ulcerative Colitis reveals dysregulated myeloid cells associated with non-response to anti-Tumour Necrosis Factor therapy.
Carmen Argmann
Icahn School of Medicine at Mount Sinai (US)
1 shared publication
- OP06 Multiomic interrogation of the epithelium in active Ulcerative Colitis reveals dysregulated myeloid cells associated with non-response to anti-Tumour Necrosis Factor therapy.
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