Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Vivek Khanal's research investigates the impact of diet on bone health, focusing on the role of osteocytes and transforming growth factor beta (TGF-β). His work examines how high-fat and high-carbohydrate diets can increase bone fragility by affecting osteocyte function through TGF-β signaling pathways. Khanal also studies the role of lipocalin-2 in regulating osteocyte ferroptosis and its influence on bone formation via crosstalk with osteoblasts through Wnt signaling. His research further explores sex-dependent mechanisms in acute obesity by analyzing transcriptional profiles of adipose tissue and their implications for interorgan communication. Khanal has published four papers, with his most recent work in 2026, and has accumulated 16 citations. He has collaborated with researchers at the University of Arkansas for Medical Sciences, including Neha S. Dole and Madeline Carroll.
Metrics
- h-index: 1
- Publications: 5
- Citations: 18
Selected Publications
-
Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression (2026)
-
Global Transcriptional Profiling of Adipose Tissue Uncovers Unique Sex-Dependent Mechanisms in Acute Obesity: Implications for Adipose-Heart Interorgan Crosstalk (2025)
-
Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation (2025)
-
High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function (2024)
Collaboration Network
Top Collaborators
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Global Transcriptional Profiling of Adipose Tissue Uncovers Unique Sex-Dependent Mechanisms in Acute Obesity: Implications for Adipose-Heart Interorgan Crosstalk
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
Similar Researchers
Based on overlapping research topics