Biography and Research Information
OverviewAI-generated summary
Jayden Carter's research focuses on the molecular mechanisms underlying bone formation and cellular communication, with a particular emphasis on the role of lipocalin-2. Recent publications investigate how osteocytic lipocalin-2 regulates bone formation through iron-dependent ferroptosis and Wnt signaling pathways, impacting crosstalk between osteocytes and osteoblasts. This work also examines the broader implications of histone deacetylases in cellular metabolism, exploring both established and potential avenues of research in this area. Carter has published four papers, with the most recent appearing in 2026. Collaborations include work with Hakan Paydak, Omar Alqaisi, Neha S. Dole, and Amy Y. Sato, all at the University of Arkansas for Medical Sciences.
Metrics
- h-index: 1
- Publications: 4
- Citations: 6
Selected Publications
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Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression (2026)
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Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation (2025)
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Histone Deacetylases in Metabolism: the Known and the Unexplored (2024)
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An Unusual Cause of Concordance in the Setting of Left Bundle Branch Block (2021)
Collaboration Network
Top Collaborators
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
- An Unusual Cause of Concordance in the Setting of Left Bundle Branch Block
- An Unusual Cause of Concordance in the Setting of Left Bundle Branch Block
- An Unusual Cause of Concordance in the Setting of Left Bundle Branch Block
- An Unusual Cause of Concordance in the Setting of Left Bundle Branch Block
- Histone Deacetylases in Metabolism: the Known and the Unexplored
- Histone Deacetylases in Metabolism: the Known and the Unexplored
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
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