Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Jayden Carter's research investigates the role of histone deacetylases (HDACs) in cellular processes, particularly focusing on their influence on metabolism. Carter has also explored the function of lipocalin-2 in bone formation, specifically its regulation of osteocyte ferroptosis and its interaction with Wnt signaling pathways to control osteoblast activity. This work has been published in peer-reviewed journals, with recent contributions in 2024, 2025, and 2026. Carter has collaborated with researchers including Neha S. Dole, Amy Y. Sato, Ryan M. Allen, and Umesh D. Wankhade, all from the University of Arkansas for Medical Sciences, with whom multiple shared publications exist. Carter's scholarship metrics include an h-index of 2 and a total of 4 publications with 12 citations.
Metrics
- h-index: 2
- Publications: 4
- Citations: 12
Selected Publications
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Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression (2026)
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Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation (2025)
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Histone Deacetylases in Metabolism: the Known and the Unexplored (2024)
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An Unusual Cause of Concordance in the Setting of Left Bundle Branch Block (2021)
Collaboration Network
Top Collaborators
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
- An Unusual Cause of Concordance in the Setting of Left Bundle Branch Block
- An Unusual Cause of Concordance in the Setting of Left Bundle Branch Block
- An Unusual Cause of Concordance in the Setting of Left Bundle Branch Block
- An Unusual Cause of Concordance in the Setting of Left Bundle Branch Block
- Histone Deacetylases in Metabolism: the Known and the Unexplored
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
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