Abhishek Mishra
Researcher
Also affiliated: Bharat Electronics (India) (2023); Centre for Cellular and Molecular Biology (2015–2019); SUNY Upstate Medical University (2020); Tripoli Central Hospital (2008); College of Medical Sciences (2024); King George's Medical University (2011–2022); Indian Institute of Science Education and Research, Bhopal (2019); IQVIA (United Kingdom) (2019); Indian Institute of Management Indore (2023); Guthrie Robert Packer Hospital (2020); All India Institute of Medical Sciences Bhopal (2023); People’s University (2024); Command Hospital (2023–2025); Maulana Azad National Institute of Technology (2023)
Faculty Researcher
Research Areas
Biomedical Subjects
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Biography and Research Information
OverviewAI-generated summary
Abhishek Mishra's research interests encompass a range of medical and scientific disciplines, as indicated by his diverse publication record. He has investigated the effects of synthetic epoxyeicosatrienoic acid mimics on mesangial cells, focusing on protection from sorafenib-induced cell death. Additionally, his work has explored the suppression of TLR signaling by IRAK-1 and -4 dual inhibitors to decrease pro-oncogenic effects in head and neck squamous cell carcinoma (HNSCC). Mishra has also examined the impact of manual techniques and breathing exercises on pulmonary function in post-COVID-19 survivors. His collaborative network includes researchers from the University of Arkansas for Medical Sciences, such as John D. Imig, Samaneh Goorani, Abdul H. Khan, and Henry A. Palfrey, with whom he has co-authored multiple publications. Mishra's scholarship metrics include an h-index of 12, with 56 total publications and 564 citations.
Metrics
- h-index: 12
- Publications: 56
- Citations: 574
Selected Publications
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EET-Based Therapeutics Mitigate Sorafenib-Associated Glomerular Cell Damage (2025)
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Transforming growth factor β receptor 1 and mitogen-activated protein 4 kinase 4 dual inhibitor, TK-850, reduces renal fibrosis in unilateral ureteral–obstructed mice (2025)
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591 Low kidney mass contributes to enhanced fractionated irradiation-induced renal hemodynamic dysfunction in mice (2025)
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Synthetic Epoxyeicosatrienoic Acid Mimics Protect Mesangial Cells from Sorafenib-Induced Cell Death (2025)
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The Immune System in Nephrotoxicity (2024)
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Kidney Injury by Unilateral Ureteral Obstruction in Mice Lacks Sex Differences (2024)
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Epoxyeicosatrienoic Acid Mimics Protect Mesangial Cells from Sorafenib Induced Cell Death (2023)
Collaboration Network
Top Collaborators
- Kidney Injury by Unilateral Ureteral Obstruction in Mice Lacks Sex Differences
- Synthetic Epoxyeicosatrienoic Acid Mimics Protect Mesangial Cells from Sorafenib-Induced Cell Death
- Epoxyeicosatrienoic Acid Mimics Protect Mesangial Cells from Sorafenib Induced Cell Death
- The Immune System in Nephrotoxicity
- 591 Low kidney mass contributes to enhanced fractionated irradiation-induced renal hemodynamic dysfunction in mice
Showing 5 of 7 shared publications
- Kidney Injury by Unilateral Ureteral Obstruction in Mice Lacks Sex Differences
- The Immune System in Nephrotoxicity
- 591 Low kidney mass contributes to enhanced fractionated irradiation-induced renal hemodynamic dysfunction in mice
- Transforming growth factor β receptor 1 and mitogen-activated protein 4 kinase 4 dual inhibitor, TK-850, reduces renal fibrosis in unilateral ureteral–obstructed mice
- EET-Based Therapeutics Mitigate Sorafenib-Associated Glomerular Cell Damage
- Kidney Injury by Unilateral Ureteral Obstruction in Mice Lacks Sex Differences
- 591 Low kidney mass contributes to enhanced fractionated irradiation-induced renal hemodynamic dysfunction in mice
- Transforming growth factor β receptor 1 and mitogen-activated protein 4 kinase 4 dual inhibitor, TK-850, reduces renal fibrosis in unilateral ureteral–obstructed mice
- EET-Based Therapeutics Mitigate Sorafenib-Associated Glomerular Cell Damage
- Synthetic Epoxyeicosatrienoic Acid Mimics Protect Mesangial Cells from Sorafenib-Induced Cell Death
- Epoxyeicosatrienoic Acid Mimics Protect Mesangial Cells from Sorafenib Induced Cell Death
- EET-Based Therapeutics Mitigate Sorafenib-Associated Glomerular Cell Damage
- Synthetic Epoxyeicosatrienoic Acid Mimics Protect Mesangial Cells from Sorafenib-Induced Cell Death
- Epoxyeicosatrienoic Acid Mimics Protect Mesangial Cells from Sorafenib Induced Cell Death
- EET-Based Therapeutics Mitigate Sorafenib-Associated Glomerular Cell Damage
- Kidney Injury by Unilateral Ureteral Obstruction in Mice Lacks Sex Differences
- Epoxyeicosatrienoic Acid Mimics Protect Mesangial Cells from Sorafenib Induced Cell Death
- Synthetic Epoxyeicosatrienoic Acid Mimics Protect Mesangial Cells from Sorafenib-Induced Cell Death
- Epoxyeicosatrienoic Acid Mimics Protect Mesangial Cells from Sorafenib Induced Cell Death
- 591 Low kidney mass contributes to enhanced fractionated irradiation-induced renal hemodynamic dysfunction in mice
- Transforming growth factor β receptor 1 and mitogen-activated protein 4 kinase 4 dual inhibitor, TK-850, reduces renal fibrosis in unilateral ureteral–obstructed mice
- Epoxyeicosatrienoic Acid Mimics Protect Mesangial Cells from Sorafenib Induced Cell Death
- Epoxyeicosatrienoic Acid Mimics Protect Mesangial Cells from Sorafenib Induced Cell Death
- Epoxyeicosatrienoic Acid Mimics Protect Mesangial Cells from Sorafenib Induced Cell Death
- Epoxyeicosatrienoic Acid Mimics Protect Mesangial Cells from Sorafenib Induced Cell Death
- Synthetic Epoxyeicosatrienoic Acid Mimics Protect Mesangial Cells from Sorafenib-Induced Cell Death
- Synthetic Epoxyeicosatrienoic Acid Mimics Protect Mesangial Cells from Sorafenib-Induced Cell Death
- 591 Low kidney mass contributes to enhanced fractionated irradiation-induced renal hemodynamic dysfunction in mice
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