Henry A. Palfrey
Postdoctoral fellow
Also affiliated: Pennington Biomedical Research Center (2024); Southern University and Agricultural and Mechanical College (2017–2024)
Formerly Arkansas Postdoctoral Research Associate / HSIE fellow, UAMS through 2026; now Postdoctoral fellow, University of Arkansas for Medical Sciences.
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Henry A. Palfrey is a postdoctoral fellow at the University of Arkansas for Medical Sciences, where his research focuses on the role of eicosanoids in kidney and cardiovascular function, with a specific interest in radiation-induced kidney injury and potential pharmacotherapeutic interventions. He is also involved in developing a novel medical device through a research commercialization fellowship, aiming for market launch. Palfrey has co-authored publications investigating the metabolic and physiological effects of dietary components and pharmaceutical agents in rodent models, including studies on sitagliptin, homocysteine, and cholesterol metabolism. His work also includes research on kidney injury models, such as those induced by sorafenib treatment and unilateral ureteral obstruction, and the impact of low kidney mass on irradiation-induced renal dysfunction. Palfrey collaborates with researchers from the University of Arkansas for Medical Sciences and the University of Arkansas at Fayetteville.
Metrics
- h-index: 4
- Publications: 11
- Citations: 424
Positions
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Postdoctoral fellow publications 2024–2026University of Arkansas for Medical Sciences Institution web page
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Postdoctoral Research Associate / HSIE fellow 2023–2026University of Arkansas for Medical Sciences Pharmaceutical Sciences ORCID
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Graduate Research Assistant 2015–2020Southern University and Agricultural and Mechanical College Environmental Toxicology ORCID
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FDA Summer Research Intern @ NCTR 2017Oak Ridge Institute for Science and Education Biochemical Toxicology ORCID
Selected Publications
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Sorafenib Treatment Results in Greater Kidney Injury in Renal Hypoplasia Mice (Abstract ID: 272359) (2026)
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Transforming growth factor β receptor 1 and mitogen-activated protein 4 kinase 4 dual inhibitor, TK-850, reduces renal fibrosis in unilateral ureteral–obstructed mice (2025)
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591 Low kidney mass contributes to enhanced fractionated irradiation-induced renal hemodynamic dysfunction in mice (2025)
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Attenuation of renal injury by administration of TK-850, a dual inhibitor of TGFβR1/MAP4K4 (2024)
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557 Dual TGFβR1/MAP4K4 inhibitor reduces kidney injury in a mouse model of renal fibrosis. (2024)
Collaboration Network
Top Collaborators
- 557 Dual TGFβR1/MAP4K4 inhibitor reduces kidney injury in a mouse model of renal fibrosis.
- Attenuation of renal injury by administration of TK-850, a dual inhibitor of TGFβR1/MAP4K4
- 591 Low kidney mass contributes to enhanced fractionated irradiation-induced renal hemodynamic dysfunction in mice
- Transforming growth factor β receptor 1 and mitogen-activated protein 4 kinase 4 dual inhibitor, TK-850, reduces renal fibrosis in unilateral ureteral–obstructed mice
- Sorafenib Treatment Results in Greater Kidney Injury in Renal Hypoplasia Mice (Abstract ID: 272359)
- 557 Dual TGFβR1/MAP4K4 inhibitor reduces kidney injury in a mouse model of renal fibrosis.
- Attenuation of renal injury by administration of TK-850, a dual inhibitor of TGFβR1/MAP4K4
- 591 Low kidney mass contributes to enhanced fractionated irradiation-induced renal hemodynamic dysfunction in mice
- Transforming growth factor β receptor 1 and mitogen-activated protein 4 kinase 4 dual inhibitor, TK-850, reduces renal fibrosis in unilateral ureteral–obstructed mice
- 557 Dual TGFβR1/MAP4K4 inhibitor reduces kidney injury in a mouse model of renal fibrosis.
- Attenuation of renal injury by administration of TK-850, a dual inhibitor of TGFβR1/MAP4K4
- Transforming growth factor β receptor 1 and mitogen-activated protein 4 kinase 4 dual inhibitor, TK-850, reduces renal fibrosis in unilateral ureteral–obstructed mice
- 557 Dual TGFβR1/MAP4K4 inhibitor reduces kidney injury in a mouse model of renal fibrosis.
- Attenuation of renal injury by administration of TK-850, a dual inhibitor of TGFβR1/MAP4K4
- Transforming growth factor β receptor 1 and mitogen-activated protein 4 kinase 4 dual inhibitor, TK-850, reduces renal fibrosis in unilateral ureteral–obstructed mice
- 557 Dual TGFβR1/MAP4K4 inhibitor reduces kidney injury in a mouse model of renal fibrosis.
- Attenuation of renal injury by administration of TK-850, a dual inhibitor of TGFβR1/MAP4K4
- 591 Low kidney mass contributes to enhanced fractionated irradiation-induced renal hemodynamic dysfunction in mice
- Transforming growth factor β receptor 1 and mitogen-activated protein 4 kinase 4 dual inhibitor, TK-850, reduces renal fibrosis in unilateral ureteral–obstructed mice
- 591 Low kidney mass contributes to enhanced fractionated irradiation-induced renal hemodynamic dysfunction in mice
- Transforming growth factor β receptor 1 and mitogen-activated protein 4 kinase 4 dual inhibitor, TK-850, reduces renal fibrosis in unilateral ureteral–obstructed mice
- 591 Low kidney mass contributes to enhanced fractionated irradiation-induced renal hemodynamic dysfunction in mice
- 591 Low kidney mass contributes to enhanced fractionated irradiation-induced renal hemodynamic dysfunction in mice
- Sorafenib Treatment Results in Greater Kidney Injury in Renal Hypoplasia Mice (Abstract ID: 272359)
- Sorafenib Treatment Results in Greater Kidney Injury in Renal Hypoplasia Mice (Abstract ID: 272359)
- Sorafenib Treatment Results in Greater Kidney Injury in Renal Hypoplasia Mice (Abstract ID: 272359)
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