Arif Hameed
Researcher
Formerly Arkansas Affiliated with UAMS through 2002; recent publications list Aga Khan University Hospital.
Faculty Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Arif Hameed's research explores the intersection of diet, metabolism, and health, with a focus on human physiology and disease states. His work has investigated the impact of dietary components like polyphenols, cinnamon, and capsicum on postprandial metabolic changes, particularly in individuals with overweight or obesity. He has also examined the role of aquaporins in insulin resistance and diabetes, extending beyond their function as simple channels. Hameed's publications also touch upon various clinical and methodological topics, including the characterization of fruit polyphenols, validation of self-collected blood for lipidomics, and adherence to reporting guidelines for surgical case reports. He has a significant publication record, with over 50 publications and more than 1,600 citations, and maintains active collaborations with researchers at the University of Arkansas for Medical Sciences.
Metrics
- h-index: 16
- Publications: 51
- Citations: 1,612
Selected Publications
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Lipolysis of host triacylglyceride-rich lipoproteins creates a toxic microenvironment for <i>Staphylococcus aureus</i> (2026)
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Comparison of the capillary and venous blood plasma lipidomes: validation of self-collected blood for plasma lipidomics (2025)
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Dietary modulation of human milk bioactives is associated with maternal FUT2 secretor phenotype: an exploratory analysis of carotenoids and polyphenol metabolites (2024)
Collaboration Network
Top Collaborators
- Comparison of the capillary and venous blood plasma lipidomes: validation of self-collected blood for plasma lipidomics
- Dietary modulation of human milk bioactives is associated with maternal FUT2 secretor phenotype: an exploratory analysis of carotenoids and polyphenol metabolites
- Comparison of the capillary and venous blood plasma lipidomes: validation of self-collected blood for plasma lipidomics
- Lipolysis of host triacylglyceride-rich lipoproteins creates a toxic microenvironment for <i>Staphylococcus aureus</i>
- Dietary modulation of human milk bioactives is associated with maternal FUT2 secretor phenotype: an exploratory analysis of carotenoids and polyphenol metabolites
- Dietary modulation of human milk bioactives is associated with maternal FUT2 secretor phenotype: an exploratory analysis of carotenoids and polyphenol metabolites
- Dietary modulation of human milk bioactives is associated with maternal FUT2 secretor phenotype: an exploratory analysis of carotenoids and polyphenol metabolites
- Dietary modulation of human milk bioactives is associated with maternal FUT2 secretor phenotype: an exploratory analysis of carotenoids and polyphenol metabolites
- Dietary modulation of human milk bioactives is associated with maternal FUT2 secretor phenotype: an exploratory analysis of carotenoids and polyphenol metabolites
- Dietary modulation of human milk bioactives is associated with maternal FUT2 secretor phenotype: an exploratory analysis of carotenoids and polyphenol metabolites
- Dietary modulation of human milk bioactives is associated with maternal FUT2 secretor phenotype: an exploratory analysis of carotenoids and polyphenol metabolites
- Comparison of the capillary and venous blood plasma lipidomes: validation of self-collected blood for plasma lipidomics
- Comparison of the capillary and venous blood plasma lipidomes: validation of self-collected blood for plasma lipidomics
- Comparison of the capillary and venous blood plasma lipidomes: validation of self-collected blood for plasma lipidomics
- Lipolysis of host triacylglyceride-rich lipoproteins creates a toxic microenvironment for <i>Staphylococcus aureus</i>
- Lipolysis of host triacylglyceride-rich lipoproteins creates a toxic microenvironment for <i>Staphylococcus aureus</i>
- Lipolysis of host triacylglyceride-rich lipoproteins creates a toxic microenvironment for <i>Staphylococcus aureus</i>
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