Dinesh Bhattarai Institution-verified

Sourced from institutional research profiles (UAMS TRI or ARA).

Post Doctoral Fellow

UAMS

Last publication 2026 Last refreshed 2026-05-23

postdoc

bhattaraidinesh22@gmail.com

14 h-index 48 pubs 857 cited

Research Areas

Links

ORCID Lab Website UAMS TRI Profile

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OverviewAI-generated summary

Dinesh Bhattarai's research focuses on kidney transplantation and related injury mechanisms. He has investigated the effects of cold storage and hypothermic machine perfusion on rat kidney allografts, examining changes in proteome and phosphoproteome landscapes, and the role of oxidative stress and tubular injury. Bhattarai's work also explores the immunoproteasome's function in kidney allografts post-transplantation and its impact on graft function. Additionally, his research has touched upon the role of complement 5 in mitochondria during kidney ischemia-reperfusion injury in rats and the regulatory function of NLRP6 in host defense against polymicrobial sepsis. Bhattarai has a publication record of 46 articles, with a total of 849 citations and an h-index of 14. He has collaborated with researchers including Nirmala Parajuli, Seongok Lee, Madison McGraw, and Lee Ann MacMillan-Crow.

Metrics

h-index: 14
Publications: 48
Citations: 857

Selected Publications

Influence of Hypothermic Machine Perfusion on Markers of Oxidative Stress and Early Tubular Injury in Rat Donor Kidneys Before Transplantation (2026)

DOI OpenAlex
Complement 5's Role in Mitochondria During Kidney Ischemia-Reperfusion Injury in Rats (2025)

DOI OpenAlex
Compared with Static Cold Storage, Hypothermic Machine Perfusion Mitigates Oxidative Stress and Tubular Injury in Rat Kidneys (2025)

DOI OpenAlex
Post‐ischemia and reperfusion kidney injury is mitigated in a novel complement 5 knockout rat (2025)

DOI OpenAlex
Cold Storage-Induced Proteasome Dysfunction in Rat Kidney Transplants: A Plausible Role of p38MAPK Signaling (Abstract ID: 161861) (2025)

DOI OpenAlex
Ischemia and Reperfusion (IRI)-Induced Kidney Injury Is Attenuated by Complement 5 Gene Deletion (Abstract ID: 161749) (2025)

DOI OpenAlex
Cold Storage Disrupts the Proteome and Phosphoproteome Landscape in Rat Kidney Transplants (2024)

1 citation DOI OpenAlex
Cold Storage-Mediated Activation of P38MAPK-MK2 Axis Induces Kidney Injury after Transplantation (2024)

DOI OpenAlex
Normal Proteasome Function Is Needed to Prevent Kidney Graft Injury during Cold Storage Followed by Transplantation (2024)

2 citations DOI OpenAlex
Cold Storage Followed by Transplantation Induces Immunoproteasome in Rat Kidney Allografts: Inhibition of Immunoproteasome Does Not Improve Function (2024)

4 citations DOI OpenAlex
Ischemia-Reperfusion-Mediated Kidney Injury Is Attenuated in a New Complement 5 Knockout Model (2023)

DOI OpenAlex
Cold Storage-Mediated p38MAPK Activation: A Potential Contributor of Proteasome Dysfunction and Kidney Damage After Transplantation (2023)

DOI OpenAlex