Biography and Research Information
OverviewAI-generated summary
Fengping Lv's research focuses on the design, synthesis, and biological evaluation of novel chemical compounds. This work includes the discovery of pyrazolo-thieno[3,2-d]pyrimidinylamino-phenyl acetamides as type-II pan-tropomyosin receptor kinase (TRK) inhibitors, as detailed in a 2021 publication. Lv also investigates biological processes at the molecular level, including research on ubiquitination-mediated degradation of TRDMT1 and its regulation of homologous recombination and therapeutic response, also published in 2021. Lv has a h-index of 12, with 28 total publications and 811 citations. Collaborations include work with Brendan Frett at the University of Arkansas for Medical Sciences, with whom Lv shares one publication.
Metrics
- h-index: 12
- Publications: 28
- Citations: 813
Selected Publications
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Ubiquitination-mediated degradation of TRDMT1 regulates homologous recombination and therapeutic response (2021)
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Discovery of pyrazolo-thieno[3,2-d]pyrimidinylamino-phenyl acetamides as type-II pan-tropomyosin receptor kinase (TRK) inhibitors: Design, synthesis, and biological evaluation (2021)
Collaboration Network
Top Collaborators
- Discovery of pyrazolo-thieno[3,2-d]pyrimidinylamino-phenyl acetamides as type-II pan-tropomyosin receptor kinase (TRK) inhibitors: Design, synthesis, and biological evaluation
- Ubiquitination-mediated degradation of TRDMT1 regulates homologous recombination and therapeutic response
- Discovery of pyrazolo-thieno[3,2-d]pyrimidinylamino-phenyl acetamides as type-II pan-tropomyosin receptor kinase (TRK) inhibitors: Design, synthesis, and biological evaluation
- Ubiquitination-mediated degradation of TRDMT1 regulates homologous recombination and therapeutic response
- Discovery of pyrazolo-thieno[3,2-d]pyrimidinylamino-phenyl acetamides as type-II pan-tropomyosin receptor kinase (TRK) inhibitors: Design, synthesis, and biological evaluation
- Discovery of pyrazolo-thieno[3,2-d]pyrimidinylamino-phenyl acetamides as type-II pan-tropomyosin receptor kinase (TRK) inhibitors: Design, synthesis, and biological evaluation
- Discovery of pyrazolo-thieno[3,2-d]pyrimidinylamino-phenyl acetamides as type-II pan-tropomyosin receptor kinase (TRK) inhibitors: Design, synthesis, and biological evaluation
- Discovery of pyrazolo-thieno[3,2-d]pyrimidinylamino-phenyl acetamides as type-II pan-tropomyosin receptor kinase (TRK) inhibitors: Design, synthesis, and biological evaluation
- Discovery of pyrazolo-thieno[3,2-d]pyrimidinylamino-phenyl acetamides as type-II pan-tropomyosin receptor kinase (TRK) inhibitors: Design, synthesis, and biological evaluation
- Discovery of pyrazolo-thieno[3,2-d]pyrimidinylamino-phenyl acetamides as type-II pan-tropomyosin receptor kinase (TRK) inhibitors: Design, synthesis, and biological evaluation
- Discovery of pyrazolo-thieno[3,2-d]pyrimidinylamino-phenyl acetamides as type-II pan-tropomyosin receptor kinase (TRK) inhibitors: Design, synthesis, and biological evaluation
- Ubiquitination-mediated degradation of TRDMT1 regulates homologous recombination and therapeutic response
- Ubiquitination-mediated degradation of TRDMT1 regulates homologous recombination and therapeutic response
- Ubiquitination-mediated degradation of TRDMT1 regulates homologous recombination and therapeutic response
- Ubiquitination-mediated degradation of TRDMT1 regulates homologous recombination and therapeutic response
- Ubiquitination-mediated degradation of TRDMT1 regulates homologous recombination and therapeutic response
- Ubiquitination-mediated degradation of TRDMT1 regulates homologous recombination and therapeutic response
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