Manuel Alejandro Ramirez-Lee
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Also affiliated: United States Food and Drug Administration (2012–2021); Autonomous University of San Luis Potosí (2011–2017)
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Manuel Alejandro Ramirez-Lee's research investigates mechanisms of neurotoxicity, with a focus on nanoparticle interactions with biological systems. His work has examined the potential for SARS-CoV-2 to infect the central nervous system and explored autophagy as a mechanism in dopaminergic degeneration. Ramirez-Lee also studies rodent models of anesthesia-induced developmental neurotoxicity. His scholarship metrics include an h-index of 12, with 18 total publications and over 500 citations. He has collaborated with researchers including John Talpos, Susan M. Burks, Héctor Rosas-Hernández, and Syed F. Ali, all from the National Center for Toxicological Research.
Metrics
- h-index: 12
- Publications: 18
- Citations: 517
Positions
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Professor of Chemistry 2017–2018Universidad Autonoma de San Luis Potosi Facultad de Ciencias ORCID
Selected Publications
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Rodent models of anesthesia-induced developmental neurotoxicity☆ (2022)
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Autophagy and protein aggregation as a mechanism of dopaminergic degeneration in a primary human dopaminergic neuronal model (2022)
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Can SARS-CoV-2 infect the central nervous system via the olfactory bulb or the blood-brain barrier? (2021)
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Amyloid Beta 25–35 induces blood-brain barrier disruption in vitro (2019)
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Cytotoxicity profile of pristine graphene on brain microvascular endothelial cells (2019)
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Nicotine alters the expression of molecular markers of endocrine disruption in zebrafish (2012)
Collaboration Network
Top Collaborators
- Can SARS-CoV-2 infect the central nervous system via the olfactory bulb or the blood-brain barrier?
- Amyloid Beta 25–35 induces blood-brain barrier disruption in vitro
- Nicotine alters the expression of molecular markers of endocrine disruption in zebrafish
- Cytotoxicity profile of pristine graphene on brain microvascular endothelial cells
- Autophagy and protein aggregation as a mechanism of dopaminergic degeneration in a primary human dopaminergic neuronal model
- Amyloid Beta 25–35 induces blood-brain barrier disruption in vitro
- Nicotine alters the expression of molecular markers of endocrine disruption in zebrafish
- Cytotoxicity profile of pristine graphene on brain microvascular endothelial cells
- Autophagy and protein aggregation as a mechanism of dopaminergic degeneration in a primary human dopaminergic neuronal model
- Can SARS-CoV-2 infect the central nervous system via the olfactory bulb or the blood-brain barrier?
- Amyloid Beta 25–35 induces blood-brain barrier disruption in vitro
- Cytotoxicity profile of pristine graphene on brain microvascular endothelial cells
- Amyloid Beta 25–35 induces blood-brain barrier disruption in vitro
- Cytotoxicity profile of pristine graphene on brain microvascular endothelial cells
- Autophagy and protein aggregation as a mechanism of dopaminergic degeneration in a primary human dopaminergic neuronal model
- Can SARS-CoV-2 infect the central nervous system via the olfactory bulb or the blood-brain barrier?
- Amyloid Beta 25–35 induces blood-brain barrier disruption in vitro
- Autophagy and protein aggregation as a mechanism of dopaminergic degeneration in a primary human dopaminergic neuronal model
- Nicotine alters the expression of molecular markers of endocrine disruption in zebrafish
- Cytotoxicity profile of pristine graphene on brain microvascular endothelial cells
- Amyloid Beta 25–35 induces blood-brain barrier disruption in vitro
- Autophagy and protein aggregation as a mechanism of dopaminergic degeneration in a primary human dopaminergic neuronal model
- Can SARS-CoV-2 infect the central nervous system via the olfactory bulb or the blood-brain barrier?
- Rodent models of anesthesia-induced developmental neurotoxicity☆
- Nicotine alters the expression of molecular markers of endocrine disruption in zebrafish
- Nicotine alters the expression of molecular markers of endocrine disruption in zebrafish
- Nicotine alters the expression of molecular markers of endocrine disruption in zebrafish
- Nicotine alters the expression of molecular markers of endocrine disruption in zebrafish
- Cytotoxicity profile of pristine graphene on brain microvascular endothelial cells
- Cytotoxicity profile of pristine graphene on brain microvascular endothelial cells
- Cytotoxicity profile of pristine graphene on brain microvascular endothelial cells
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