Madison Clements
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Also affiliated: East Carolina University (2022)
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Madison Clements' research focuses on the intersection of cardiovascular diseases and viral infections, particularly in the context of COVID-19. Her work investigates the roles of cardiac fibroblasts and myofibroblasts in the development and progression of cardiovascular pathology during infection. Clements has also explored the immunological mechanisms underlying hypertension, specifically the contribution of kidney-resident memory T cells and cytokine-mediated macrophage transitions to cardiac fibrosis.
She has published four papers, with a total of 24 citations and an h-index of 1. Her recent publications in 2026 address the immune cell dynamics in hypertension and the potential for cytokine-mediated processes to promote cardiac fibrosis. Clements collaborates with Lance Benson at the University of Arkansas for Medical Sciences, with whom she shares one publication. Her recent activity indicates ongoing research in these areas.
Metrics
- h-index: 1
- Publications: 4
- Citations: 24
Selected Publications
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Antigen-independent formation of renal Trm-like CD8 T cells perpetuates hypertension 2259853 (2026)
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Kidney Resident Memory CD8+ T cells drive the immortality of hypertension (2026)
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Cytokine-mediated macrophage transition promotes early cardiac fibrosis in chronic hypertension (2026)
Collaboration Network
Top Collaborators
- Cytokine-mediated macrophage transition promotes early cardiac fibrosis in chronic hypertension
- Kidney Resident Memory CD8+ T cells drive the immortality of hypertension
- Antigen-independent formation of renal Trm-like CD8 T cells perpetuates hypertension 2259853
- Cytokine-mediated macrophage transition promotes early cardiac fibrosis in chronic hypertension
- Kidney Resident Memory CD8+ T cells drive the immortality of hypertension
- Antigen-independent formation of renal Trm-like CD8 T cells perpetuates hypertension 2259853
- Cytokine-mediated macrophage transition promotes early cardiac fibrosis in chronic hypertension
- Kidney Resident Memory CD8+ T cells drive the immortality of hypertension
- Antigen-independent formation of renal Trm-like CD8 T cells perpetuates hypertension 2259853
- Cytokine-mediated macrophage transition promotes early cardiac fibrosis in chronic hypertension
- Kidney Resident Memory CD8+ T cells drive the immortality of hypertension
- Antigen-independent formation of renal Trm-like CD8 T cells perpetuates hypertension 2259853
- Cytokine-mediated macrophage transition promotes early cardiac fibrosis in chronic hypertension
- Kidney Resident Memory CD8+ T cells drive the immortality of hypertension
- Antigen-independent formation of renal Trm-like CD8 T cells perpetuates hypertension 2259853
- Cytokine-mediated macrophage transition promotes early cardiac fibrosis in chronic hypertension
- Kidney Resident Memory CD8+ T cells drive the immortality of hypertension
- Antigen-independent formation of renal Trm-like CD8 T cells perpetuates hypertension 2259853
- Cytokine-mediated macrophage transition promotes early cardiac fibrosis in chronic hypertension
- Kidney Resident Memory CD8+ T cells drive the immortality of hypertension
- Cytokine-mediated macrophage transition promotes early cardiac fibrosis in chronic hypertension
- Kidney Resident Memory CD8+ T cells drive the immortality of hypertension
- Cytokine-mediated macrophage transition promotes early cardiac fibrosis in chronic hypertension
- Kidney Resident Memory CD8+ T cells drive the immortality of hypertension
- Kidney Resident Memory CD8+ T cells drive the immortality of hypertension
- Kidney Resident Memory CD8+ T cells drive the immortality of hypertension
- Antigen-independent formation of renal Trm-like CD8 T cells perpetuates hypertension 2259853
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