Biography and Research Information
OverviewAI-generated summary
Li Xia's research investigates molecular mechanisms underlying disease, with recent work focusing on the role of metabolic pathways and epigenetic modifications in cancer progression, specifically pancreatic ductal adenocarcinoma. Xia has also explored the impact of non-coding RNAs, such as circular RNAs, on inflammatory responses in skin conditions like atopic dermatitis and psoriasis, often examining these processes in the context of specific molecular pathways, including N6-methyladenosine modification. Further research interests include the cellular sensing of nutrient availability and its connection to cellular signaling pathways like mTOR. Additionally, Xia's work has touched upon the development and application of targeted radiotracers for cancer imaging and therapy, as well as the environmental impact of nanoplastics on cellular health. Xia has published 124 papers, with a total of 5,455 citations and an h-index of 15.
Metrics
- h-index: 15
- Publications: 123
- Citations: 5,527
Selected Publications
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N-nitroso-ethylisopropylamine mutagenicity in rat liver using the cII transgenic mutation assay and duplex sequencing analysis of genomic DNA (2025)
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Germline mutagenicity of molnupiravir and its active form, β-d-N4-hydroxycytidine, in Caenorhabditis elegans evaluated using whole-genome sequencing (2025)
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Genotoxicity of nanoparticles evaluated using the <i>in vitro</i> micronucleus assay, a review of recent data <sup>§</sup> (2025)
Collaboration Network
Top Collaborators
- High Affinity and FAP-Targeted Radiotracers: A Potential Design Strategy to Improve the Pharmacokinetics and Tumor Uptake for FAP Inhibitors
- Improve the Biodistribution with Bulky and Lipophilic Modification Strategies on Lys-Urea-Glu-Based PSMA-Targeting Radiotracers
- High Affinity and FAP-Targeted Radiotracers: A Potential Design Strategy to Improve the Pharmacokinetics and Tumor Uptake for FAP Inhibitors
- Improve the Biodistribution with Bulky and Lipophilic Modification Strategies on Lys-Urea-Glu-Based PSMA-Targeting Radiotracers
- High Affinity and FAP-Targeted Radiotracers: A Potential Design Strategy to Improve the Pharmacokinetics and Tumor Uptake for FAP Inhibitors
- Improve the Biodistribution with Bulky and Lipophilic Modification Strategies on Lys-Urea-Glu-Based PSMA-Targeting Radiotracers
- High Affinity and FAP-Targeted Radiotracers: A Potential Design Strategy to Improve the Pharmacokinetics and Tumor Uptake for FAP Inhibitors
- Improve the Biodistribution with Bulky and Lipophilic Modification Strategies on Lys-Urea-Glu-Based PSMA-Targeting Radiotracers
- High Affinity and FAP-Targeted Radiotracers: A Potential Design Strategy to Improve the Pharmacokinetics and Tumor Uptake for FAP Inhibitors
- Improve the Biodistribution with Bulky and Lipophilic Modification Strategies on Lys-Urea-Glu-Based PSMA-Targeting Radiotracers
- Germline mutagenicity of molnupiravir and its active form, β-d-N4-hydroxycytidine, in Caenorhabditis elegans evaluated using whole-genome sequencing
- N-nitroso-ethylisopropylamine mutagenicity in rat liver using the cII transgenic mutation assay and duplex sequencing analysis of genomic DNA
- Research on the Influence of the Trust of the Government in Different Order on the Environmental Performance Evaluation of the People s Government and the Intermediary Mechanism
- Research on the Influence of the Trust of the Government in Different Order on the Environmental Performance Evaluation of the People s Government and the Intermediary Mechanism
- CD34 is a target for covalent EGFR inhibitors to eliminate stem/progenitor cells in acute and chronic myeloid leukemia
- CD34 is a target for covalent EGFR inhibitors to eliminate stem/progenitor cells in acute and chronic myeloid leukemia
- CD34 is a target for covalent EGFR inhibitors to eliminate stem/progenitor cells in acute and chronic myeloid leukemia
- CD34 is a target for covalent EGFR inhibitors to eliminate stem/progenitor cells in acute and chronic myeloid leukemia
- CD34 is a target for covalent EGFR inhibitors to eliminate stem/progenitor cells in acute and chronic myeloid leukemia
- CD34 is a target for covalent EGFR inhibitors to eliminate stem/progenitor cells in acute and chronic myeloid leukemia
- CD34 is a target for covalent EGFR inhibitors to eliminate stem/progenitor cells in acute and chronic myeloid leukemia
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