Biography and Research Information
OverviewAI-generated summary
Mustafa N. Mithaiwala's research focuses on the intersection of neuroinflammation, substance use disorders, and neurodegenerative diseases. His work investigates how factors such as family history of substance use and stressful life events impact adolescent brain development, specifically the maturation of cerebral white matter. Mithaiwala also studies the role of the kynurenine pathway in central nervous system diseases and its potential as a therapeutic target. His research has explored the relationship between immune challenges, genetic polymorphisms like the Val66Met BDNF variant, and neuroimmune responses, including anhedonia-like behaviors. He has collaborated with researchers at the University of Arkansas for Medical Sciences on multiple publications.
Metrics
- h-index: 2
- Publications: 8
- Citations: 190
Selected Publications
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Integrated Family History Measures Identify Brain Structural Differences Associated with Risk for Alcohol and Other Substance Use Disorders (2026)
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KMO deletion preserves non-associative learning and SVZ neurogenesis in aging mice (2026)
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Family History of Substance Use and Stressful Life Events Impact Adolescent Maturation of Cerebral White Matter (2025)
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Dysregulated Neuroimmune and Anhedonia-like Behavioral Response Following Peripheral Immune Challenge in Mice Carrying the Val66Met Brain-Derived Neurotrophic Factor Polymorphism (2025)
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KMO Deletion Preserves Non-Associative Learning and SVZ Neurogenesis in Aging Mice (2025)
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Increased white blood cell in young adults with family histories of alcohol and other substance use disorders (2024)
Collaboration Network
Top Collaborators
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- KMO Deletion Preserves Non-Associative Learning and SVZ Neurogenesis in Aging Mice
- Dysregulated Neuroimmune and Anhedonia-like Behavioral Response Following Peripheral Immune Challenge in Mice Carrying the Val66Met Brain-Derived Neurotrophic Factor Polymorphism
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- Dysregulated Neuroimmune and Anhedonia-like Behavioral Response Following Peripheral Immune Challenge in Mice Carrying the Val66Met Brain-Derived Neurotrophic Factor Polymorphism
- Family History of Substance Use and Stressful Life Events Impact Adolescent Maturation of Cerebral White Matter
- KMO Deletion Preserves Non-Associative Learning and SVZ Neurogenesis in Aging Mice
- Dysregulated Neuroimmune and Anhedonia-like Behavioral Response Following Peripheral Immune Challenge in Mice Carrying the Val66Met Brain-Derived Neurotrophic Factor Polymorphism
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- KMO Deletion Preserves Non-Associative Learning and SVZ Neurogenesis in Aging Mice
- KMO Deletion Preserves Non-Associative Learning and SVZ Neurogenesis in Aging Mice
- KMO Deletion Preserves Non-Associative Learning and SVZ Neurogenesis in Aging Mice
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