Mustafa N. Mithaiwala
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Also affiliated: The University of Texas at San Antonio Health Science Center (2021–2026)
Formerly Arkansas Postdoctoral Research Fellow, UAMS through 2026.
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Mustafa N. Mithaiwala's research focuses on the intersection of neuroinflammation, substance use disorders, and neurodegenerative diseases. His work investigates how factors such as family history of substance use and stressful life events impact adolescent brain development, specifically the maturation of cerebral white matter. Mithaiwala also studies the role of the kynurenine pathway in central nervous system diseases and its potential as a therapeutic target. His research has explored the relationship between immune challenges, genetic polymorphisms like the Val66Met BDNF variant, and neuroimmune responses, including anhedonia-like behaviors. He has collaborated with researchers at the University of Arkansas for Medical Sciences on multiple publications.
Metrics
- h-index: 2
- Publications: 8
- Citations: 197
Positions
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Postdoctoral Research Fellow 2023–2026University of Arkansas for Medical Sciences Psychiatry and Behavioral Sciences ORCID
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Postdoctoral Research Fellow 2022–2023The University of Texas Health Science Center at San Antonio Pharmacology ORCID
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Graduate Research Assistant 2016–2022The University of Texas Health Science Center at San Antonio Department of Pharmacology Pharmacology ORCID
Selected Publications
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Integrated Family History Measures Identify Brain Structural Differences Associated with Risk for Alcohol and Other Substance Use Disorders (2026)
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KMO deletion preserves non-associative learning and SVZ neurogenesis in aging mice (2026)
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Family History of Substance Use and Stressful Life Events Impact Adolescent Maturation of Cerebral White Matter (2025)
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Dysregulated Neuroimmune and Anhedonia-like Behavioral Response Following Peripheral Immune Challenge in Mice Carrying the Val66Met Brain-Derived Neurotrophic Factor Polymorphism (2025)
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KMO Deletion Preserves Non-Associative Learning and SVZ Neurogenesis in Aging Mice (2025)
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Increased white blood cell in young adults with family histories of alcohol and other substance use disorders (2024)
Collaboration Network
Top Collaborators
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- Family History of Substance Use and Stressful Life Events Impact Adolescent Maturation of Cerebral White Matter
- Dysregulated Neuroimmune and Anhedonia-like Behavioral Response Following Peripheral Immune Challenge in Mice Carrying the Val66Met Brain-Derived Neurotrophic Factor Polymorphism
- Integrated Family History Measures Identify Brain Structural Differences Associated with Risk for Alcohol and Other Substance Use Disorders
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- KMO Deletion Preserves Non-Associative Learning and SVZ Neurogenesis in Aging Mice
- Dysregulated Neuroimmune and Anhedonia-like Behavioral Response Following Peripheral Immune Challenge in Mice Carrying the Val66Met Brain-Derived Neurotrophic Factor Polymorphism
- KMO Deletion Preserves Non-Associative Learning and SVZ Neurogenesis in Aging Mice
- Dysregulated Neuroimmune and Anhedonia-like Behavioral Response Following Peripheral Immune Challenge in Mice Carrying the Val66Met Brain-Derived Neurotrophic Factor Polymorphism
- KMO deletion preserves non-associative learning and SVZ neurogenesis in aging mice
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- Integrated Family History Measures Identify Brain Structural Differences Associated with Risk for Alcohol and Other Substance Use Disorders
- KMO Deletion Preserves Non-Associative Learning and SVZ Neurogenesis in Aging Mice
- KMO deletion preserves non-associative learning and SVZ neurogenesis in aging mice
- KMO Deletion Preserves Non-Associative Learning and SVZ Neurogenesis in Aging Mice
- KMO deletion preserves non-associative learning and SVZ neurogenesis in aging mice
- KMO Deletion Preserves Non-Associative Learning and SVZ Neurogenesis in Aging Mice
- KMO deletion preserves non-associative learning and SVZ neurogenesis in aging mice
- KMO Deletion Preserves Non-Associative Learning and SVZ Neurogenesis in Aging Mice
- KMO deletion preserves non-associative learning and SVZ neurogenesis in aging mice
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- Increased white blood cell in young adults with family histories of alcohol and other substance use disorders
- KMO Deletion Preserves Non-Associative Learning and SVZ Neurogenesis in Aging Mice
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