Nicole L. Ward
Professor and Vice-Chair Basic Research
faculty
Baccalaureate Education, College of Nursing
Research Areas
Biography and Research Information
OverviewAI-generated summary
Nicole L. Ward's research focuses on understanding the mechanisms of inflammatory skin diseases, particularly psoriasis. Her work investigates the cellular and molecular pathways involved in disease pathogenesis, utilizing advanced techniques such as single-cell and spatial sequencing to identify how keratinocytes and fibroblasts contribute to inflammatory amplification. She has also explored the relationship between psoriasis and cardiovascular risk in patients.
Ward's publications include reviews on animal models of psoriasis and the role of specific enzymes and cytokines, such as Phospholipase A2 and IFN-κ, in the development of psoriatic inflammation. Her research group at the University of Arkansas for Medical Sciences collaborates with other researchers, including Leslie McCormack, Natalie Capps, Beverly English, and Keneshia Bryant-Moore, on projects related to these conditions.
Her scholarly contributions are recognized by a high h-index of 46, with over 164 publications and 6,666 citations, designating her as a highly cited researcher. Ward also holds a leadership position as Professor and Vice-Chair for Basic Research in the College of Nursing at the University of Arkansas for Medical Sciences, contributing to the educational and research endeavors within the institution.
Metrics
- h-index: 46
- Publications: 164
- Citations: 6,666
Selected Publications
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Care interrupted: a mixed-methods study of lapse in primary care and cardiology services for women with CHD (2025)
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jabbrv-ltwa-all.ldf jabbrv-ltwa-en.ldf Her Heart, Her Health: Lived Experiences of Healthcare Transitions and Unmet Women’s Health Needs in Adult Females with Congenital Heart Disease (2025)
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Screening and Treatment Guidelines Addressing Adolescent Vaping in Primary Care (2023)
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Motherhood Together: Effects of an Adapted Prenatal Curriculum on Mother and Infant Outcomes. (2022)
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Remote Virtual Simulation With Prelicensure Nursing Students During Rapid Course Transition (2022)
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Evaluation of a simulation-based learning experience using a prenatal telehealth scenario with prelicensure nursing students (2022)
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Piloting a high-fidelity postpartum hemorrhage simulation with prelicensure nursing students: Evaluating knowledge, confidence, and satisfaction (2021)
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Maintaining Academic Performance and Student Satisfaction during the Remote Transition of a Nursing Obstetrics Course to Online Instruction (2021)
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The development and implementation of an advanced practice registered nurse–led prenatal education program for housing insecure women (2021)
Collaboration Network
Top Collaborators
- Mouse Models of Psoriasis: A Comprehensive Review
- Phospholipase A2 enzymes represent a shared pathogenic pathway in psoriasis and pityriasis rubra pilaris
- New Frontiers in Psoriatic Disease Research, Part I: Genetics, Environmental Triggers, Immunology, Pathophysiology, and Precision Medicine
- IL-17-induced dimerization of IL-17RA drives the formation of the IL-17 signalosome to potentiate signaling
- IFN-κ Is a Rheostat for Development of Psoriasiform Inflammation
Showing 5 of 13 shared publications
- Single cell and spatial sequencing define processes by which keratinocytes and fibroblasts amplify inflammatory responses in psoriasis
- Phospholipase A2 enzymes represent a shared pathogenic pathway in psoriasis and pityriasis rubra pilaris
- IFN-κ Is a Rheostat for Development of Psoriasiform Inflammation
- IRAK2 Has a Critical Role in Promoting Feed-Forward Amplification of Epidermal Inflammatory Responses
- Suppression of TCF4 promotes a ZC3H12A-mediated self-sustaining inflammatory feedback cycle involving IL-17RA/IL-17RE epidermal signaling
Showing 5 of 10 shared publications
- Maintaining Academic Performance and Student Satisfaction during the Remote Transition of a Nursing Obstetrics Course to Online Instruction
- Piloting a high-fidelity postpartum hemorrhage simulation with prelicensure nursing students: Evaluating knowledge, confidence, and satisfaction
- Current Practice to Address Adolescent Vaping in Primary Care
- Evaluation of a simulation-based learning experience using a prenatal telehealth scenario with prelicensure nursing students
- The development and implementation of an advanced practice registered nurse–led prenatal education program for housing insecure women
Showing 5 of 9 shared publications
- Single cell and spatial sequencing define processes by which keratinocytes and fibroblasts amplify inflammatory responses in psoriasis
- Phospholipase A2 enzymes represent a shared pathogenic pathway in psoriasis and pityriasis rubra pilaris
- IL-17-induced dimerization of IL-17RA drives the formation of the IL-17 signalosome to potentiate signaling
- IRAK2 Has a Critical Role in Promoting Feed-Forward Amplification of Epidermal Inflammatory Responses
- Suppression of TCF4 promotes a ZC3H12A-mediated self-sustaining inflammatory feedback cycle involving IL-17RA/IL-17RE epidermal signaling
Showing 5 of 8 shared publications
- Single cell and spatial sequencing define processes by which keratinocytes and fibroblasts amplify inflammatory responses in psoriasis
- Phospholipase A2 enzymes represent a shared pathogenic pathway in psoriasis and pityriasis rubra pilaris
- New Frontiers in Psoriatic Disease Research, Part I: Genetics, Environmental Triggers, Immunology, Pathophysiology, and Precision Medicine
- IRAK2 Has a Critical Role in Promoting Feed-Forward Amplification of Epidermal Inflammatory Responses
- Suppression of TCF4 promotes a ZC3H12A-mediated self-sustaining inflammatory feedback cycle involving IL-17RA/IL-17RE epidermal signaling
Showing 5 of 7 shared publications
- Single cell and spatial sequencing define processes by which keratinocytes and fibroblasts amplify inflammatory responses in psoriasis
- Phospholipase A2 enzymes represent a shared pathogenic pathway in psoriasis and pityriasis rubra pilaris
- IRAK2 Has a Critical Role in Promoting Feed-Forward Amplification of Epidermal Inflammatory Responses
- Suppression of TCF4 promotes a ZC3H12A-mediated self-sustaining inflammatory feedback cycle involving IL-17RA/IL-17RE epidermal signaling
- VGLL3-centered network connects placental, vascular, and immune defects in preeclampsia
Showing 5 of 6 shared publications
- Single cell and spatial sequencing define processes by which keratinocytes and fibroblasts amplify inflammatory responses in psoriasis
- Phospholipase A2 enzymes represent a shared pathogenic pathway in psoriasis and pityriasis rubra pilaris
- IFN-κ Is a Rheostat for Development of Psoriasiform Inflammation
- IRAK2 Has a Critical Role in Promoting Feed-Forward Amplification of Epidermal Inflammatory Responses
- Suppression of TCF4 promotes a ZC3H12A-mediated self-sustaining inflammatory feedback cycle involving IL-17RA/IL-17RE epidermal signaling
Showing 5 of 6 shared publications
- Maintaining Academic Performance and Student Satisfaction during the Remote Transition of a Nursing Obstetrics Course to Online Instruction
- Piloting a high-fidelity postpartum hemorrhage simulation with prelicensure nursing students: Evaluating knowledge, confidence, and satisfaction
- The development and implementation of an advanced practice registered nurse–led prenatal education program for housing insecure women
- Remote Virtual Simulation With Prelicensure Nursing Students During Rapid Course Transition
- Motherhood Together: Effects of an Adapted Prenatal Curriculum on Mother and Infant Outcomes.
- Single cell and spatial sequencing define processes by which keratinocytes and fibroblasts amplify inflammatory responses in psoriasis
- Phospholipase A2 enzymes represent a shared pathogenic pathway in psoriasis and pityriasis rubra pilaris
- New Frontiers in Psoriatic Disease Research, Part I: Genetics, Environmental Triggers, Immunology, Pathophysiology, and Precision Medicine
- IRAK2 Has a Critical Role in Promoting Feed-Forward Amplification of Epidermal Inflammatory Responses
- Suppression of TCF4 promotes a ZC3H12A-mediated self-sustaining inflammatory feedback cycle involving IL-17RA/IL-17RE epidermal signaling
- Single cell and spatial sequencing define processes by which keratinocytes and fibroblasts amplify inflammatory responses in psoriasis
- Phospholipase A2 enzymes represent a shared pathogenic pathway in psoriasis and pityriasis rubra pilaris
- IFN-κ Is a Rheostat for Development of Psoriasiform Inflammation
- Suppression of TCF4 promotes a ZC3H12A-mediated self-sustaining inflammatory feedback cycle involving IL-17RA/IL-17RE epidermal signaling
- VGLL3-centered network connects placental, vascular, and immune defects in preeclampsia
- Phospholipase A2 enzymes represent a shared pathogenic pathway in psoriasis and pityriasis rubra pilaris
- IRAK2 Has a Critical Role in Promoting Feed-Forward Amplification of Epidermal Inflammatory Responses
- Suppression of TCF4 promotes a ZC3H12A-mediated self-sustaining inflammatory feedback cycle involving IL-17RA/IL-17RE epidermal signaling
- VGLL3-centered network connects placental, vascular, and immune defects in preeclampsia
- Single cell and spatial sequencing define processes by which keratinocytes and fibroblasts amplify inflammatory responses in psoriasis
- Phospholipase A2 enzymes represent a shared pathogenic pathway in psoriasis and pityriasis rubra pilaris
- IRAK2 Has a Critical Role in Promoting Feed-Forward Amplification of Epidermal Inflammatory Responses
- Retrospective pharmacogenetic study of psoriasis highlights the role of <i>KLK7</i> in tumour necrosis factor signalling
- Maintaining Academic Performance and Student Satisfaction during the Remote Transition of a Nursing Obstetrics Course to Online Instruction
- Piloting a high-fidelity postpartum hemorrhage simulation with prelicensure nursing students: Evaluating knowledge, confidence, and satisfaction
- Remote Virtual Simulation With Prelicensure Nursing Students During Rapid Course Transition
- Phospholipase A2 enzymes represent a shared pathogenic pathway in psoriasis and pityriasis rubra pilaris
- IRAK2 Has a Critical Role in Promoting Feed-Forward Amplification of Epidermal Inflammatory Responses
- Suppression of TCF4 promotes a ZC3H12A-mediated self-sustaining inflammatory feedback cycle involving IL-17RA/IL-17RE epidermal signaling
- Single cell and spatial sequencing define processes by which keratinocytes and fibroblasts amplify inflammatory responses in psoriasis
- IRAK2 Has a Critical Role in Promoting Feed-Forward Amplification of Epidermal Inflammatory Responses
- Suppression of TCF4 promotes a ZC3H12A-mediated self-sustaining inflammatory feedback cycle involving IL-17RA/IL-17RE epidermal signaling
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