Nukhet Aykin-Burns
Sourced from institutional research profiles (UAMS TRI or ARA).
Role not yet determined
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Nukhet Aykin-Burns studies the impact of environmental pollutants, specifically per- and polyfluoroalkyl substances (PFAS), on liver health and mitochondrial function. Her work investigates how these "forever chemicals" can disrupt cellular processes, potentially leading to conditions like hepatocellular carcinoma. Aykin-Burns also explores therapeutic interventions, such as the use of SS-31 peptide to mitigate radiation-induced cellular damage and the beneficial effects of indole derivatives in breast cancer treatment. Her research collaborations include work with C. Godwin, L. Evans, F. LoBianco, and Kimberly J. Krager at the University of Arkansas for Medical Sciences and University of Arkansas at Fayetteville.
Metrics
- h-index: 1
- Publications: 2
- Citations: 12
Selected Publications
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Mitochondrial-targeted SS-31 peptide attenuates radiation-induced cardiomyocyte senescence (2026)
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Beneficial effects of an indole derivative melatonin analogue in estrogen receptor positive breast cancer (2026)
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Hepatocellular carcinoma’s inflammatory secret: ferroptosis as the key (2026)
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Forever Chemicals, Finite Defenses: PFAS Burden the Liver, Break Mitochondria, and Outpace Modern Regulation (2026)
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Abstract No. 106 Where are Women in Academic Interventional Radiology? (2023)
Grants & Funding
As listed on this researcher's institutional profile.
- Photobiomodulation to Ameliorate Neuronal Degeneration and Cognitive Decline after Mixed Field Irradiation - Continuation National Aeronautics & Space Administration
- Center for Studies of Host Response to Cancer Therapy NIH
- Sirt3: A Key Player Against PCB-Induced Mitochondrial Injury NIH
- MnBuOE as a Novel Chemosensitizer for Breast Cancer and Neuroprotector NIH
- The Role of Mitochondrial Reactive Oxygen Species and Sirtuin3 in Radiation-Induced Liver Damage Arkansas Science & Technology Authority
- Ryanodine Receptors as Therapeutic Targets to Prevent Doxorubicin-Induced Lymphatic Dysfunction NIH/Nat. Cancer Institute
Collaboration Network
Top Collaborators
- Abstract No. 106 Where are Women in Academic Interventional Radiology?
- Abstract No. 106 Where are Women in Academic Interventional Radiology?
- Abstract No. 106 Where are Women in Academic Interventional Radiology?
- Abstract No. 106 Where are Women in Academic Interventional Radiology?
- Abstract No. 106 Where are Women in Academic Interventional Radiology?
- Forever Chemicals, Finite Defenses: PFAS Burden the Liver, Break Mitochondria, and Outpace Modern Regulation
- Forever Chemicals, Finite Defenses: PFAS Burden the Liver, Break Mitochondria, and Outpace Modern Regulation
- Hepatocellular carcinoma’s inflammatory secret: ferroptosis as the key
- Hepatocellular carcinoma’s inflammatory secret: ferroptosis as the key
- Beneficial effects of an indole derivative melatonin analogue in estrogen receptor positive breast cancer
- Beneficial effects of an indole derivative melatonin analogue in estrogen receptor positive breast cancer
- Beneficial effects of an indole derivative melatonin analogue in estrogen receptor positive breast cancer
- Beneficial effects of an indole derivative melatonin analogue in estrogen receptor positive breast cancer
- Beneficial effects of an indole derivative melatonin analogue in estrogen receptor positive breast cancer
- Mitochondrial-targeted SS-31 peptide attenuates radiation-induced cardiomyocyte senescence
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