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Research Areas
Biography and Research Information
OverviewAI-generated summary
Thomas M. Badger's research focuses on the biological and health impacts of diet and environmental factors, particularly in animal models and human studies. He has investigated the effects of ethanol consumption on hepatocarcinogenesis in male mice, examining the role of the Wnt/β-Catenin signaling pathway. His work also includes studies on the potential hormonal effects and reproductive toxicity of soy-based formulas in male piglets, finding no evidence of estrogenicity or reproductive harm.
In human health, Badger's research has explored the associations between maternal depressive symptoms during pregnancy and newborn brain functional connectivity. He has also examined the relationship between white matter microstructures and cognitive functioning in children, utilizing track-weighted imaging. Further studies have investigated correlations between sleep disturbance and brain cortical morphometry, as well as cortical asymmetry and cognitive functions in healthy children. His research group has also conducted longitudinal observational studies on skeletal development in children fed different infant formulas or human milk.
Badger holds a high-impact researcher designation, evidenced by his h-index of 71 and over 15,000 citations across more than 400 publications. He actively collaborates with researchers from the National Center for Toxicological Research and the University of Arkansas for Medical Sciences, including Kelly E. Mercer, Rebecca Wynne, Xiawei Ou, and Xiaoxu Na.
Metrics
- h-index: 71
- Publications: 425
- Citations: 15,543
Selected Publications
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Associations between mother’s depressive symptoms during pregnancy and newborn’s brain functional connectivity (2023)
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A longitudinal observational study of skeletal development between ages 3 mo and 6 y in children fed human milk, milk formula, or soy formula (2023)
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Soy Formula Is Not Estrogenic and Does Not Result in Reproductive Toxicity in Male Piglets: Results from a Controlled Feeding Study (2022)
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Associations Between White Matter Microstructures and Cognitive Functioning in 8-Year-Old Children: A Track-Weighted Imaging Study (2022)
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Track-weighted imaging analysis of white matter microstructures in healthy children: Sex and hemispheric differences (2022)
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Associations between Cortical Asymmetry and Domain Specific Cognitive Functions in Healthy Children (2021)
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Correlations between sleep disturbance and brain cortical morphometry in healthy children (2021)
Grants & Funding
Collaboration Network
Top Collaborators
Martin J. J. Ronis
7 shared publications
- Soy Formula Is Not Estrogenic and Does Not Result in Reproductive Toxicity in Male Piglets: Results from a Controlled Feeding Study
- Supplementary Table 2 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Supplementary Table 1 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Data from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Supplementary Table 2 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
Showing 5 of 7 shared publications
Leah Hennings
7 shared publications
- Soy Formula Is Not Estrogenic and Does Not Result in Reproductive Toxicity in Male Piglets: Results from a Controlled Feeding Study
- Supplementary Table 2 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Supplementary Table 1 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Data from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Supplementary Table 2 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
Showing 5 of 7 shared publications
Kelly E. Mercer
7 shared publications
In database
- Soy Formula Is Not Estrogenic and Does Not Result in Reproductive Toxicity in Male Piglets: Results from a Controlled Feeding Study
- Supplementary Table 2 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Supplementary Table 1 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Data from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Supplementary Table 2 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
Showing 5 of 7 shared publications
Keith Lai
6 shared publications
- Supplementary Table 2 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Supplementary Table 1 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Data from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Supplementary Table 2 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Supplementary Table 1 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
Showing 5 of 6 shared publications
Mario A. Cleves
6 shared publications
- Supplementary Table 2 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Supplementary Table 1 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Data from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Supplementary Table 2 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Supplementary Table 1 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
Showing 5 of 6 shared publications
Rebecca Wynne
6 shared publications
In database
- Supplementary Table 2 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Supplementary Table 1 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Data from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Supplementary Table 2 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Supplementary Table 1 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
Showing 5 of 6 shared publications
Xiaoxu Na
University of Arkansas for Medical Sciences
5 shared publications
In database
- Associations between mother’s depressive symptoms during pregnancy and newborn’s brain functional connectivity
- Correlations between sleep disturbance and brain cortical morphometry in healthy children
- Associations Between White Matter Microstructures and Cognitive Functioning in 8-Year-Old Children: A Track-Weighted Imaging Study
- Associations between Cortical Asymmetry and Domain Specific Cognitive Functions in Healthy Children
- Track-weighted imaging analysis of white matter microstructures in healthy children: Sex and hemispheric differences
Xiawei Ou
University of Arkansas for Medical Sciences
5 shared publications
In database
- Associations between mother’s depressive symptoms during pregnancy and newborn’s brain functional connectivity
- Correlations between sleep disturbance and brain cortical morphometry in healthy children
- Associations Between White Matter Microstructures and Cognitive Functioning in 8-Year-Old Children: A Track-Weighted Imaging Study
- Associations between Cortical Asymmetry and Domain Specific Cognitive Functions in Healthy Children
- Track-weighted imaging analysis of white matter microstructures in healthy children: Sex and hemispheric differences
Charles M. Glasier
University of Arkansas for Medical Sciences
4 shared publications
In database
- Associations between mother’s depressive symptoms during pregnancy and newborn’s brain functional connectivity
- Associations Between White Matter Microstructures and Cognitive Functioning in 8-Year-Old Children: A Track-Weighted Imaging Study
- Associations between Cortical Asymmetry and Domain Specific Cognitive Functions in Healthy Children
- Track-weighted imaging analysis of white matter microstructures in healthy children: Sex and hemispheric differences
Neha Sharma
4 shared publications
- Supplementary Table 2 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Supplementary Table 1 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Data from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Data from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
Rajikha Raja
University of Arkansas for Medical Sciences (US)
3 shared publications
- Associations Between White Matter Microstructures and Cognitive Functioning in 8-Year-Old Children: A Track-Weighted Imaging Study
- Associations between Cortical Asymmetry and Domain Specific Cognitive Functions in Healthy Children
- Track-weighted imaging analysis of white matter microstructures in healthy children: Sex and hemispheric differences
Jayne Bellando
University of Arkansas for Medical Sciences
2 shared publications
In database
- Associations between mother’s depressive symptoms during pregnancy and newborn’s brain functional connectivity
- Associations between Cortical Asymmetry and Domain Specific Cognitive Functions in Healthy Children
Neha Sharma
2 shared publications
- Supplementary Table 2 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
- Supplementary Table 1 from Alcohol Consumption Promotes Diethylnitrosamine-Induced Hepatocarcinogenesis in Male Mice through Activation of the Wnt/β-Catenin Signaling Pathway
Aline Andres
Arkansas Children's Hospital
2 shared publications
In database
- Associations between mother’s depressive symptoms during pregnancy and newborn’s brain functional connectivity
- A longitudinal observational study of skeletal development between ages 3 mo and 6 y in children fed human milk, milk formula, or soy formula
Ting Li
University of Arkansas for Medical Sciences
1 shared publication
In database
- Correlations between sleep disturbance and brain cortical morphometry in healthy children
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