Research Areas
Links
Biography and Research Information
OverviewAI-generated summary
U Thaung Shwe's research investigates the role of canonical transient receptor potential (TRPC) channels in neurological disorders, particularly epilepsy. His work examines how specific TRPC channel subtypes, such as TRPC3 and TRPC1/4, contribute to hippocampal hyperexcitability and neuronal cell death following seizures. Recent publications by Shwe and collaborators have explored the increased susceptibility to pilocarpine-induced status epilepticus in TRPC1/4 double knockout mice, suggesting a protective role for these channels in seizure regulation. Further studies have delved into the pharmacological distinctions between native homomeric TRPC4 channels and heteromeric TRPC1/4 channels in lateral septal neurons. Shwe's scholarship metrics include an h-index of 5, with 8 total publications and 399 citations. He has collaborated with Fang Zheng and Kevin D. Phelan at the University of Arkansas for Medical Sciences on multiple publications.
Metrics
- h-index: 6
- Publications: 8
- Citations: 404
Selected Publications
-
Investigating Contributions of Canonical Transient Receptor Potential Channel 3 to Hippocampal Hyperexcitability and Seizure-Induced Neuronal Cell Death (2024)
-
Increased Susceptibility to Pilocarpine-Induced Status Epilepticus and Reduced Latency in TRPC1/4 Double Knockout Mice (2023)
-
Pharmacological Differences between Native Homomeric Transient Receptor Potential Canonical Type 4 Channels and Heteromeric Transient Receptor Potential Canonical Type 1/4 Channels in Lateral Septal Neurons (2023)
Collaboration Network
Top Collaborators
- Pharmacological Differences between Native Homomeric Transient Receptor Potential Canonical Type 4 Channels and Heteromeric Transient Receptor Potential Canonical Type 1/4 Channels in Lateral Septal Neurons
- Investigating Contributions of Canonical Transient Receptor Potential Channel 3 to Hippocampal Hyperexcitability and Seizure-Induced Neuronal Cell Death
- Increased Susceptibility to Pilocarpine-Induced Status Epilepticus and Reduced Latency in TRPC1/4 Double Knockout Mice
- Pharmacological Differences between Native Homomeric Transient Receptor Potential Canonical Type 4 Channels and Heteromeric Transient Receptor Potential Canonical Type 1/4 Channels in Lateral Septal Neurons
- Investigating Contributions of Canonical Transient Receptor Potential Channel 3 to Hippocampal Hyperexcitability and Seizure-Induced Neuronal Cell Death
- Increased Susceptibility to Pilocarpine-Induced Status Epilepticus and Reduced Latency in TRPC1/4 Double Knockout Mice
- Investigating Contributions of Canonical Transient Receptor Potential Channel 3 to Hippocampal Hyperexcitability and Seizure-Induced Neuronal Cell Death
Similar Researchers
Based on overlapping research topics