Match tier
Listed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-09
V
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Vivek Khanal's research focuses on the impact of diet on bone health and the underlying molecular mechanisms. His work investigates how high-fat and high-carbohydrate diets can increase bone fragility, identifying the transforming growth factor-beta (TGF-β) signaling pathway as a key regulator of osteocyte function in this process. Khanal also studies the role of lipocalin-2 in osteocytes, exploring its regulation of bone formation through ferroptosis and Wnt signaling pathways. His research extends to autophagy's essential role in bone development, growth, and maintenance. Additionally, Khanal examines global transcriptional profiles of adipose tissue to uncover sex-dependent mechanisms in acute obesity and their implications for interorgan crosstalk. His collaborators at the University of Arkansas for Medical Sciences include Neha S. Dole, Madeline Carroll, Amy Y. Sato, and Ryan M. Allen.
Metrics
- h-index: 2
- Publications: 5
- Citations: 21
Positions
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Postdoctoral Fellow publications 2024–2026University of Arkansas for Medical Sciences Institution web page
Selected Publications
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Autophagy in Osx1-Cre-targeted cells is essential for development, growth, and maintenance of bone (2026)
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Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression (2026)
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Global Transcriptional Profiling of Adipose Tissue Uncovers Unique Sex-Dependent Mechanisms in Acute Obesity: Implications for Adipose-Heart Interorgan Crosstalk (2025)
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Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation (2025)
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High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function (2024)
Collaboration Network
Top Collaborators
Neha S. Dole
University of Arkansas for Medical Sciences
5 shared publications
In database
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
- Global Transcriptional Profiling of Adipose Tissue Uncovers Unique Sex-Dependent Mechanisms in Acute Obesity: Implications for Adipose-Heart Interorgan Crosstalk
- Autophagy in Osx1-Cre-targeted cells is essential for development, growth, and maintenance of bone
Madeline D. Carroll
University of Arkansas for Medical Sciences
3 shared publications
In database
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
Jayden Carter
University of Arkansas for Medical Sciences
2 shared publications
In database
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
Amy Y. Sato
University of Arkansas for Medical Sciences
2 shared publications
In database
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
Ryan M. Allen
University of Arkansas for Medical Sciences
2 shared publications
In database
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
Umesh D. Wankhade
Arkansas Children's Nutrition Center
2 shared publications
In database
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression
Andrés Betancourt-Torres
University of California, San Francisco (US)
1 shared publication
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
Serra Kaya
University of California, San Francisco (US)
1 shared publication
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
Yoshihiro Obata
University of Utah (US)
1 shared publication
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
Charles A. Schurman
University of California, San Francisco (US)
1 shared publication
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
Jihee Yoon
University of California, San Francisco (US)
1 shared publication
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
Cristal S. Yee
University of California, San Francisco (US)
1 shared publication
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
Clarissa Aguirre Luna
University of California, San Francisco (US)
1 shared publication
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
Jennifer J. Salinas
University of California, San Francisco (US)
1 shared publication
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
Elizabeth Miclau
University of California, San Francisco (US)
1 shared publication
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
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