Biography and Research Information
OverviewAI-generated summary
Vivek Khanal investigates the molecular mechanisms underlying metabolic disorders and their impact on bone health and interorgan communication. His recent publications focus on how high-fat and high-carbohydrate diets affect bone fragility by influencing osteocyte function through TGF-β signaling. Khanal also studies the role of lipocalin-2 in regulating osteocyte ferroptosis and its crosstalk with osteoblasts via Wnt signaling, which impacts bone formation. Additionally, his research examines the global transcriptional profiles of adipose tissue to uncover sex-dependent mechanisms in acute obesity and their implications for adipose-heart interorgan crosstalk. Khanal has authored four publications with 14 citations and an h-index of 1. He collaborates with researchers at the University of Arkansas for Medical Sciences, including Neha S. Dole, Madeline Carroll, Amy Y. Sato, and Ryan M. Allen.
Metrics
- h-index: 1
- Publications: 4
- Citations: 16
Selected Publications
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Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression (2026)
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Global Transcriptional Profiling of Adipose Tissue Uncovers Unique Sex-Dependent Mechanisms in Acute Obesity: Implications for Adipose-Heart Interorgan Crosstalk (2025)
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Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation (2025)
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High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function (2024)
Collaboration Network
Top Collaborators
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Global Transcriptional Profiling of Adipose Tissue Uncovers Unique Sex-Dependent Mechanisms in Acute Obesity: Implications for Adipose-Heart Interorgan Crosstalk
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- High-fat and high-carbohydrate diets increase bone fragility through TGF-β–dependent control of osteocyte function
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
- Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation
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