Ying Fu
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Also affiliated: South China Agricultural University (2016–2020); Beijing Institute of Technology (2025); Brigham and Women's Hospital (1998); Ford Foundation (2016); Universiti Sains Malaysia (2024); Central South University (2017); Nanchang University (2020–2024); Northwestern Polytechnical University (2006–2020); Shanghai Jiao Tong University (2005–2025); Georgetown University (2015); Guangdong University of Foreign Studies (2024); South China Normal University (2009–2011); Bloomsburg University (2017); Chengde Medical University (2024–2025); Chinese Academy of Sciences (2013–2023); Hainan University (2023–2024); Ministry of Natural Resources (2021); Fudan University (2025); China University of Mining and Technology (2021–2022); Wenzhou Medical University (2008); Discovery Institute (2024); Georgetown University Medical Center (2015); Australian Regenerative Medicine Institute (2024–2025); Sir Run Run Shaw Hospital (2012–2025); Chang Gung Memorial Hospital (2025); First Affiliated Hospital of Jiangxi Medical College (2024); Zhejiang Provincial Institute of Communications Planning,Design & Research (2025); Key Laboratory of Guangdong Province (2016–2018); Research Support Instruments (United States) (2025); People's Liberation Army No. 150 Hospital (2025); Seva Mandir (2016); Shenzhen Center for Disease Control and Prevention (2025); Shanghai Cancer Institute (2017); Chengde Medical University (2024–2025); Beijing Luhe Hospital Affiliated to Capital Medical University (2017–2021); Linkou Chang Gung Memorial Hospital (2025); Dana-Farber Cancer Institute (1998); Zhejiang Environmental Monitoring Center (2024–2025); Yunnan Water Conservancy and Hydropower Survey and Design Institute (2024); Institute of Microbiology (2020–2023); State Key Laboratory of Oncogene and Related Genes (2017); Third Affiliated Hospital of Zhengzhou University (2025); Shenzhen Luohu People's Hospital (2023–2025); Ministry of Water Resources of the People's Republic of China (2021); Zhejiang Taizhou Hospital (2008); Institute of Geographic Sciences and Natural Resources Research (2013–2014); Qilu Hospital of Shandong University (2016–2023); Dana-Farber Brigham Cancer Center (1998); Zhejiang Medicine (China) (2025); Shanghai Institute of Pharmaceutical Industry (2024); University of Chinese Academy of Sciences (2013–2014); First Affiliated Hospital Zhejiang University (2015); PRG S&Tech (South Korea) (2025); Huazhong University of Science and Technology (2009–2012); Zhejiang University of Technology (2022–2024); Monash University (2024–2025); Ocean University of China (2005); The University of Tokyo (2025); Zhejiang University (2012–2025); Southwestern University (2024); University of Oklahoma (2018–2024); Capital Normal University (2015–2017)
Upstream record may be merged OpenAlex, the source of these figures, lists 63 institutions in 7 countries for this author record — a pattern that usually means it combines several researchers with similar names. The totals above may include work by other people.
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Ying Fu's research program investigates the genetic and environmental factors influencing complex traits, with a particular focus on gene-environment interactions and the underlying molecular mechanisms. This work is supported by multiple federal grants from the National Institutes of Health (NIH), totaling over $1.4 million. These grants fund investigations into the mechanobiology of multicellular morphogenesis, the development of advanced microscopy techniques for studying 3D cellular structures, and the characterization of mechanically heterogeneous cell populations. Additionally, funding supports research into cryptic genetic variation in *Drosophila* and the decoding of gene-environment interactions through multi-omics approaches.
Fu's scholarly contributions include a significant body of work on microbial communities and antibiotic resistance. Publications examine the impact of climate warming on grassland soil microbial diversity and the evolution and dissemination of multidrug-resistant bacteria, including *Acinetobacter baumannii* and *Klebsiella pneumoniae*, in China. Fu's scholarship metrics reflect a prolific research career, with an h-index of 34, 197 total publications, and over 4,200 citations. Fu also collaborates with researchers at the University of Arkansas at Fayetteville, including Xiaolun Sun and Tahrir Alenezi.
Metrics
- h-index: 34
- Publications: 197
- Citations: 4,296
Selected Publications
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The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid (2025)
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Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence (2024)
Federal Grants 5 $1,433,308 total
Investigating the mechanobiology of multicellular morphogenesis
Decoding Gene-Environment Interactions in Complex Traits via Multi-Omics Approaches
Dissecting cryptic genetic variation underlying complex traits in Drosophila
Developing branch stress microscopy for the mechanobiology of 3D morphogenesis and invasive diseases
Collaboration Network
Top Collaborators
- A secondary bile acid from microbiota metabolism attenuates ileitis and bile acid reduction in subclinical necrotic enteritis in chickens
- Clostridium perfringens-Induced Necrotic Diseases: An Overview
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
Showing 5 of 9 shared publications
- Clostridium perfringens-Induced Necrotic Diseases: An Overview
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- Microbiota attenuates chicken transmission-exacerbated campylobacteriosis in Il10−/− mice
Showing 5 of 8 shared publications
- A secondary bile acid from microbiota metabolism attenuates ileitis and bile acid reduction in subclinical necrotic enteritis in chickens
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- Microbiota attenuates chicken transmission-exacerbated campylobacteriosis in Il10−/− mice
Showing 5 of 7 shared publications
- A secondary bile acid from microbiota metabolism attenuates ileitis and bile acid reduction in subclinical necrotic enteritis in chickens
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Microbiota attenuates chicken transmission-exacerbated campylobacteriosis in Il10−/− mice
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
Showing 5 of 6 shared publications
- A secondary bile acid from microbiota metabolism attenuates ileitis and bile acid reduction in subclinical necrotic enteritis in chickens
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- Microbiota attenuates chicken transmission-exacerbated campylobacteriosis in Il10−/− mice
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
- A secondary bile acid from microbiota metabolism attenuates ileitis and bile acid reduction in subclinical necrotic enteritis in chickens
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- A secondary bile acid from microbiota metabolism attenuates ileitis and bile acid reduction in subclinical necrotic enteritis in chickens
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- A secondary bile acid from microbiota metabolism attenuates ileitis and bile acid reduction in subclinical necrotic enteritis in chickens
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
- A secondary bile acid from microbiota metabolism attenuates ileitis and bile acid reduction in subclinical necrotic enteritis in chickens
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- A secondary bile acid from microbiota metabolism attenuates ileitis and bile acid reduction in subclinical necrotic enteritis in chickens
- Microbiota attenuates chicken transmission-exacerbated campylobacteriosis in Il10−/− mice
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
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