Match tier Listed
Presence Current · Arkansas
Last published 2024
Sources OpenAlex · ORCID
Refreshed 2026-08-08

Rachel S. Palmer

Researcher

UAMS

Faculty Researcher

2 h-index 13 pubs 11 cited

Research Areas

Biomedical Subjects

  • Chlamydia Infections
  • Homeodomain Proteins
  • Chlamydia muridarum
  • Basic Helix-Loop-Helix Transcription Factors
  • Animals
  • Cell Differentiation
  • Female
  • Mice, Inbred C57BL
  • CD4-Positive T-Lymphocytes
  • Interleukin-10
  • Th1 Cells
  • Mice

OverviewAI-generated summary

Rachel S. Palmer's research investigates the role of the transcription factor BHLHE40 in the immune response to Chlamydia infections, particularly within the female reproductive tract. Her work has demonstrated that BHLHE40 promotes the differentiation of polyfunctional CD4 T cells, which are crucial for controlling Chlamydia muridarum infections in mice. Palmer has published 13 papers, with a total of 9 citations and an h-index of 2. She has collaborated with several researchers at the University of Arkansas for Medical Sciences, including Lin‐Xi Li, Miguel A. B. Mercado, and Yejin Kim, co-authoring three publications with each. Her most recent publication was in 2024, indicating recent activity in her field.

Metrics

  • h-index: 2
  • Publications: 13
  • Citations: 11

Selected Publications

  • BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia (2024)
    PLoS Pathogens 4 citations DOI OpenAlex
  • BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against <i>Chlamydia</i> (2023)
    bioRxiv (Cold Spring Harbor Laboratory) DOI OpenAlex
  • Transcription factor Bhlhe40 plays a protective role during intravaginal <i>Chlamydia muridarum</i> infection in mice (2023)
    The Journal of Immunology DOI OpenAlex

View all publications on OpenAlex →

Collaboration Network

7 Collaborators 1 Institution 1 Country

Top Collaborators

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