Yejin Kim
Researcher
Also affiliated: Namseoul University (2020); Harvard University (2022); Seoul National University (2012–2026); Dongguk University (2013–2016); New Generation University College (2012–2026); Seoul National University Hospital (2020); Kyung Hee University (2022–2025); Massachusetts General Hospital (2022); Obihiro University of Agriculture and Veterinary Medicine (2020); Chung-Ang University (2019); Myongji University (2021); The University of Texas Health Science Center at Houston (2023)
Faculty Researcher
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Yejin Kim's research investigates host-pathogen interactions and immune responses, with a particular focus on T cell differentiation and function in the context of infectious diseases. Recent work has explored the role of specific T cell populations, such as CXCR6+ polyfunctional CD4 T cells, in providing protective immunity against Chlamydia infection in the female reproductive tract. This research also examines the regulatory pathways influencing T cell responses, including the involvement of TGF-beta signaling. Additionally, Kim's work has touched upon natural products and their potential therapeutic applications, such as investigating the cytotoxic properties of plant-derived compounds and their effects on cancer cells and immune cells. This includes research on how natural compounds like Hydnocarpin can induce apoptosis in ovarian cancer cells and reprogram tumor-associated immune cells, as well as the potential anti-skin-aging effects of compounds derived from Rosa rugosa.
Kim has contributed to studies utilizing artificial intelligence for disease regimen planning, specifically in the context of Alzheimer's disease. Collaborations include work with researchers at the University of Arkansas for Medical Sciences, such as Lin-Xi Li, Miguel A. B. Mercado, and Rachel S. Palmer. Kim's scholarship metrics include an h-index of 8, with 26 total publications and 229 total citations.
Metrics
- h-index: 7
- Publications: 27
- Citations: 192
Selected Publications
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CXCR6 marks polyfunctional effector CD4 T cells required for anti- <i>Chlamydia</i> immunity in the female reproductive tract (2026)
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The roles of TGFb signaling in CD4 T cell responses to Chlamydia infection in the female reproductive tract 9284 (2025)
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CXCR6+ polyfunctional CD4 T cells are essential for protective immunity against Chlamydia in the female reproductive tract (2024)
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BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia (2024)
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BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against <i>Chlamydia</i> (2023)
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Transcription factor Bhlhe40 plays a protective role during intravaginal <i>Chlamydia muridarum</i> infection in mice (2023)
Collaboration Network
Top Collaborators
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- Transcription factor Bhlhe40 plays a protective role during intravaginal <i>Chlamydia muridarum</i> infection in mice
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against <i>Chlamydia</i>
- CXCR6+ polyfunctional CD4 T cells are essential for protective immunity against Chlamydia in the female reproductive tract
- The roles of TGFb signaling in CD4 T cell responses to Chlamydia infection in the female reproductive tract 9284
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- Transcription factor Bhlhe40 plays a protective role during intravaginal <i>Chlamydia muridarum</i> infection in mice
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against <i>Chlamydia</i>
- The roles of TGFb signaling in CD4 T cell responses to Chlamydia infection in the female reproductive tract 9284
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- Transcription factor Bhlhe40 plays a protective role during intravaginal <i>Chlamydia muridarum</i> infection in mice
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against <i>Chlamydia</i>
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- Transcription factor Bhlhe40 plays a protective role during intravaginal <i>Chlamydia muridarum</i> infection in mice
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against <i>Chlamydia</i>
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- Transcription factor Bhlhe40 plays a protective role during intravaginal <i>Chlamydia muridarum</i> infection in mice
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against <i>Chlamydia</i>
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against <i>Chlamydia</i>
- CXCR6+ polyfunctional CD4 T cells are essential for protective immunity against Chlamydia in the female reproductive tract
- Transcription factor Bhlhe40 plays a protective role during intravaginal <i>Chlamydia muridarum</i> infection in mice
- CXCR6+ polyfunctional CD4 T cells are essential for protective immunity against Chlamydia in the female reproductive tract
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