Yejin Kim

Researcher

Last publication 2025 Last refreshed 2026-05-16

faculty

8 h-index 26 pubs 224 cited

Biography and Research Information

OverviewAI-generated summary

Yejin Kim's research investigates the molecular mechanisms underlying immune responses and cellular processes, with a particular focus on infectious diseases and cancer. Her work has explored the role of specific T cell populations, such as CXCR6+ polyfunctional CD4 T cells, in providing protective immunity against Chlamydia infection in the female reproductive tract. Kim has also studied the signaling pathways, including TGFb signaling, that influence CD4 T cell responses in this context. Furthermore, her research extends to the cytotoxic properties of natural compounds, examining their potential in cancer treatment. For instance, she has investigated the apoptosis-inducing effects of Hydnocarpin, a flavonolignan, on ovarian cancer cells and its impact on tumor-associated immune cells. Her publications also include studies on the potential anti-skin-aging effects of compounds derived from Rosa rugosa and the application of artificial intelligence in developing treatment regimens for Alzheimer's disease. Kim has collaborated with researchers at the University of Arkansas for Medical Sciences, including Lin‐Xi Li, Miguel A. B. Mercado, and Rachel S. Palmer.

Metrics

  • h-index: 8
  • Publications: 26
  • Citations: 224

Selected Publications

  • The roles of TGFb signaling in CD4 T cell responses to Chlamydia infection in the female reproductive tract 9284 (2025)
  • CXCR6+ polyfunctional CD4 T cells are essential for protective immunity against Chlamydia in the female reproductive tract (2024)
  • BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia (2024)
    2 citations DOI OpenAlex
  • BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against <i>Chlamydia</i> (2023)
  • Transcription factor Bhlhe40 plays a protective role during intravaginal <i>Chlamydia muridarum</i> infection in mice (2023)

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Collaboration Network

43 Collaborators 11 Institutions 2 Countries

Top Collaborators

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