Yejin Kim
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Also affiliated: Harvard University (2022); Seoul National University (2012–2026); Dongguk University (2013–2016); Kyung Hee University (2022–2025); Massachusetts General Hospital (2022); Obihiro University of Agriculture and Veterinary Medicine (2020); Chung-Ang University (2019); Myongji University (2021); The University of Texas Health Science Center at Houston (2023)
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Yejin Kim's research has focused on the immune system and cellular responses, with publications investigating the effects of various agents on immune cell function and tumor cells. This includes work on alloferon's impact on NK cell cytotoxicity against cancer, and how CD40 on tumor cells interacts with CD40L on T cells to influence tumor cell proliferation and Th17 differentiation. Kim has also studied the mechanisms of vitamin C in up-regulating dendritic cell expression via the p38 MAPK pathway. Further investigations have explored regulatory T cells' role in suppressing skin inflammation in mouse models and the activation of the PI3K/Akt pathway by a viral interferon regulatory factor. Kim's work has involved collaborations with researchers at the University of Arkansas for Medical Sciences, including Lin-Xi Li, Miguel A. B. Mercado, and Rachel S. Palmer, with whom Kim shares multiple publications. Kim has an h-index of 7, with 27 total publications and 194 citations.
Metrics
- h-index: 7
- Publications: 27
- Citations: 195
Selected Publications
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CXCR6 marks polyfunctional effector CD4 T cells required for anti- Chlamydia immunity in the female reproductive tract (2026)
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The roles of TGFb signaling in CD4 T cell responses to Chlamydia infection in the female reproductive tract 9284 (2025)
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CXCR6+ polyfunctional CD4 T cells are essential for protective immunity against Chlamydia in the female reproductive tract (2024)
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BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia (2024)
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BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia (2023)
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Transcription factor Bhlhe40 plays a protective role during intravaginal Chlamydia muridarum infection in mice (2023)
Collaboration Network
Top Collaborators
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- Transcription factor Bhlhe40 plays a protective role during intravaginal Chlamydia muridarum infection in mice
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- CXCR6+ polyfunctional CD4 T cells are essential for protective immunity against Chlamydia in the female reproductive tract
- The roles of TGFb signaling in CD4 T cell responses to Chlamydia infection in the female reproductive tract 9284
Showing 5 of 6 shared publications
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- Transcription factor Bhlhe40 plays a protective role during intravaginal Chlamydia muridarum infection in mice
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- The roles of TGFb signaling in CD4 T cell responses to Chlamydia infection in the female reproductive tract 9284
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- Transcription factor Bhlhe40 plays a protective role during intravaginal Chlamydia muridarum infection in mice
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- Transcription factor Bhlhe40 plays a protective role during intravaginal Chlamydia muridarum infection in mice
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- Transcription factor Bhlhe40 plays a protective role during intravaginal Chlamydia muridarum infection in mice
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- CXCR6+ polyfunctional CD4 T cells are essential for protective immunity against Chlamydia in the female reproductive tract
- Transcription factor Bhlhe40 plays a protective role during intravaginal Chlamydia muridarum infection in mice
- CXCR6 marks polyfunctional effector CD4 T cells required for anti- Chlamydia immunity in the female reproductive tract
- CXCR6 marks polyfunctional effector CD4 T cells required for anti- Chlamydia immunity in the female reproductive tract
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