Match tier
Listed
Presence
Not recently confirmed
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-06
M
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Mason McCrury's research investigates the role of DNA secondary structures, particularly G-quadruplexes and i-motifs, in gene regulation and cellular processes. His work has explored these structures in the promoter regions of key genes involved in innate immunity and oncogenesis, such as MYD88 and CARD11. McCrury has also examined how these DNA structures influence the binding and activity of enzymes like activation-induced cytidine deaminase (AID) and Rev1, and how they interact with proteins like DHX36.
His publications include studies on therapeutic targets in lymphoma, such as NEK2, and the potential of small molecules to interact with specific DNA structures. McCrury's research network includes extensive collaboration with Kennith Swafford, Vijay Patel, Ying-Zhi Xu, and Claire Doshier at the University of Arkansas for Medical Sciences, with whom he has co-authored a significant number of publications. His scholarship metrics include an h-index of 4 across 60 publications.
Metrics
- h-index: 4
- Publications: 60
- Citations: 35
Selected Publications
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Human REV1 interacts with DHX36 to promote replication and tolerance of G-quadruplex DNA (2026)
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DNA secondary structures in BCL2 and MYC elicit activation-induced cytidine deaminase binding and activity (2026)
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G-quadruplex and i-motif DNA structures form in the promoter of the key innate immune adaptor MYD88 (2025)
Collaboration Network
Top Collaborators
Samantha Kendrick
University of Arkansas for Medical Sciences
4 shared publications
In database
- Bifunctional Inhibitor Reveals NEK2 as a Therapeutic Target and Regulator of Oncogenic Pathways in Lymphoma
- G-quadruplex and i-motif DNA structures form in the promoter of the key innate immune adaptor MYD88
- Targeting a Novel G-Quadruplex in the CARD11 Oncogene Promoter with Naptho(2,1-b)furan-1-ethanol,2-nitro- Requires the Nitro Group
- DNA secondary structures in BCL2 and MYC elicit activation-induced cytidine deaminase binding and activity
Kennith Swafford
University of Arkansas for Medical Sciences
3 shared publications
In database
- Bifunctional Inhibitor Reveals NEK2 as a Therapeutic Target and Regulator of Oncogenic Pathways in Lymphoma
- G-quadruplex and i-motif DNA structures form in the promoter of the key innate immune adaptor MYD88
- Targeting a Novel G-Quadruplex in the CARD11 Oncogene Promoter with Naptho(2,1-b)furan-1-ethanol,2-nitro- Requires the Nitro Group
Baku Acharya
University of Arkansas for Medical Sciences
2 shared publications
In database
- Bifunctional Inhibitor Reveals NEK2 as a Therapeutic Target and Regulator of Oncogenic Pathways in Lymphoma
- Targeting a Novel G-Quadruplex in the CARD11 Oncogene Promoter with Naptho(2,1-b)furan-1-ethanol,2-nitro- Requires the Nitro Group
Ying-Zhi Xu
University of Arkansas for Medical Sciences
2 shared publications
In database
- Bifunctional Inhibitor Reveals NEK2 as a Therapeutic Target and Regulator of Oncogenic Pathways in Lymphoma
- Targeting a Novel G-Quadruplex in the CARD11 Oncogene Promoter with Naptho(2,1-b)furan-1-ethanol,2-nitro- Requires the Nitro Group
Debasmita Saha
University of Arkansas for Medical Sciences (US)
2 shared publications
- Bifunctional Inhibitor Reveals NEK2 as a Therapeutic Target and Regulator of Oncogenic Pathways in Lymphoma
- Targeting a Novel G-Quadruplex in the CARD11 Oncogene Promoter with Naptho(2,1-b)furan-1-ethanol,2-nitro- Requires the Nitro Group
Brendan Frett
University of Arkansas for Medical Sciences
2 shared publications
In database
- Bifunctional Inhibitor Reveals NEK2 as a Therapeutic Target and Regulator of Oncogenic Pathways in Lymphoma
- Targeting a Novel G-Quadruplex in the CARD11 Oncogene Promoter with Naptho(2,1-b)furan-1-ethanol,2-nitro- Requires the Nitro Group
Reham S. Sewilam
University of Arkansas for Medical Sciences
2 shared publications
In database
- Conservation of the insert-2 motif confers Rev1 from different species with an ability to disrupt G-quadruplexes and stimulate translesion DNA synthesis
- Human REV1 interacts with DHX36 to promote replication and tolerance of G-quadruplex DNA
Bethany C. Paxton
University of Arkansas for Medical Sciences
2 shared publications
In database
- Conservation of the insert-2 motif confers Rev1 from different species with an ability to disrupt G-quadruplexes and stimulate translesion DNA synthesis
- Human REV1 interacts with DHX36 to promote replication and tolerance of G-quadruplex DNA
Robert L. Eoff
University of Arkansas for Medical Sciences
2 shared publications
In database
- Conservation of the insert-2 motif confers Rev1 from different species with an ability to disrupt G-quadruplexes and stimulate translesion DNA synthesis
- Human REV1 interacts with DHX36 to promote replication and tolerance of G-quadruplex DNA
Claire Doshier
University of Arkansas for Medical Sciences
2 shared publications
In database
- Bifunctional Inhibitor Reveals NEK2 as a Therapeutic Target and Regulator of Oncogenic Pathways in Lymphoma
- DNA secondary structures in BCL2 and MYC elicit activation-induced cytidine deaminase binding and activity
Thomas Raney
University of Arkansas for Medical Sciences
1 shared publication
In database
- Targeting a Novel G-Quadruplex in the CARD11 Oncogene Promoter with Naptho(2,1-b)furan-1-ethanol,2-nitro- Requires the Nitro Group
Amit Ketkar
University of Arkansas for Medical Sciences
1 shared publication
In database
- Conservation of the insert-2 motif confers Rev1 from different species with an ability to disrupt G-quadruplexes and stimulate translesion DNA synthesis
Ashtyn Bell
University of Arkansas for Medical Sciences
1 shared publication
In database
- Conservation of the insert-2 motif confers Rev1 from different species with an ability to disrupt G-quadruplexes and stimulate translesion DNA synthesis
Shreyam Tripathi
1 shared publication
- Conservation of the insert-2 motif confers Rev1 from different species with an ability to disrupt G-quadruplexes and stimulate translesion DNA synthesis
Julie Gunderson
Hendrix College
1 shared publication
In database
- Conservation of the insert-2 motif confers Rev1 from different species with an ability to disrupt G-quadruplexes and stimulate translesion DNA synthesis
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