Match tier
Listed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-01
S
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Sheldon L. Zeltner's research focuses on the immune response during Plasmodium yoelii infection in mice. His work investigates the role of cytokines, specifically Interleukin-10 (IL-10) and Interferon-gamma (IFN-γ), in controlling malaria progression. Zeltner's publications explore how the absence of IL-10 affects early parasite control and whether this control is mediated by IFN-γ, Interleukin-12 (IL-12), or humoral immune responses. He also studies the influence of genes, such as Bhlhe40, on T cell cytokine production during infection. Zeltner collaborates with researchers at the University of Arkansas for Medical Sciences, including Kara A. O’Neal, Jason S. Stumhofer, Camille L. Foscue, and Alison Gouch. His scholarly contributions include three publications with a total of four citations and an h-index of 1.
Metrics
- h-index: 1
- Publications: 3
- Citations: 4
Selected Publications
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Early enhanced control of Plasmodium yoelii infection in IL-10-deficient mice is independent of IFN-γ, IL-12, and the humoral response (2026)
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Early enhanced control of Plasmodium yoelii infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response (2026)
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Bhlhe40 limits early IL-10 production from CD4 + T cells during Plasmodium yoelii 17X infection (2023)
Collaboration Network
Top Collaborators
Jason S. Stumhofer
University of Arkansas for Medical Sciences
3 shared publications
In database
- Bhlhe40 limits early IL-10 production from CD4 + T cells during Plasmodium yoelii 17X infection
- Early enhanced control of Plasmodium yoelii infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response
- Early enhanced control of Plasmodium yoelii infection in IL-10-deficient mice is independent of IFN-γ, IL-12, and the humoral response
Kara A. O’Neal
University of Arkansas for Medical Sciences
2 shared publications
In database
- Bhlhe40 limits early IL-10 production from CD4 + T cells during Plasmodium yoelii 17X infection
- Early enhanced control of Plasmodium yoelii infection in IL-10-deficient mice is independent of IFN-γ, IL-12, and the humoral response
Meghan D. Jones
University of Arkansas for Medical Sciences
2 shared publications
In database
- Early enhanced control of Plasmodium yoelii infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response
- Early enhanced control of Plasmodium yoelii infection in IL-10-deficient mice is independent of IFN-γ, IL-12, and the humoral response
Alison Gouch
University of Arkansas for Medical Sciences
2 shared publications
In database
- Early enhanced control of Plasmodium yoelii infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response
- Early enhanced control of Plasmodium yoelii infection in IL-10-deficient mice is independent of IFN-γ, IL-12, and the humoral response
Camille L. Foscue
University of Arkansas for Medical Sciences
1 shared publication
In database
- Bhlhe40 limits early IL-10 production from CD4 + T cells during Plasmodium yoelii 17X infection
Kara A O'Neal
University of Arkansas for Medical Sciences
1 shared publication
In database
- Early enhanced control of Plasmodium yoelii infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response
Jeanette M. Ramos
University of Arkansas for Medical Sciences
1 shared publication
In database
- Early enhanced control of Plasmodium yoelii infection in IL-10-deficient mice is independent of IFN-γ, IL-12, and the humoral response
Similar Researchers
Based on overlapping research topics
Kara A. O’Neal
University of Arkansas for Medical Sciences
Host-Pathogen Interactions
Immune Response and Inflammation
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Alison R. Gouch
University of Arkansas for Medical Sciences
Host-Pathogen Interactions
Immune Response and Inflammation
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Bernabe Mercado
University of Arkansas for Medical Sciences
Host-Pathogen Interactions
Immune Response and Inflammation
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Jessica I. Gann
University of Arkansas for Medical Sciences
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Lin‐Xi Li
University of Arkansas for Medical Sciences
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Marli S.P. Azevedo
National Center for Toxicological Research
Host-Pathogen Interactions
Immune Response and Inflammation
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