Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Ananya Ranaraja's research focuses on the interaction between host immune cells and the bacterium *Mycobacterium tuberculosis* (Mtb). Their work investigates how neutrophils, a type of white blood cell, can create an environment that supports Mtb growth. Ranaraja has also explored the protective role of alveolar macrophages, which are derived from monocytes, in combating Mtb infection. Another area of study involves the role of fatty acid oxidation in maintaining the balance of alveolar macrophages and their ability to resist infection. Ranaraja's scholarship metrics include an h-index of 1, with 3 publications and 33 citations. Key collaborators include J. Tucker Andrews, Lin‐Xi Li, Zijing Zhang, and Fischer Huey, all from the University of Arkansas for Medical Sciences.
Metrics
- h-index: 1
- Publications: 3
- Citations: 36
Selected Publications
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β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395 (2025)
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Fatty acid oxidation regulates alveolar macrophage homeostasis and resistance to infection 4350 (2025)
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Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis (2024)
Collaboration Network
Top Collaborators
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Fatty acid oxidation regulates alveolar macrophage homeostasis and resistance to infection 4350
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Fatty acid oxidation regulates alveolar macrophage homeostasis and resistance to infection 4350
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
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