Biography and Research Information
OverviewAI-generated summary
Ananya Ranaraja's research focuses on the immune system's role in combating infectious diseases, particularly tuberculosis. Their work investigates how different types of immune cells, such as neutrophils and macrophages, respond to and influence the progression of Mycobacterium tuberculosis infections. Recent publications explore the metabolic activity of neutrophils as a potential niche for the bacteria and how monocyte-derived alveolar macrophages, induced by β-glucan, contribute to protection against infection. Additionally, their research examines the function of fatty acid oxidation in maintaining alveolar macrophage health and resistance to pathogens. Ranaraja has published three papers, accumulating 28 citations, and holds an h-index of 1. They have collaborated with researchers including J. Tucker Andrews and Lin‐Xi Li at the University of Arkansas for Medical Sciences.
Metrics
- h-index: 1
- Publications: 3
- Citations: 30
Selected Publications
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β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395 (2025)
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Fatty acid oxidation regulates alveolar macrophage homeostasis and resistance to infection 4350 (2025)
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Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis (2024)
Collaboration Network
Top Collaborators
J. Tucker Andrews
University of Arkansas for Medical Sciences
3 shared publications
In database
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Fatty acid oxidation regulates alveolar macrophage homeostasis and resistance to infection 4350
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
Lu Huang
University of Arkansas for Medical Sciences
3 shared publications
In database
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Fatty acid oxidation regulates alveolar macrophage homeostasis and resistance to infection 4350
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
Zijing Zhang
University of Arkansas for Medical Sciences
2 shared publications
In database
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
Lin‐Xi Li
University of Arkansas for Medical Sciences
2 shared publications
In database
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
Nan Zhang
The Wistar Institute (US)
2 shared publications
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
Gauri Prasad
Washington University in St. Louis (US)
1 shared publication
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
Fischer Huey
University of Arkansas for Medical Sciences
1 shared publication
In database
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
Evgeniya V. Nazarova
Cornell University (US)
1 shared publication
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
Jocelyn Wang
Cornell University (US)
1 shared publication
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
Tiffany Weinkopff
University of Arkansas for Medical Sciences
1 shared publication
In database
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
Shengyu Mu
University of Arkansas for Medical Sciences
1 shared publication
In database
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
Michael J. Birrer
1 shared publication
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
Stanley Ching‐Cheng Huang
1 shared publication
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
Rafael J. Argüello
Centre d’Immunologie de Marseille-Luminy (FR)
1 shared publication
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
Jennifer A. Philips
1 shared publication
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
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