A. Moerman
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Postdoctoral Fellow
Also affiliated: Washington University in St. Louis (1998); TuBerculosis Vaccine Initiative (1994); Ghent University Hospital (2024); Centre Hospitalier Universitaire de Lille (2017–2021); Arkansas Cardiology (2006); Hôpital Roger Salengro (2001); Hôpital Jeanne de Flandre (1999–2020); Geriatric Research Education and Clinical Center (1993); Hôpitaux Universitaires de Strasbourg (1999); John L. McClellan Memorial Veterans Hospital (1993–1999); International Commission for the Hydrology of the Rhine Basin (1993); Arkansas Department of Agriculture (2015); Hôpital Civil, Strasbourg (1999); Saint Louis University (1997–1998)
Postdoc Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
A. Moerman's research investigates molecular mechanisms underlying neurological and cellular processes, with a focus on gene expression and signaling pathways. Recent work has explored the role of the reelin signaling pathway in cognitive function, demonstrating how a central repeat fragment can rescue deficits in mouse models of reelin deficiency. This research also examines the modulation of apolipoprotein E receptor-2 by various factors, including ApoE4 and amyloid β-peptide, suggesting a common point of impact in Alzheimer's disease pathogenesis. Additionally, Moerman has contributed to studies on cutaneous T-cell lymphoma, investigating the differential regulation of gene expression by normal and cancer fibroblasts. Other research interests include transcriptomic profiling of peripheral blood mononuclear cells and CD4+ T cells from Sézary syndrome patients, and the impact of next-generation sequencing strategies on diagnosing rare cardiovascular conditions such as Fabry disease and hereditary transthyretin amyloidosis.
Moerman's scholarly output includes 60 publications with 1,310 citations and an h-index of 21. Key collaborators include Syed Hassan Mehdi and Henry N. C. Wong from the University of Arkansas for Medical Sciences, with whom Moerman has co-authored multiple publications. Other collaborators include Steven W. Barger from the University of Arkansas for Medical Sciences and Pascale Richard from the University of Arkansas at Little Rock. Moerman has been recently active, with publications extending to 2026.
Metrics
- h-index: 21
- Publications: 60
- Citations: 1,320
Selected Publications
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Modulation of apolipoprotein E receptor-2 by ApoE4, amyloid β-peptide, reelin, and secreted amyloid precursor protein: a common point of impact in Alzheimer’s disease pathogenesis (2026)
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Central repeat fragment of reelin leads to active reelin intracellular signaling and rescues cognitive deficits in a mouse model of reelin deficiency (2023)
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Isolating Human Peripheral Blood Mononuclear Cells and CD4+ T cells from Sézary Syndrome Patients for Transcriptomic Profiling (2021)
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Isolating Human Peripheral Blood Mononuclear Cells and CD4+ T cells from Sézary Syndrome Patients for Transcriptomic Profiling (2021)
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Normal and cancer fibroblasts differentially regulate TWIST1, TOX and cytokine gene expression in cutaneous T-cell lymphoma (2021)
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Normal and Cancer Fibroblasts Differentially Regulate Cytokine Genes and TWIST1 and TOX  Expression in Cutaneous T-cell Lymphoma  (2020)
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Gene Expression Comparison between Sézary Syndrome and Lymphocytic-Variant Hypereosinophilic Syndrome Refines Biomarkers for Sézary Syndrome (2020)
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154 Shared CpG methylation defects in mycosis fungoides and Sézary syndrome (2020)
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Transcriptome analysis of Sézary syndrome and lymphocytic-variant hypereosinophilic syndrome T cells reveals common and divergent genes (2019)
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1037 Transcriptome analysis in Sézary syndrome and lymphocytic variant hypereosinophilic syndrome identify common and unique genes (2019)
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222 DNA methylomic profiles of TWIST1, PLS3 and GATA6 genes in Sezary Syndrome (2018)
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129 Cutaneous T cell lymphoma tumor cells expanded in vitro retain expression of biomarker genes (2017)
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Presenilin 1 mutations influence processing and trafficking of the ApoE receptor apoER2 (2016)
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NFκB‐inducing kinase inhibits NFκB activity specifically in neurons of the CNS (2016)
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Human papillomavirus type 16 viral load is decreased following a therapeutic vaccination (2016)
Collaboration Network
Top Collaborators
- Normal and cancer fibroblasts differentially regulate TWIST1, TOX and cytokine gene expression in cutaneous T-cell lymphoma
- Isolating Human Peripheral Blood Mononuclear Cells and CD4+ T cells from Sézary Syndrome Patients for Transcriptomic Profiling
- Isolating Human Peripheral Blood Mononuclear Cells and CD4+ T cells from Sézary Syndrome Patients for Transcriptomic Profiling
- Normal and cancer fibroblasts differentially regulate TWIST1, TOX and cytokine gene expression in cutaneous T-cell lymphoma
- Isolating Human Peripheral Blood Mononuclear Cells and CD4+ T cells from Sézary Syndrome Patients for Transcriptomic Profiling
- Isolating Human Peripheral Blood Mononuclear Cells and CD4+ T cells from Sézary Syndrome Patients for Transcriptomic Profiling
- Central repeat fragment of reelin leads to active reelin intracellular signaling and rescues cognitive deficits in a mouse model of reelin deficiency
- Modulation of apolipoprotein E receptor-2 by ApoE4, amyloid β-peptide, reelin, and secreted amyloid precursor protein: a common point of impact in Alzheimer’s disease pathogenesis
- Central repeat fragment of reelin leads to active reelin intracellular signaling and rescues cognitive deficits in a mouse model of reelin deficiency
- Central repeat fragment of reelin leads to active reelin intracellular signaling and rescues cognitive deficits in a mouse model of reelin deficiency
- Central repeat fragment of reelin leads to active reelin intracellular signaling and rescues cognitive deficits in a mouse model of reelin deficiency
- Central repeat fragment of reelin leads to active reelin intracellular signaling and rescues cognitive deficits in a mouse model of reelin deficiency
- Central repeat fragment of reelin leads to active reelin intracellular signaling and rescues cognitive deficits in a mouse model of reelin deficiency
- Central repeat fragment of reelin leads to active reelin intracellular signaling and rescues cognitive deficits in a mouse model of reelin deficiency
- Central repeat fragment of reelin leads to active reelin intracellular signaling and rescues cognitive deficits in a mouse model of reelin deficiency
- Central repeat fragment of reelin leads to active reelin intracellular signaling and rescues cognitive deficits in a mouse model of reelin deficiency
- Central repeat fragment of reelin leads to active reelin intracellular signaling and rescues cognitive deficits in a mouse model of reelin deficiency
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