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Biography and Research Information
OverviewAI-generated summary
Dominique J. Laster's research investigates the biological mechanisms underlying bone health, with a recent focus on the role of autophagy. Laster's work has explored the impact of chaperone-mediated autophagy loss on bone mass, particularly in the context of aging and sex differences in mice. Publications include studies on the association between chaperone-mediated autophagy and low vertebral cancellous bone mass, and the lack of alteration in age-related bone loss in male mice following the loss of this process. Additionally, Laster has contributed to research on molecular biology techniques, specifically comparing CRISPR interference with the Cre-loxP system for enhanced cell type specificity. Scholarship metrics indicate an h-index of 3, with 3 total publications and 21 total citations. Laster has collaborated with researchers at the University of Arkansas for Medical Sciences, including Melda Onal, Julie Crawford, Nisreen Akel, and Stuart B. Berryhill, with whom multiple publications are shared.
Metrics
- h-index: 3
- Publications: 3
- Citations: 21
Selected Publications
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Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice (2024)
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CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system (2023)
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Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass (2022)
Collaboration Network
Top Collaborators
- Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
- Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
- Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
- Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
- Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass
- Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass
- Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
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