Match tier
Listed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-08-16
Q
Qiang Fu
High Impact
Assistant Research Professor
Faculty Researcher
Internal Med, College of Medicine
25 h-index
61 pubs
2,894 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Qiang Fu's research investigates the molecular mechanisms underlying bone health and loss, with a particular focus on osteocyte function, autophagy, and the impact of ionizing radiation. His work has explored how reduced osteoprotegerin expression in osteocytes may contribute to bone resorption following denosumab discontinuation. Additionally, Fu has studied the role of long non-coding RNAs, such as LncRNA ROR, in modulating osteoblast proliferation and apoptosis, which is relevant to osteoporosis.
Further research by Fu examines the effects of ionizing radiation on bone, specifically how it activates mitochondrial function in osteoclasts and leads to bone loss in young adult male mice. His investigations also extend to the connection between autophagy and bone mass, as demonstrated by his work on the association between loss of chaperone-mediated autophagy and low vertebral cancellous bone mass. Fu has also contributed to the understanding of mesenchymal cell types in bone regeneration and the role of short-chain fatty acids in metabolic syndrome and its associated immune and inflammatory complications.
Metrics
- h-index: 25
- Publications: 61
- Citations: 2,894
Selected Publications
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Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells (2025)
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Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice (2025)
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Sirtuin-3 promotes osteoclast maturation and bone loss by regulating mitochondrial ROS production during ionizing radiation exposure (2025)
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Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells (2025)
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Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness (2025)
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Refining the identity of mesenchymal cell types associated with murine periosteal and endosteal bone (2024)
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A framework for defining mesenchymal cell types associated with murine periosteal and endosteal bone (2023)
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Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation (2023)
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A Rab33b missense mouse model for Smith-McCort dysplasia shows bone resorption defects and altered protein glycosylation (2023)
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Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass (2022)
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Ionizing Radiation Activates Mitochondrial Function in Osteoclasts and Causes Bone Loss in Young Adult Male Mice (2022)
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Deuteration of the farnesyl terminal methyl groups of δ-tocotrienol and its effects on the metabolic stability and ability of inducing G-CSF production (2020)
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C/EBPδ protects from radiation-induced intestinal injury and sepsis by suppression of inflammatory and nitrosative stress (2019)
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The Role of Tetrahydrobiopterin (BH4) Bioavailability in Radiation-Induced Skin Injury (2015)
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Characterization of Transgenic <i>Gfrp</i> Knock-In Mice: Implications for Tetrahydrobiopterin in Modulation of Normal Tissue Radiation Responses (2013)
Grants & Funding
As listed on this researcher's institutional profile.
- Synergistic Radioprotection of Gastrointestinal Tract by Gamma-Tocotrienol (GT3) and Statins: Efficacy and Mechanisms Uniformed Services University of the Health Sciences via Henry M. Jackson Foundation for the Advancement of Military Medicine, Inc. Co-Investigator
- Evaluation of IPW-5371, a TGFRI kinase inhibitor, dosed as a single agent or in combination with G-CSF, as a medical countermeasure against the delayed effects of total body irradiation in mice - Continuation NIH/Nat. Inst. of Allergy & Infectious Diseases via Innovation Pathways Principal Investigator
- Osteocyte Control of Bone Remodeling NIH/Nat. Inst. of Arthritis & Musculoskeletal & Skin Diseases Principal Investigator
- Advanced Development of Gamma-Tocotrienol as a Radiation Countermeasure US Department of Defense via Armed Forces Radiobiology Research Institute Co-Investigator
- Center for Musculoskeletal Disease Research (CMDR) NIH/Nat. Inst. of General Medical Sciences Principal Investigator
- Endonuclease-targeted radioprotection NIH/Nat. Inst. of Allergy & Infectious Diseases via Duke University Co-Investigator
Collaboration Network
Top Collaborators
Ha‐Neui Kim
University of Arkansas for Medical Sciences
6 shared publications
In database
- Refining the identity of mesenchymal cell types associated with murine periosteal and endosteal bone
- Ionizing Radiation Activates Mitochondrial Function in Osteoclasts and Causes Bone Loss in Young Adult Male Mice
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- A framework for defining mesenchymal cell types associated with murine periosteal and endosteal bone
- Sirtuin-3 promotes osteoclast maturation and bone loss by regulating mitochondrial ROS production during ionizing radiation exposure
Showing 5 of 6 shared publications
Charles A. O’Brien
University of Arkansas for Medical Sciences
6 shared publications
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
- Refining the identity of mesenchymal cell types associated with murine periosteal and endosteal bone
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- A framework for defining mesenchymal cell types associated with murine periosteal and endosteal bone
Showing 5 of 6 shared publications
Intawat Nookaew
University of Arkansas for Medical Sciences
5 shared publications
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
- Refining the identity of mesenchymal cell types associated with murine periosteal and endosteal bone
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- A framework for defining mesenchymal cell types associated with murine periosteal and endosteal bone
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
Maria Almeida
University of Arkansas for Medical Sciences
5 shared publications
In database
- Refining the identity of mesenchymal cell types associated with murine periosteal and endosteal bone
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- A framework for defining mesenchymal cell types associated with murine periosteal and endosteal bone
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
Melda Onal
University of Arkansas for Medical Sciences
4 shared publications
In database
- Refining the identity of mesenchymal cell types associated with murine periosteal and endosteal bone
- Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- A framework for defining mesenchymal cell types associated with murine periosteal and endosteal bone
Jeff D. Thostenson
University of Arkansas for Medical Sciences
3 shared publications
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
- Sirtuin-3 promotes osteoclast maturation and bone loss by regulating mitochondrial ROS production during ionizing radiation exposure
Jinhu Xiong
University of Arkansas for Medical Sciences
3 shared publications
In database
- Refining the identity of mesenchymal cell types associated with murine periosteal and endosteal bone
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- A framework for defining mesenchymal cell types associated with murine periosteal and endosteal bone
Cecile Bustamante-Gomez
University of Arkansas for Medical Sciences
3 shared publications
In database
- Refining the identity of mesenchymal cell types associated with murine periosteal and endosteal bone
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
- A framework for defining mesenchymal cell types associated with murine periosteal and endosteal bone
Ana I. Coelho
University of Arkansas for Medical Sciences
3 shared publications
In database
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
Md Mohsin Ali
University of Arkansas for Medical Sciences (US)
3 shared publications
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
Aaron Warren
University of Arkansas for Medical Sciences
3 shared publications
In database
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
Kimberly Richardson
University of Arkansas for Medical Sciences (US)
2 shared publications
- Ionizing Radiation Activates Mitochondrial Function in Osteoclasts and Causes Bone Loss in Young Adult Male Mice
- Sirtuin-3 promotes osteoclast maturation and bone loss by regulating mitochondrial ROS production during ionizing radiation exposure
Nükhet Aykin‐Burns
University of Arkansas for Medical Sciences
2 shared publications
In database
- Ionizing Radiation Activates Mitochondrial Function in Osteoclasts and Causes Bone Loss in Young Adult Male Mice
- Sirtuin-3 promotes osteoclast maturation and bone loss by regulating mitochondrial ROS production during ionizing radiation exposure
Milena Dimori
University of Arkansas for Medical Sciences
2 shared publications
In database
- Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass
- A Rab33b missense mouse model for Smith-McCort dysplasia shows bone resorption defects and altered protein glycosylation
Humberto Reyes-Pardo
University of Arkansas for Medical Sciences
2 shared publications
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
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