Match tier
Listed
Presence
Current · Arkansas
Last published
2025
Sources
OpenAlex · ORCID
Refreshed
2026-08-08
Biography and Research Information
OverviewAI-generated summary
James A. Hendrixson's research focuses on the molecular mechanisms regulating bone mass and strength. He has investigated the role of the master autophagy regulator Tfeb in osteoblast lineage cells, finding that its elevation can increase bone mass and strength. His work also explores the efficacy of CRISPR interference compared to the Cre-loxP system for gene editing in specific cell types. Hendrixson has studied the impact of chaperone-mediated autophagy loss on age-related bone loss in male mice, indicating no significant alteration. His collaborations include work with Melda Onal, A. Gordon James, Julie Crawford, and Intawat Nookaew at the University of Arkansas for Medical Sciences, with whom he has co-authored multiple publications. Hendrixson's scholarship metrics include an h-index of 3 across 4 total publications with 19 citations.
Metrics
- h-index: 3
- Publications: 4
- Citations: 22
Selected Publications
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Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength (2025)
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CRISPR activation of <i>Tfeb</i> , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength (2024)
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Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice (2024)
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CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system (2023)
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Retroaortic Left Renal Vein: An Incidental Finding During Cadaver Dissection (2022)
Collaboration Network
Top Collaborators
Julie Crawford
Institute for Musculoskeletal Health
4 shared publications
In database
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
- CRISPR activation of <i>Tfeb</i> , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
Melda Onal
Institute for Musculoskeletal Health
4 shared publications
In database
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
- CRISPR activation of <i>Tfeb</i> , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
A. Gordon James
University of Arkansas for Medical Sciences
3 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
- CRISPR activation of <i>Tfeb</i> , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
Dominique J. Laster
University of Arkansas for Medical Sciences
2 shared publications
In database
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
Nisreen Akel
University of Arkansas for Medical Sciences
2 shared publications
In database
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
Stuart B. Berryhill
University of Arkansas for Medical Sciences
2 shared publications
In database
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
Intawat Nookaew
Institute for Musculoskeletal Health
2 shared publications
In database
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
Ilham Kadhim
University of Arkansas for Medical Sciences
2 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- CRISPR activation of <i>Tfeb</i> , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
Jacob Laster
University of Arkansas for Medical Sciences
2 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- CRISPR activation of <i>Tfeb</i> , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
Jeff Thostenson
University of Arkansas for Medical Sciences
2 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- CRISPR activation of <i>Tfeb</i> , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
Amy Y. Sato
University of Arkansas for Medical Sciences
2 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- CRISPR activation of <i>Tfeb</i> , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
Maria Almeida
University of Arkansas Medical Center
2 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- CRISPR activation of <i>Tfeb</i> , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
Mohsin Syed
University of Arkansas for Medical Sciences
1 shared publication
In database
- Retroaortic Left Renal Vein: An Incidental Finding During Cadaver Dissection
Noor Akhter
University of Arkansas for Medical Sciences
1 shared publication
In database
- Retroaortic Left Renal Vein: An Incidental Finding During Cadaver Dissection
Cade Haynie
University of Arkansas for Medical Sciences (US)
1 shared publication
- Retroaortic Left Renal Vein: An Incidental Finding During Cadaver Dissection
Similar Researchers
Based on overlapping research topics
Nisreen Akel
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Molecular Biology Techniques and Applications
Disease Mechanisms Research
Melda Onal
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Molecular Biology Techniques and Applications
Disease Mechanisms Research
Nancy Cecile Bustamante-Gomez
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Molecular Biology Techniques and Applications
Disease Mechanisms Research
Robert L. Jilka
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Disease Mechanisms Research
Animal, Disease Models
Kathryn Miller Priest
University of Arkansas at Fayetteville
Molecular Biology Techniques and Applications
Disease Mechanisms Research
Animal, Disease Models
Julie Crawford
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Molecular Biology Techniques and Applications
CRISPR and Genetic Engineering