Biography and Research Information
OverviewAI-generated summary
Stuart B. Berryhill's research focuses on the molecular mechanisms underlying bone health and age-related changes. His work has investigated the role of chaperone-mediated autophagy in bone mass, particularly in the context of cancellous bone. Recent publications include studies on the impact of autophagy loss on vertebral bone mass in female mice and the effect of chaperone-mediated autophagy loss on age-related bone loss in male mice. Berryhill has also explored advancements in genetic engineering techniques, specifically comparing CRISPR interference with the Cre-loxP system for cell type specificity. His research collaborations include work with Melda Onal, Dominique J. Laster, Julie Crawford, and Nisreen Akel at the University of Arkansas for Medical Sciences, with whom he has co-authored multiple publications. Berryhill's scholarship metrics include an h-index of 4 with 5 total publications and 52 total citations.
Metrics
- h-index: 4
- Publications: 5
- Citations: 52
Selected Publications
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Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice (2024)
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CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system (2023)
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Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass (2022)
Collaboration Network
Top Collaborators
- Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
- Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
- Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
- Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
- Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass
- Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass
- Loss of chaperone-mediated autophagy is associated with low vertebral cancellous bone mass
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
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