Katelynn R. Brann
Postdoctoral Fellow
Also affiliated: Virginia Commonwealth University (2021); Virginia Commonwealth University Medical Center (2021)
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Katelynn R. Brann's research focuses on host-pathogen interactions, particularly concerning the bacterium *Coxiella burnetii*, the causative agent of Q fever. Her work investigates how this pathogen influences host cellular processes and explores potential therapeutic interventions. She has examined the role of neurotransmitter system-targeting drugs in inhibiting *Coxiella burnetii* growth in human cells and has explored the contribution of microRNAs to the host's immune response to infection. Brann has also studied the modulation of cellular NF-κB inhibitor p105 by *Orientia tsutsugamushi*. Her scholarship metrics include an h-index of 5, with 7 total publications and 89 total citations. She has collaborated with researchers at the University of Arkansas for Medical Sciences, including Daniel E. Voth, Marissa S. Fullerton, Richard C. Kurten, and Punsiri M. Colonne.
Metrics
- h-index: 5
- Publications: 7
- Citations: 100
Positions
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Postdoctoral Fellow 2020–presentVirginia Commonwealth University Microbiology and Immunology ORCID
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Postdoctoral Fellow publications 2018–2022University of Arkansas for Medical Sciences ORCID
Selected Publications
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MicroRNAs Contribute to Host Response to Coxiella burnetii (2022)
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MicroRNAs contribute to the host response to Coxiella burnetii (2022)
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Neurotransmitter System-Targeting Drugs Antagonize Growth of the Q Fever Agent, Coxiella burnetii, in Human Cells (2021)
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Coxiella burnetii Requires Host Eukaryotic Initiation Factor 2α Activity for Efficient Intracellular Replication (2020)
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Infection of Primary Human Alveolar Macrophages Alters Staphylococcus aureus Toxin Production and Activity (2019)
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Coxiella burnetii Subverts p62/Sequestosome 1 and Activates Nrf2 Signaling in Human Macrophages (2018)
Collaboration Network
Top Collaborators
- Coxiella burnetii Subverts p62/Sequestosome 1 and Activates Nrf2 Signaling in Human Macrophages
- Infection of Primary Human Alveolar Macrophages Alters Staphylococcus aureus Toxin Production and Activity
- Coxiella burnetii Requires Host Eukaryotic Initiation Factor 2α Activity for Efficient Intracellular Replication
- Neurotransmitter System-Targeting Drugs Antagonize Growth of the Q Fever Agent, Coxiella burnetii, in Human Cells
- MicroRNAs Contribute to Host Response to Coxiella burnetii
Showing 5 of 6 shared publications
- Infection of Primary Human Alveolar Macrophages Alters Staphylococcus aureus Toxin Production and Activity
- Coxiella burnetii Requires Host Eukaryotic Initiation Factor 2α Activity for Efficient Intracellular Replication
- Neurotransmitter System-Targeting Drugs Antagonize Growth of the Q Fever Agent, Coxiella burnetii, in Human Cells
- MicroRNAs Contribute to Host Response to Coxiella burnetii
- Coxiella burnetii Subverts p62/Sequestosome 1 and Activates Nrf2 Signaling in Human Macrophages
- Infection of Primary Human Alveolar Macrophages Alters Staphylococcus aureus Toxin Production and Activity
- Neurotransmitter System-Targeting Drugs Antagonize Growth of the Q Fever Agent, Coxiella burnetii, in Human Cells
- Coxiella burnetii Subverts p62/Sequestosome 1 and Activates Nrf2 Signaling in Human Macrophages
- Neurotransmitter System-Targeting Drugs Antagonize Growth of the Q Fever Agent, Coxiella burnetii, in Human Cells
- Coxiella burnetii Subverts p62/Sequestosome 1 and Activates Nrf2 Signaling in Human Macrophages
- Infection of Primary Human Alveolar Macrophages Alters Staphylococcus aureus Toxin Production and Activity
- MicroRNAs Contribute to Host Response to Coxiella burnetii
- MicroRNAs contribute to the host response to Coxiella burnetii
- MicroRNAs Contribute to Host Response to Coxiella burnetii
- MicroRNAs contribute to the host response to Coxiella burnetii
- Coxiella burnetii Subverts p62/Sequestosome 1 and Activates Nrf2 Signaling in Human Macrophages
- Infection of Primary Human Alveolar Macrophages Alters Staphylococcus aureus Toxin Production and Activity
- Infection of Primary Human Alveolar Macrophages Alters Staphylococcus aureus Toxin Production and Activity
- Infection of Primary Human Alveolar Macrophages Alters Staphylococcus aureus Toxin Production and Activity
- Infection of Primary Human Alveolar Macrophages Alters Staphylococcus aureus Toxin Production and Activity
- Neurotransmitter System-Targeting Drugs Antagonize Growth of the Q Fever Agent, Coxiella burnetii, in Human Cells
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