Biography and Research Information
OverviewAI-generated summary
Marissa S. Fullerton investigates host-pathogen interactions, with a focus on the bacterium *Coxiella burnetii*, the causative agent of Q fever. Her research explores how host cells, such as THP-1 cells, respond to infection and examines potential therapeutic strategies. Recent work has investigated the repurposing of neurotransmitter system-targeting drugs for their anti-growth properties against *Coxiella burnetii* in human cells. Fullerton also studies the role of microRNAs in the host's immune response to *Coxiella burnetii*. Her publication record includes work on the genetic manipulation of other bacterial species, such as *Borrelia duttonii*, and the development of new antibacterial derivatives through chemical synthesis. She has published 8 papers, with a total of 69 citations, and holds an h-index of 4. Fullerton has collaborated with researchers at the University of Arkansas for Medical Sciences, including Daniel E. Voth and Katelynn R. Brann.
Metrics
- h-index: 4
- Publications: 8
- Citations: 71
Selected Publications
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Late-Stage Functionalization of the Rifamycin Core via Click Chemistry Toward New Antibacterial Derivatives (2026)
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Development and validation of systems for genetic manipulation of the Old World tick-borne relapsing fever spirochete, Borrelia duttonii (2024)
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MicroRNAs Contribute to Host Response to <i>Coxiella burnetii</i> (2022)
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Neurotransmitter System-Targeting Drugs Antagonize Growth of the Q Fever Agent, Coxiella burnetii, in Human Cells (2021)
Collaboration Network
Top Collaborators
- Neurotransmitter System-Targeting Drugs Antagonize Growth of the Q Fever Agent, Coxiella burnetii, in Human Cells
- MicroRNAs Contribute to Host Response to <i>Coxiella burnetii</i>
- Development and validation of systems for genetic manipulation of the Old World tick-borne relapsing fever spirochete, Borrelia duttonii
- Neurotransmitter System-Targeting Drugs Antagonize Growth of the Q Fever Agent, Coxiella burnetii, in Human Cells
- Late-Stage Functionalization of the Rifamycin Core via Click Chemistry Toward New Antibacterial Derivatives
- Neurotransmitter System-Targeting Drugs Antagonize Growth of the Q Fever Agent, Coxiella burnetii, in Human Cells
- MicroRNAs Contribute to Host Response to <i>Coxiella burnetii</i>
- Neurotransmitter System-Targeting Drugs Antagonize Growth of the Q Fever Agent, Coxiella burnetii, in Human Cells
- Neurotransmitter System-Targeting Drugs Antagonize Growth of the Q Fever Agent, Coxiella burnetii, in Human Cells
- MicroRNAs Contribute to Host Response to <i>Coxiella burnetii</i>
- MicroRNAs Contribute to Host Response to <i>Coxiella burnetii</i>
- Development and validation of systems for genetic manipulation of the Old World tick-borne relapsing fever spirochete, Borrelia duttonii
- Development and validation of systems for genetic manipulation of the Old World tick-borne relapsing fever spirochete, Borrelia duttonii
- Development and validation of systems for genetic manipulation of the Old World tick-borne relapsing fever spirochete, Borrelia duttonii
- Development and validation of systems for genetic manipulation of the Old World tick-borne relapsing fever spirochete, Borrelia duttonii
- Development and validation of systems for genetic manipulation of the Old World tick-borne relapsing fever spirochete, Borrelia duttonii
- Development and validation of systems for genetic manipulation of the Old World tick-borne relapsing fever spirochete, Borrelia duttonii
- Development and validation of systems for genetic manipulation of the Old World tick-borne relapsing fever spirochete, Borrelia duttonii
- Late-Stage Functionalization of the Rifamycin Core via Click Chemistry Toward New Antibacterial Derivatives
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