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OverviewAI-generated summary
Mei Bai's research investigates the molecular mechanisms underlying various disease states, with a focus on the role of natural compounds and specific cellular pathways. Recent work includes studying the direct inhibition of GSDMD by PEITC to reduce hepatocyte pyroptosis and alleviate acute liver injury in mice, as well as the amelioration of chemotherapeutic intestinal injury by PEITC through the suppression of neutrophil extracellular traps. Bai has also explored how STAT3 activates lncRNA NR2F1‐AS1 to promote melanoma progression and how the natural compound fraxinellone intervenes in the HSP47-collagen interaction to ameliorate intestinal fibrosis. Other studies have examined the association of Fc receptor-like variants with rheumatoid arthritis risk and the potential of HINT2 as a clinical target for diabetes mellitus by reducing ROS-induced oxidative stress and ferroptosis. Bai's scholarship is supported by 76 publications, with an h-index of 18 and 1,357 total citations.
Metrics
- h-index: 18
- Publications: 76
- Citations: 1,377
Selected Publications
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Psychological Adaptation to Newly Diagnosed Advanced Cancer (2025)
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Self-affirmation intervention for patients newly diagnosed with advanced cancer: a preliminary efficacy trial (2025)
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Psychological Response to the Diagnosis of Advanced Cancer: A Systematic Review (2021)
Collaboration Network
Top Collaborators
- Direct Inhibition of GSDMD by PEITC Reduces Hepatocyte Pyroptosis and Alleviates Acute Liver Injury in Mice
- Natural compound fraxinellone ameliorates intestinal fibrosis in mice via direct intervention of HSP47-collagen interaction in the epithelium
- Suppression of neutrophil extracellular traps is responsible for the amelioration of chemotherapeutic intestinal injury by the natural compound PEITC
- Direct inhibition of the TXNIP-NLRP3-GSDMD pathway reduces pyroptosis in colonocytes and alleviates ulcerative colitis in mice by the small compound PEITC
- Phenylethyl isothiocyanate mitigates drug-induced liver injury in mice by inhibiting hepatocyte pyroptosis through the NLRP3-caspase-1-GSDMD pathway
Showing 5 of 6 shared publications
- Direct Inhibition of GSDMD by PEITC Reduces Hepatocyte Pyroptosis and Alleviates Acute Liver Injury in Mice
- Natural compound fraxinellone ameliorates intestinal fibrosis in mice via direct intervention of HSP47-collagen interaction in the epithelium
- Suppression of neutrophil extracellular traps is responsible for the amelioration of chemotherapeutic intestinal injury by the natural compound PEITC
- Direct inhibition of the TXNIP-NLRP3-GSDMD pathway reduces pyroptosis in colonocytes and alleviates ulcerative colitis in mice by the small compound PEITC
- Phenylethyl isothiocyanate mitigates drug-induced liver injury in mice by inhibiting hepatocyte pyroptosis through the NLRP3-caspase-1-GSDMD pathway
Showing 5 of 6 shared publications
- Direct Inhibition of GSDMD by PEITC Reduces Hepatocyte Pyroptosis and Alleviates Acute Liver Injury in Mice
- Suppression of neutrophil extracellular traps is responsible for the amelioration of chemotherapeutic intestinal injury by the natural compound PEITC
- Direct inhibition of the TXNIP-NLRP3-GSDMD pathway reduces pyroptosis in colonocytes and alleviates ulcerative colitis in mice by the small compound PEITC
- Phenylethyl isothiocyanate mitigates drug-induced liver injury in mice by inhibiting hepatocyte pyroptosis through the NLRP3-caspase-1-GSDMD pathway
- The natural compound PEITC ameliorates imiquimod-induced psoriasis in mice by suppressing neutrophil extracellular traps formation
- Suppression of neutrophil extracellular traps is responsible for the amelioration of chemotherapeutic intestinal injury by the natural compound PEITC
- Direct inhibition of the TXNIP-NLRP3-GSDMD pathway reduces pyroptosis in colonocytes and alleviates ulcerative colitis in mice by the small compound PEITC
- Phenylethyl isothiocyanate mitigates drug-induced liver injury in mice by inhibiting hepatocyte pyroptosis through the NLRP3-caspase-1-GSDMD pathway
- The natural compound PEITC ameliorates imiquimod-induced psoriasis in mice by suppressing neutrophil extracellular traps formation
- Direct Inhibition of GSDMD by PEITC Reduces Hepatocyte Pyroptosis and Alleviates Acute Liver Injury in Mice
- Natural compound fraxinellone ameliorates intestinal fibrosis in mice via direct intervention of HSP47-collagen interaction in the epithelium
- Direct inhibition of the TXNIP-NLRP3-GSDMD pathway reduces pyroptosis in colonocytes and alleviates ulcerative colitis in mice by the small compound PEITC
- Direct Inhibition of GSDMD by PEITC Reduces Hepatocyte Pyroptosis and Alleviates Acute Liver Injury in Mice
- Natural compound fraxinellone ameliorates intestinal fibrosis in mice via direct intervention of HSP47-collagen interaction in the epithelium
- Direct inhibition of the TXNIP-NLRP3-GSDMD pathway reduces pyroptosis in colonocytes and alleviates ulcerative colitis in mice by the small compound PEITC
- Direct Inhibition of GSDMD by PEITC Reduces Hepatocyte Pyroptosis and Alleviates Acute Liver Injury in Mice
- Phenylethyl isothiocyanate mitigates drug-induced liver injury in mice by inhibiting hepatocyte pyroptosis through the NLRP3-caspase-1-GSDMD pathway
- The natural compound PEITC ameliorates imiquimod-induced psoriasis in mice by suppressing neutrophil extracellular traps formation
- A dual-modality sensing probe of fluorescent and colorimetric for detection of cobalt ion based on silver nanoparticles functionalized rhodamine 6G derivatives
- Colorimetric and Fluorescent Dual-Modality Sensing Platform Based on Functionalized Silver Nanoparticles for Cobalt Ion Detection
- Development of a Dual-Modal Sensing Platform for Cobalt Ion Detection: Combining Colorimetry and Fluorescence with Functionalized Silver Nanoparticles
- A dual-modality sensing probe of fluorescent and colorimetric for detection of cobalt ion based on silver nanoparticles functionalized rhodamine 6G derivatives
- Colorimetric and Fluorescent Dual-Modality Sensing Platform Based on Functionalized Silver Nanoparticles for Cobalt Ion Detection
- Development of a Dual-Modal Sensing Platform for Cobalt Ion Detection: Combining Colorimetry and Fluorescence with Functionalized Silver Nanoparticles
- Natural compound fraxinellone ameliorates intestinal fibrosis in mice via direct intervention of HSP47-collagen interaction in the epithelium
- Suppression of neutrophil extracellular traps is responsible for the amelioration of chemotherapeutic intestinal injury by the natural compound PEITC
- Direct inhibition of the TXNIP-NLRP3-GSDMD pathway reduces pyroptosis in colonocytes and alleviates ulcerative colitis in mice by the small compound PEITC
- Fc receptor-like 1, 3, and 6 variants are associated with rheumatoid arthritis risk in the Chinese Han population
- Age- and gender-dependent association of SLC11A1 polymorphisms with tuberculosis susceptibility
- Fc receptor-like 1, 3, and 6 variants are associated with rheumatoid arthritis risk in the Chinese Han population
- Age- and gender-dependent association of SLC11A1 polymorphisms with tuberculosis susceptibility
- Fc receptor-like 1, 3, and 6 variants are associated with rheumatoid arthritis risk in the Chinese Han population
- Age- and gender-dependent association of SLC11A1 polymorphisms with tuberculosis susceptibility
- Direct Inhibition of GSDMD by PEITC Reduces Hepatocyte Pyroptosis and Alleviates Acute Liver Injury in Mice
- Direct inhibition of the TXNIP-NLRP3-GSDMD pathway reduces pyroptosis in colonocytes and alleviates ulcerative colitis in mice by the small compound PEITC
- Direct Inhibition of GSDMD by PEITC Reduces Hepatocyte Pyroptosis and Alleviates Acute Liver Injury in Mice
- Suppression of neutrophil extracellular traps is responsible for the amelioration of chemotherapeutic intestinal injury by the natural compound PEITC
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