Michael W. Webb

Researcher

Last publication 2026 Last refreshed 2026-04-18

faculty

4 h-index 14 pubs 42 cited

Biography and Research Information

OverviewAI-generated summary

Michael W. Webb's research program investigates the biological mechanisms of platelet function and thrombus formation, with a particular focus on the role of the Syk kinase pathway. His work includes studies on platelet activation, thrombus organization, and the contrasting effects of genetic deletion versus pharmacological inhibition of key platelet receptors and signaling molecules in murine models. Webb has also examined the cost-effectiveness of universal suicide risk screening for adolescents in emergency departments. He has published 14 works with an h-index of 4 and 40 total citations. Key collaborators include Irina D. Pokrovskaya, Brian Storrie, Kelly K. Ball, and Sung W. Rhee, all from the University of Arkansas for Medical Sciences.

Metrics

  • h-index: 4
  • Publications: 14
  • Citations: 42

Selected Publications

  • Murine Thrombus Organization Limits Access to High Platelet Activation States While Supporting Platelet Recruitment (2026)
  • The Syk inhibitor BI 1002494 impairs thrombus infill in a murine femoral artery occlusion without affecting hemostasis (2025)
  • Contrasting Effects of Platelet GPVI Deletion Versus Syk Inhibition on Mouse Jugular Vein Puncture Wound Structure (2025)
  • Contrasting Effects of Platelet GPVI Deletion versus Syk Inhibition on Mouse Jugular Vein Puncture Wound Structure (2025)
  • Differential Effects of GPVI Deletion and SYK Inhibition on Thrombus Organization and Platelet Adhesion in a Murine Jugular Puncture Wound Model (2024)
  • Single-Platelet Mapping of Jugular, Puncture-Wound Thrombi Reveals the Spatial Evolution of Platelet Activation (2024)
    1 citation DOI OpenAlex
  • Screening for Key Structural Differences in Thrombosis Versus Hemostasis through Single Platelet Analysis (2023)
  • Theme and Variation: Structuring Thrombus Formation from Jugular/Arterial Puncture Wounds to Occlusive Clots in a Mouse Model (2022)

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Collaboration Network

22 Collaborators 9 Institutions 1 Country

Top Collaborators

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