Pamela I. Rogers
Researcher
Also affiliated: Rose–Hulman Institute of Technology (2001–2002); Indianapolis Zoo (2009); Plastic Surgery Hospital (2009); Richard L. Roudebush VA Medical Center (2000–2014); Institute of Cardiology (2009); Center for Vascular Biology Research (2009–2014); University School (2001); Indiana University Indianapolis (2014); Indiana University School of Medicine (1994–2014); Indiana University – Purdue University Indianapolis (1994–2015); Indiana University (2001–2009)
Faculty Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Pamela I. Rogers' research investigates the complex interplay of immune responses in chronic diseases, including hypertension, type 2 diabetes, and cardiovascular complications. Her recent publications explore the role of specific immune cells and molecular pathways in disease progression. For instance, her work examines how IL-10 influences macrophage transition and promotes fibrosis in failing hearts, and how P2X7 receptors drive T-cell activation during high blood pressure and obesity-induced type 2 diabetes. Rogers also studies the contribution of kidney-resident memory T cells to chronic hypertension and explores immune dysregulation as a connecting factor between type 2 diabetes and cardiovascular issues. Collaborating with Yunmeng Liu, Katherine Deck, Christoph Mora, and Shuoqiu Deng at the University of Arkansas for Medical Sciences, Rogers has co-authored nine publications with each of them. Her work has resulted in 26 publications, garnering 1,138 citations and an h-index of 12.
Metrics
- h-index: 12
- Publications: 26
- Citations: 1,146
Selected Publications
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Macrophage Plasticity Drives Cardiac Dysfunction in Hypertension 2253031 (2026)
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Antigen-independent formation of renal Trm-like CD8 T cells perpetuates hypertension 2259853 (2026)
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Kidney Resident Memory CD8+ T cells drive the immortality of hypertension (2026)
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Cytokine-mediated macrophage transition promotes early cardiac fibrosis in chronic hypertension (2026)
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IL-10 Drives Macrophage to Myofibroblast Transition Promoting Chronic Fibrosis in the Failing Heart 9287 (2025)
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P2X7 Drives T-cell Activation Promoting Chronic Inflammation During High Blood Pressure 9289 (2025)
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Kidney resident-memory CD8+ T cells drive chronic high blood pressure 9288 (2025)
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Energy Overload: ATP-P2X7-Mediated T Cell Activation in Obesity-Induced Type 2 Diabetes 9286 (2025)
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Kidney Memory for Salt Sensitivity Resides in T Cells Driving Chronic Hypertension (2025)
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Uncovering immune pathways for therapeutic targeting of hypertension (2025)
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Immune Dysregulation Connecting Type 2 Diabetes and Cardiovascular Complications (2025)
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Cytokine-induced Macrophage Transition Drives Cardiac Fibrosis and Diastolic Dysfunction (2025)
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Establishment of resident memory T cells anchors hypertension in the kidney (2025)
Collaboration Network
Top Collaborators
- Establishment of resident memory T cells anchors hypertension in the kidney
- Cytokine-induced Macrophage Transition Drives Cardiac Fibrosis and Diastolic Dysfunction
- Immune Dysregulation Connecting Type 2 Diabetes and Cardiovascular Complications
- Uncovering immune pathways for therapeutic targeting of hypertension
- Kidney Memory for Salt Sensitivity Resides in T Cells Driving Chronic Hypertension
Showing 5 of 9 shared publications
- Establishment of resident memory T cells anchors hypertension in the kidney
- Cytokine-induced Macrophage Transition Drives Cardiac Fibrosis and Diastolic Dysfunction
- Immune Dysregulation Connecting Type 2 Diabetes and Cardiovascular Complications
- Uncovering immune pathways for therapeutic targeting of hypertension
- Kidney Memory for Salt Sensitivity Resides in T Cells Driving Chronic Hypertension
Showing 5 of 9 shared publications
- Establishment of resident memory T cells anchors hypertension in the kidney
- Cytokine-induced Macrophage Transition Drives Cardiac Fibrosis and Diastolic Dysfunction
- Immune Dysregulation Connecting Type 2 Diabetes and Cardiovascular Complications
- Uncovering immune pathways for therapeutic targeting of hypertension
- Kidney Memory for Salt Sensitivity Resides in T Cells Driving Chronic Hypertension
Showing 5 of 9 shared publications
- Establishment of resident memory T cells anchors hypertension in the kidney
- Cytokine-induced Macrophage Transition Drives Cardiac Fibrosis and Diastolic Dysfunction
- Immune Dysregulation Connecting Type 2 Diabetes and Cardiovascular Complications
- Uncovering immune pathways for therapeutic targeting of hypertension
- Kidney Memory for Salt Sensitivity Resides in T Cells Driving Chronic Hypertension
Showing 5 of 9 shared publications
- Establishment of resident memory T cells anchors hypertension in the kidney
- Cytokine-induced Macrophage Transition Drives Cardiac Fibrosis and Diastolic Dysfunction
- Immune Dysregulation Connecting Type 2 Diabetes and Cardiovascular Complications
- Uncovering immune pathways for therapeutic targeting of hypertension
- Kidney Memory for Salt Sensitivity Resides in T Cells Driving Chronic Hypertension
Showing 5 of 9 shared publications
- Establishment of resident memory T cells anchors hypertension in the kidney
- Cytokine-induced Macrophage Transition Drives Cardiac Fibrosis and Diastolic Dysfunction
- Immune Dysregulation Connecting Type 2 Diabetes and Cardiovascular Complications
- Uncovering immune pathways for therapeutic targeting of hypertension
- Kidney resident-memory CD8+ T cells drive chronic high blood pressure 9288
Showing 5 of 6 shared publications
- Kidney Memory for Salt Sensitivity Resides in T Cells Driving Chronic Hypertension
- Energy Overload: ATP-P2X7-Mediated T Cell Activation in Obesity-Induced Type 2 Diabetes 9286
- P2X7 Drives T-cell Activation Promoting Chronic Inflammation During High Blood Pressure 9289
- Immune Dysregulation Connecting Type 2 Diabetes and Cardiovascular Complications
- Uncovering immune pathways for therapeutic targeting of hypertension
- Establishment of resident memory T cells anchors hypertension in the kidney
- Establishment of resident memory T cells anchors hypertension in the kidney
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