Biography and Research Information
OverviewAI-generated summary
Piyanan Chuesiang's research focuses on the development and characterization of various delivery systems for bioactive compounds, with a particular emphasis on enhancing stability and bioavailability. Chuesiang has investigated nanostructured lipid carriers, oleogels, and Pickering emulsions stabilized by complex biopolymers like cellulose nanocrystals and whey protein isolate for delivering compounds such as curcumin.
Further work includes exploring the effects of cold plasma treatment on chitosan films to improve their properties and developing green active coatings from chitosan incorporated with cinnamon oil nanoemulsion for food preservation. Chuesiang has also studied microfluidized essential oil nanoemulsions for their antimicrobial activity and the use of micellar delivery systems to enhance curcumin's stability and photoinactivation capacity. The researcher's work has resulted in 30 publications with an h-index of 16 and 884 total citations.
Metrics
- h-index: 16
- Publications: 30
- Citations: 884
Selected Publications
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CD5L promotes efferocytosis and resolution of retinal ischemic injury (2026)
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Myeloid HDAC3 deletion protects against traumatic optic injury (2026)
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HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis (2025)
Collaboration Network
Top Collaborators
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- Myeloid HDAC3 deletion protects against traumatic optic injury
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- Myeloid HDAC3 deletion protects against traumatic optic injury
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- Myeloid HDAC3 deletion protects against traumatic optic injury
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- Myeloid HDAC3 deletion protects against traumatic optic injury
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
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