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Biography and Research Information
OverviewAI-generated summary
Zijing Zhang's research investigates the molecular and cellular mechanisms underlying various disease states, with a particular focus on immune responses and cellular signaling. Zhang has published work examining the role of neutrophils as a niche for Mycobacterium tuberculosis and the protective mechanisms conferred by monocyte-derived alveolar macrophages against this bacterium. Further research includes single-cell analysis of ovarian myeloid cells to identify age-associated changes in macrophages and signaling dynamics. Zhang also studies the impact of SARS-CoV-2 infection on disease severity in mouse models of Down syndrome and explores the molecular characteristics and immune microenvironment of gastrointestinal stromal tumors as potential therapeutic targets. Zhang's work has resulted in 12 publications with 299 citations and an h-index of 6. Key collaborators include Lu Huang, Lin-Xi Li, J. Tucker Andrews, and Ananya Ranaraja, all from the University of Arkansas for Medical Sciences.
Metrics
- h-index: 7
- Publications: 12
- Citations: 311
Selected Publications
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β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395 (2025)
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Single-cell analysis of ovarian myeloid cells identifies age-associated changes in macrophages and signaling dynamics (2025)
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Single-cell analysis of ovarian myeloid cells identifies aging associated changes in macrophages and signaling dynamics (2024)
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Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis (2024)
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SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome (2024)
Collaboration Network
Top Collaborators
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Single-cell analysis of ovarian myeloid cells identifies age-associated changes in macrophages and signaling dynamics
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Single-cell analysis of ovarian myeloid cells identifies age-associated changes in macrophages and signaling dynamics
- Single-cell analysis of ovarian myeloid cells identifies aging associated changes in macrophages and signaling dynamics
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
- Single-cell analysis of ovarian myeloid cells identifies age-associated changes in macrophages and signaling dynamics
- Single-cell analysis of ovarian myeloid cells identifies aging associated changes in macrophages and signaling dynamics
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
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