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Listed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-08
T
Tsigereda Weldemichael
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Also affiliated: Tianjin University (2022)
5 h-index
8 pubs
146 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Tsigereda Weldemichael's research investigates mechanisms of drug delivery and therapeutic interventions, with a focus on kidney diseases and cancer. Her work includes studying the CD36-mediated endocytosis of proteolysis-targeting chimeras and the delivery efficiency of oligonucleotides using lipid conjugation. Weldemichael has also explored renoprotective approaches against anthracycline nephrotoxicity and the impact of specific compounds, such as EET-based therapeutics, on mitigating drug-induced cellular damage in the kidneys. Her recent publications also touch on synthetic biology applications and industrial uses of enzymes. Weldemichael collaborates with researchers at the University of Arkansas for Medical Sciences, including Abdul H. Khan and Phuc Tran, and at the University of Arkansas at Fayetteville, including Samaneh Goorani.
Metrics
- h-index: 5
- Publications: 8
- Citations: 146
Selected Publications
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Renal hemodynamics underlie hepatorenal physiology in bile duct-ligated rats (2026)
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Renoprotective approaches against anthracycline nephrotoxicity (2026)
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EET-Based Therapeutics Mitigate Sorafenib-Associated Glomerular Cell Damage (2025)
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Insect‐Microbe‐Based Laccase: Untapped Natural Resource for Industrial and Biotechnological Applications (2025)
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CD36-mediated endocytosis of proteolysis-targeting chimeras (2025)
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An imidazo[1,2-a]pyridine-pyridine derivative potently inhibits FLT3-ITD and FLT3-ITD secondary mutants, including gilteritinib-resistant FLT3-ITD/F691L (2023)
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Delivery of Oligonucleotides: Efficiency with Lipid Conjugation and Clinical Outcome (2022)
Collaboration Network
Top Collaborators
Phuc Tran
University of Arkansas for Medical Sciences
3 shared publications
In database
- CD36-mediated endocytosis of proteolysis-targeting chimeras
- Delivery of Oligonucleotides: Efficiency with Lipid Conjugation and Clinical Outcome
- An imidazo[1,2-a]pyridine-pyridine derivative potently inhibits FLT3-ITD and FLT3-ITD secondary mutants, including gilteritinib-resistant FLT3-ITD/F691L
Hongyu Li
The University of Texas at San Antonio Health Science Center (US)
3 shared publications
- CD36-mediated endocytosis of proteolysis-targeting chimeras
- Delivery of Oligonucleotides: Efficiency with Lipid Conjugation and Clinical Outcome
- An imidazo[1,2-a]pyridine-pyridine derivative potently inhibits FLT3-ITD and FLT3-ITD secondary mutants, including gilteritinib-resistant FLT3-ITD/F691L
John D. Imig
Medical College of Wisconsin
3 shared publications
In database
- Renoprotective approaches against anthracycline nephrotoxicity
- Renal hemodynamics underlie hepatorenal physiology in bile duct-ligated rats
- EET-Based Therapeutics Mitigate Sorafenib-Associated Glomerular Cell Damage
Samaneh Goorani
University of Arkansas for Medical Sciences
2 shared publications
In database
- Renal hemodynamics underlie hepatorenal physiology in bile duct-ligated rats
- EET-Based Therapeutics Mitigate Sorafenib-Associated Glomerular Cell Damage
Md. Abdul Hye Khan
University of Arkansas for Medical Sciences
2 shared publications
In database
- Renoprotective approaches against anthracycline nephrotoxicity
- Renal hemodynamics underlie hepatorenal physiology in bile duct-ligated rats
Zhichao Liu
National Center for Toxicological Research (US)
1 shared publication
- Delivery of Oligonucleotides: Efficiency with Lipid Conjugation and Clinical Outcome
Xiuqi Wang
University of Arkansas for Medical Sciences
1 shared publication
In database
- An imidazo[1,2-a]pyridine-pyridine derivative potently inhibits FLT3-ITD and FLT3-ITD secondary mutants, including gilteritinib-resistant FLT3-ITD/F691L
Rosa Anna DeFilippis
University of California, San Francisco (US)
1 shared publication
- An imidazo[1,2-a]pyridine-pyridine derivative potently inhibits FLT3-ITD and FLT3-ITD secondary mutants, including gilteritinib-resistant FLT3-ITD/F691L
Gunaganti Naresh
University of Arkansas for Medical Sciences
1 shared publication
In database
- An imidazo[1,2-a]pyridine-pyridine derivative potently inhibits FLT3-ITD and FLT3-ITD secondary mutants, including gilteritinib-resistant FLT3-ITD/F691L
Yuet‐Kin Leung
University of Arkansas for Medical Sciences
1 shared publication
In database
- An imidazo[1,2-a]pyridine-pyridine derivative potently inhibits FLT3-ITD and FLT3-ITD secondary mutants, including gilteritinib-resistant FLT3-ITD/F691L
Neil P. Shah
University of California, San Francisco (US)
1 shared publication
- An imidazo[1,2-a]pyridine-pyridine derivative potently inhibits FLT3-ITD and FLT3-ITD secondary mutants, including gilteritinib-resistant FLT3-ITD/F691L
Zheng‐Yu Wang
The University of Texas at San Antonio Health Science Center
1 shared publication
In database
- CD36-mediated endocytosis of proteolysis-targeting chimeras
Bo‐Syong Pan
Wake Forest University (US)
1 shared publication
- CD36-mediated endocytosis of proteolysis-targeting chimeras
Rajesh Manne
Wake Forest University (US)
1 shared publication
- CD36-mediated endocytosis of proteolysis-targeting chimeras
Jungang Chen
Winthrop Rockefeller Foundation (US)
1 shared publication
- CD36-mediated endocytosis of proteolysis-targeting chimeras
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