Match tier
Listed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-08-15
K
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Kara A. O’Neal's research investigates host-pathogen interactions, specifically focusing on the immune response to malaria caused by *Plasmodium yoelii* in mouse models. Her work has explored the role of specific T cell subsets and cytokines in controlling infection. A key area of her investigation is the function of Bhlhe40 in limiting early Interleukin-10 (IL-10) production from CD4+ T cells during *Plasmodium yoelii* infection. Additionally, her research has examined the involvement of ICOS (Inducible T-cell costimulator) expression in the maintenance of germinal centers within the spleen following *P. yoelii* infection. O’Neal’s publications include studies on the independence of early parasite control from interferon-gamma (IFN-γ), IL-12, and humoral responses in IL-10-deficient mice. She collaborates with researchers at the University of Arkansas for Medical Sciences and the National Center for Toxicological Research.
Metrics
- h-index: 2
- Publications: 4
- Citations: 8
Selected Publications
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Early enhanced control of <i>Plasmodium yoelii</i> infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response (2026)
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Bhlhe40 limits early IL-10 production from CD4 <sup>+</sup> T cells during <i>Plasmodium yoelii</i> 17X infection (2023)
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ICOS Expression Is Required for Maintenance but Not the Formation of Germinal Centers in the Spleen in Response to Plasmodium yoelii Infection (2022)
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ICOS expression is required for maintenance but not the formation of germinal centers in the spleen in response to <i>P. yoelii</i> infection (2021)
Collaboration Network
Top Collaborators
Jason S. Stumhofer
University of Arkansas for Medical Sciences
4 shared publications
In database
- Bhlhe40 limits early IL-10 production from CD4 <sup>+</sup> T cells during <i>Plasmodium yoelii</i> 17X infection
- ICOS Expression Is Required for Maintenance but Not the Formation of Germinal Centers in the Spleen in Response to Plasmodium yoelii Infection
- ICOS expression is required for maintenance but not the formation of germinal centers in the spleen in response to <i>P. yoelii</i> infection
- Early enhanced control of <i>Plasmodium yoelii</i> infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response
Camille L. Foscue
University of Arkansas for Medical Sciences
3 shared publications
In database
- Bhlhe40 limits early IL-10 production from CD4 <sup>+</sup> T cells during <i>Plasmodium yoelii</i> 17X infection
- ICOS Expression Is Required for Maintenance but Not the Formation of Germinal Centers in the Spleen in Response to Plasmodium yoelii Infection
- ICOS expression is required for maintenance but not the formation of germinal centers in the spleen in response to <i>P. yoelii</i> infection
Leah E. Latham
University of Arkansas for Medical Sciences
2 shared publications
In database
- ICOS Expression Is Required for Maintenance but Not the Formation of Germinal Centers in the Spleen in Response to Plasmodium yoelii Infection
- ICOS expression is required for maintenance but not the formation of germinal centers in the spleen in response to <i>P. yoelii</i> infection
Enatha Ntirandekura
University of Arkansas for Medical Sciences
2 shared publications
In database
- ICOS Expression Is Required for Maintenance but Not the Formation of Germinal Centers in the Spleen in Response to Plasmodium yoelii Infection
- ICOS expression is required for maintenance but not the formation of germinal centers in the spleen in response to <i>P. yoelii</i> infection
Sheldon L. Zeltner
University of Arkansas for Medical Sciences
2 shared publications
In database
- Bhlhe40 limits early IL-10 production from CD4 <sup>+</sup> T cells during <i>Plasmodium yoelii</i> 17X infection
- Early enhanced control of <i>Plasmodium yoelii</i> infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response
Alison Gouch
University of Arkansas for Medical Sciences
1 shared publication
In database
- Early enhanced control of <i>Plasmodium yoelii</i> infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response
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Based on overlapping research topics
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