Nathan Serrano
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Postdoc Fellow, University of Missouri–Columbia
Also affiliated: California State University, Fullerton (2018–2019); Arizona State University (2019–2026); University of Missouri (2026)
Formerly Arkansas Postdoc Scholar, University of Arkansas through 2026; now Postdoc Fellow, University of Missouri–Columbia.
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Nathan Serrano's research investigates the physiological adaptations and molecular mechanisms underlying skeletal muscle function and adaptation in various contexts, including elite athletic performance, obesity, and cancer cachexia. His work has explored the abundance of fast-twitch muscle fibers in elite weightlifters and the role of muscle fiber phenotype in abnormal metabolism associated with obesity. Serrano has also examined mitochondrial protein synthesis during exercise and recovery in fasting states, irrespective of obesity, and has contributed to understanding the role of MYC in skeletal muscle. His research extends to the impact of exercise on muscle mass and tumor growth in cancer models and has investigated cardiovascular adaptations in transgender athletes. Serrano's scholarship metrics include an h-index of 4 with 112 citations across 28 publications. He has collaborated with researchers such as Pieter J. Koopmans and Kevin A. Murach at the University of Arkansas at Fayetteville.
Metrics
- h-index: 4
- Publications: 42
- Citations: 118
Positions
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Postdoc Fellow 2026–presentUniversity of Missouri–Columbia ORCID
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Postdoc Scholar 2025–2026University of Arkansas at Fayetteville Human Health Performance and Recreation ORCID
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TA 2019–2024Arizona State University School of Life Sciences ORCID
Selected Publications
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Myonuclear Dynamics After Skeletal Muscle Surgical Injury (2026)
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Transient MYC Mimicking the Exercise Response Orchestrates Multifaceted Skeletal Muscle Adaptations (2026)
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Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia (2026)
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Myonuclear Dynamics After Skeletal Muscle Surgical Injury (2026)
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Myonuclear Dynamics After Skeletal Muscle Surgical Resection Injury (2026)Journal of the Arkansas Academy of Science OpenAlex
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Displaced myonuclei are attributable to both resident myonuclear migration and stem cell fusion during mechanical loading in adult skeletal muscle (2025)
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Displaced myonuclei are attributable to both resident myonuclear migration and stem cell fusion during mechanical loading in adult skeletal muscle (2025)
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Muscle memory theory: A critical evaluation (2025)
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Making sense of MYC in skeletal muscle: location, duration, and magnitude (2025)
Collaboration Network
Top Collaborators
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- Muscle memory theory: A critical evaluation
- Displaced myonuclei are attributable to both resident myonuclear migration and stem cell fusion during mechanical loading in adult skeletal muscle
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Displaced myonuclei are attributable to both resident myonuclear migration and stem cell fusion during mechanical loading in adult skeletal muscle
Showing 5 of 8 shared publications
- Displaced myonuclei are attributable to both resident myonuclear migration and stem cell fusion during mechanical loading in adult skeletal muscle
- Displaced myonuclei are attributable to both resident myonuclear migration and stem cell fusion during mechanical loading in adult skeletal muscle
- Myonuclear Dynamics After Skeletal Muscle Surgical Resection Injury
- Myonuclear Dynamics After Skeletal Muscle Surgical Injury
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- Transient MYC Mimicking the Exercise Response Orchestrates Multifaceted Skeletal Muscle Adaptations
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Myonuclear Dynamics After Skeletal Muscle Surgical Injury
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Transient MYC Mimicking the Exercise Response Orchestrates Multifaceted Skeletal Muscle Adaptations
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- Muscle memory theory: A critical evaluation
- Myonuclear Dynamics After Skeletal Muscle Surgical Resection Injury
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
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