Ana Regina Cabrera Source Confirmed

Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.

Graduate Assistant

University of Arkansas at Fayetteville

faculty

8 h-index 36 pubs 163 cited

Research Areas

Cancer-related molecular mechanisms research Muscle Physiology and Disorders Diet and metabolism studies Mitochondrial Function and Pathology MicroRNA in disease regulation Health Sciences Research and Education Birth, Development, and Health

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ORCID Research Group

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OverviewAI-generated summary

Ana Regina Cabrera's research investigates molecular and physiological alterations associated with cancer cachexia, a complex metabolic syndrome characterized by involuntary weight loss and muscle wasting. Her work has explored the time-course of cachexia onset in preclinical models, identifying biphasic transcriptional disruptions in skeletal muscle that differ between sexes. Cabrera has published findings indicating that females may exhibit relatively preserved muscle quality compared to males during the early stages of cancer cachexia. Further research has examined the role of specific microRNAs, such as miR-16, in regulating insulin sensitivity and muscle contractile function in a sex-dependent manner. Additionally, her publications address the impact of mitochondrial antioxidants, like SkQ1, on mitigating muscle wasting and improving contractile function in tumor-bearing mice, as well as the mechanosensitive gene arrestin domain containing 2 in relation to disuse atrophy.

Metrics

h-index: 8
Publications: 36
Citations: 163

Selected Publications

Skeletal muscle methylome-transcriptome disruptions during the onset and progression of colorectal cancer-induced cachexia (2025) DOI
The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus (2025) DOI
Transcriptomic analysis demonstrates moderators of muscle quality are altered in age-related sarcopenic obesity (2025) DOI
Aerobic Exercise Training Does Not Attenuate Fibrosis In Autologous Repaired Vml-injured Skeletal Muscle (2025) DOI
Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females (2025) DOI
Landscape of clinical trials in cancer cachexia: assessment of trends from 1995–2024 (2025) DOI
Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice (2025) DOI
Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice (2025) DOI
Landscape of Clinical Trials in Cancer Cachexia: Assessment of Trends From 1995-2024 (2025) DOI
Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex (2024) DOI
Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice (2024) DOI
Exploring heterogeneity: a dive into preclinical models of cancer cachexia (2024) DOI
The mechanosensitive gene <i>arrestin domain containing 2</i> regulates myotube diameter with direct implications for disuse atrophy with aging (2024) DOI
Leucine Supplementation Exacerbates Morbidity in Male but Not Female Mice with Colorectal Cancer-Induced Cachexia (2023) DOI
Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy (2023) DOI