Match tier
Confirmed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-08-15
P
Pieter J. Koopmans
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Graduate Research Assistant
Also affiliated: Appalachian State University (2021–2023)
Graduate Student Researcher
6 h-index
20 pubs
109 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Pieter J. Koopmans' research investigates molecular adaptations in skeletal muscle, particularly in response to exercise and disease states. His work has explored the role of microRNAs (miRNAs) in adult skeletal muscle satellite cells, examining how specific miRNAs, such as miR-16, influence the myogenic cell transcriptome and proteome. Koopmans has also studied the effects of exercise on muscle growth, with recent publications implicating MYC as a sufficient regulator for hypertrophy following resistance exercise in humans. His research extends to animal models, investigating the impact of interventions like the mitochondrial antioxidant SkQ1 on cancer-induced muscle wasting and contractility in mice. Additionally, his work has delved into the intersection of epigenetics and cellular senescence, examining the effects of senolytic administration on DNA methylation in aged and regenerated skeletal muscle.
Metrics
- h-index: 6
- Publications: 20
- Citations: 109
Selected Publications
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Myonuclear Dynamics After Skeletal Muscle Surgical Injury (2026)
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Transient MYC Mimicking the Exercise Response Orchestrates Multifaceted Skeletal Muscle Adaptations (2026)
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Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia (2026)
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Myonuclear Dynamics After Skeletal Muscle Surgical Injury (2026)
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Resident myonuclear adaptations to mechanical overload in single muscle fibers of HSA-GFP mice (2026)
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TRAIL: A potential new biomarker for muscle health in anorexia nervosa (2026)
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Ribosome dynamics during skeletal muscle repair and regeneration in mice and humans (2026)
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Myonuclear Dynamics After Skeletal Muscle Surgical Resection Injury (2026)Journal of the Arkansas Academy of Science OpenAlex
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The Age‐Dependent Resident Myonuclear Multi‐Omic Response to an Acute Skeletal Muscle Hypertrophic Stimulus in Mice (2026)
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Displaced myonuclei are attributable to both resident myonuclear migration and stem cell fusion during mechanical loading in adult skeletal muscle (2025)
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The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus (2025)
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Displaced myonuclei are attributable to both resident myonuclear migration and stem cell fusion during mechanical loading in adult skeletal muscle (2025)
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Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females (2025)
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Corrigendum to “microRNA-1 regulates metabolic flexibility by programming skeletal muscle pyruvate metabolism” [Mol Metabol 98 (2025) 1–23/102182] (2025)
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microRNA-1 regulates metabolic flexibility by programming adult skeletal muscle pyruvate metabolism (2025)
Collaboration Network
Top Collaborators
Kevin A. Murach
University of Arkansas at Fayetteville
18 shared publications
In database
- The roles of miRNAs in adult skeletal muscle satellite cells
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Going nuclear: Molecular adaptations to exercise mediated by myonuclei
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- MicroRNA control of the myogenic cell transcriptome and proteome: the role of miR-16
Showing 5 of 18 shared publications
Francielly Morena da Silva
University of Arkansas at Fayetteville
9 shared publications
In database
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- MicroRNA control of the myogenic cell transcriptome and proteome: the role of miR-16
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- At the Nexus Between Epigenetics and Senescence: The Effects of Senolytic ( <scp>BI01</scp> ) Administration on <scp>DNA</scp> Methylation Clock Age and the Methylome in Aged and Regenerated Skeletal Muscle
Showing 5 of 9 shared publications
Nicholas P. Greene
University of Arkansas at Fayetteville
9 shared publications
In database
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- MicroRNA control of the myogenic cell transcriptome and proteome: the role of miR-16
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- At the Nexus Between Epigenetics and Senescence: The Effects of Senolytic ( <scp>BI01</scp> ) Administration on <scp>DNA</scp> Methylation Clock Age and the Methylome in Aged and Regenerated Skeletal Muscle
Showing 5 of 9 shared publications
Ahmed Ismaeel
Auburn University (US)
6 shared publications
- The roles of miRNAs in adult skeletal muscle satellite cells
- microRNA-1 Regulates Metabolic Flexibility in Skeletal Muscle via Pyruvate Metabolism
- microRNA-1 regulates metabolic flexibility by programming adult skeletal muscle pyruvate metabolism
- The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus
- Corrigendum to “microRNA-1 regulates metabolic flexibility by programming skeletal muscle pyruvate metabolism” [Mol Metabol 98 (2025) 1–23/102182]
Showing 5 of 6 shared publications
Sabin Khadgi
University of Arkansas at Fayetteville
6 shared publications
In database
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
Showing 5 of 6 shared publications
Ana Regina Cabrera
University of Arkansas at Fayetteville
6 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Showing 5 of 6 shared publications
Ronald G. Jones
University of Arkansas at Fayetteville
5 shared publications
In database
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus
- Insight on the loading-mediated regulation of Runx1 in skeletal muscle
- The Age‐Dependent Resident Myonuclear Multi‐Omic Response to an Acute Skeletal Muscle Hypertrophic Stimulus in Mice
Calvin Peterson
University of Arkansas at Fayetteville
5 shared publications
In database
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Seongkyun Lim
University of Arkansas at Fayetteville
4 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- MicroRNA control of the myogenic cell transcriptome and proteome: the role of miR-16
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Ferdinand von Walden
Karolinska Institutet (SE)
4 shared publications
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- MicroRNA control of the myogenic cell transcriptome and proteome: the role of miR-16
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- Insight on the loading-mediated regulation of Runx1 in skeletal muscle
Katarzyna Goljanek‐Whysall
Ollscoil na Gaillimhe – University of Galway (IE)
4 shared publications
- The roles of miRNAs in adult skeletal muscle satellite cells
- microRNA-1 Regulates Metabolic Flexibility in Skeletal Muscle via Pyruvate Metabolism
- microRNA-1 regulates metabolic flexibility by programming adult skeletal muscle pyruvate metabolism
- Corrigendum to “microRNA-1 regulates metabolic flexibility by programming skeletal muscle pyruvate metabolism” [Mol Metabol 98 (2025) 1–23/102182]
Yuan Wen
University of Kentucky (US)
4 shared publications
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- At the Nexus Between Epigenetics and Senescence: The Effects of Senolytic ( <scp>BI01</scp> ) Administration on <scp>DNA</scp> Methylation Clock Age and the Methylome in Aged and Regenerated Skeletal Muscle
- The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus
John J. McCarthy
University of Kentucky (US)
4 shared publications
- microRNA-1 Regulates Metabolic Flexibility in Skeletal Muscle via Pyruvate Metabolism
- microRNA-1 regulates metabolic flexibility by programming adult skeletal muscle pyruvate metabolism
- The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus
- Corrigendum to “microRNA-1 regulates metabolic flexibility by programming skeletal muscle pyruvate metabolism” [Mol Metabol 98 (2025) 1–23/102182]
Stavroula Tsitkanou
University of Arkansas at Fayetteville
4 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Ruqaiza Muhyudin
University of Arkansas at Fayetteville
4 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
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