R
Ronald G. Jones Data-verified
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Researcher
faculty
17 h-index
60 pubs
919 cited
Research Areas
Biography and Research Information
OverviewAI-generated summary
Ronald G. Jones' research investigates molecular and physiological adaptations in skeletal muscle, with a particular focus on aging, exercise, and disease states like cancer cachexia. His work utilizes transcriptomic analysis to identify molecular signatures associated with these conditions. Recent publications explore how partial reprogramming and exercise impact skeletal muscle adaptation with aging, the distinct time courses of transcriptional disruptions in male and female skeletal muscle during cancer cachexia, and the role of MYC in regulating muscle growth following resistance exercise in humans. Jones has also examined neuromuscular dysfunction in mouse models of Alzheimer's disease and the interplay between muscle weakness, mitochondrial stress, and metastasis in ovarian cancer cachexia models. His scholarship includes 58 publications with 913 citations and an h-index of 17. He collaborates extensively with researchers at the University of Arkansas at Fayetteville, including Francielly Morena da Silva, Nicholas P. Greene, Kevin A. Murach, and Ana Regina Cabrera.
Metrics
- h-index: 17
- Publications: 60
- Citations: 919
Selected Publications
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The Age‐Dependent Resident Myonuclear Multi‐Omic Response to an Acute Skeletal Muscle Hypertrophic Stimulus in Mice (2026)
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Myonuclear loss, rather than senescent myonuclei, associates with fiber type-specific atrophy in aging human skeletal muscle (2026)
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Skeletal muscle methylome-transcriptome disruptions during the onset and progression of colorectal cancer-induced cachexia (2025)
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The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus (2025)
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Transcriptomic analysis demonstrates moderators of muscle quality are altered in age-related sarcopenic obesity (2025)
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Making sense of MYC in skeletal muscle: location, duration, and magnitude (2025)
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The Skeletal Muscle Proteomic Signature of Long-Term Repeated Transient MYC Induction (2025)
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The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth (2024)
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Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex (2024)
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Muscle weakness and mitochondrial stress occur before severe metastasis in a novel mouse model of ovarian cancer cachexia (2024)
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Muscle weakness and mitochondrial stress occur before metastasis in a novel mouse model of ovarian cancer cachexia (2024)
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The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth (2024)
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Exercise-Induced MYC as an Epigenetic Reprogramming Factor That Combats Skeletal Muscle Aging (2024)
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Neuromuscular Dysfunction Precedes Cognitive Impairment in a Mouse Model of Alzheimer’s Disease (2023)
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The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males (2023)
Collaboration Network
Top Collaborators
Kevin A. Murach
University of Arkansas at Fayetteville
20 shared publications
In database
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Neuromuscular Dysfunction Precedes Cognitive Impairment in a Mouse Model of Alzheimer’s Disease
- Muscle weakness and mitochondrial stress occur before severe metastasis in a novel mouse model of ovarian cancer cachexia
Showing 5 of 20 shared publications
Nicholas P. Greene
University of Arkansas at Fayetteville
13 shared publications
In database
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Muscle weakness and mitochondrial stress occur before severe metastasis in a novel mouse model of ovarian cancer cachexia
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
Showing 5 of 13 shared publications
Francielly Morena da Silva
University of Arkansas at Fayetteville
11 shared publications
In database
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
Showing 5 of 11 shared publications
Ana Regina Cabrera
University of Arkansas at Fayetteville
7 shared publications
In database
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- The Time-Course of Cancer Cachexia Onset Reveals Biphasic Transcriptional Disruptions in Female Skeletal Muscle Distinct from Males
- The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus
- Transcriptomic analysis demonstrates moderators of muscle quality are altered in age-related sarcopenic obesity
Showing 5 of 7 shared publications
Yuan Wen
University of Kentucky (US)
7 shared publications
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus
Showing 5 of 7 shared publications
Tyrone A. Washington
University of Arkansas at Fayetteville
6 shared publications
In database
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- The Time-Course of Cancer Cachexia Onset Reveals Biphasic Transcriptional Disruptions in Female Skeletal Muscle Distinct from Males
- Mitochondria-Targeted Antioxidant SkQ1 Improves Muscle Contractility in Female C26 Tumor-Bearing Mice
- Transcriptomic analysis demonstrates moderators of muscle quality are altered in age-related sarcopenic obesity
Showing 5 of 6 shared publications
Seongkyun Lim
University of Arkansas at Fayetteville
5 shared publications
In database
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- The Time-Course of Cancer Cachexia Onset Reveals Biphasic Transcriptional Disruptions in Female Skeletal Muscle Distinct from Males
- A Rejuvenation Signature in Skeletal Muscle That Is Mediated By Late-Life Exercise
- Mitochondria-Targeted Antioxidant SkQ1 Improves Muscle Contractility in Female C26 Tumor-Bearing Mice
Eleanor R. Schrems
University of Arkansas at Fayetteville
5 shared publications
In database
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- The Time-Course of Cancer Cachexia Onset Reveals Biphasic Transcriptional Disruptions in Female Skeletal Muscle Distinct from Males
- Transcriptomic analysis demonstrates moderators of muscle quality are altered in age-related sarcopenic obesity
- Skeletal muscle methylome-transcriptome disruptions during the onset and progression of colorectal cancer-induced cachexia
Sabin Khadgi
University of Arkansas at Fayetteville
5 shared publications
In database
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- A Rejuvenation Signature in Skeletal Muscle That Is Mediated By Late-Life Exercise
- Mitochondria-Targeted Antioxidant SkQ1 Improves Muscle Contractility in Female C26 Tumor-Bearing Mice
Christopher S. Fry
Society for Muscle Biology (US)
5 shared publications
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- A Rejuvenation Signature in Skeletal Muscle That Is Mediated By Late-Life Exercise
Ferdinand von Walden
Karolinska Institutet (SE)
5 shared publications
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Exercise-Induced MYC as an Epigenetic Reprogramming Factor That Combats Skeletal Muscle Aging
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- Insight on the loading-mediated regulation of Runx1 in skeletal muscle
John J. McCarthy
University of Kentucky (US)
4 shared publications
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus
- A Rejuvenation Signature in Skeletal Muscle That Is Mediated By Late-Life Exercise
Cory M. Dungan
Baylor University (US)
3 shared publications
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- A Rejuvenation Signature in Skeletal Muscle That Is Mediated By Late-Life Exercise
- Myonuclear loss, rather than senescent myonuclei, associates with fiber type-specific atrophy in aging human skeletal muscle
Pieter J Koopmans
University of Arkansas at Fayetteville
3 shared publications
In database
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Insight on the loading-mediated regulation of Runx1 in skeletal muscle
- Mitochondria-Targeted Antioxidant SkQ1 Improves Muscle Contractility in Female C26 Tumor-Bearing Mice
Sebastian Edman
Karolinska Institutet (SE)
3 shared publications
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
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