S
Stavroula Tsitkanou Data-verified
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Post Doctoral Research Fellow
postdoc
10 h-index
28 pubs
498 cited
Research Areas
Links
Biography and Research Information
OverviewAI-generated summary
Stavroula Tsitkanou's research focuses on understanding muscle wasting conditions, particularly cancer cachexia and muscular atrophy. Her work investigates the mechanisms underlying muscle weakness and loss, exploring the role of mitochondrial stress and dysfunction. Tsitkanou has examined how factors like biological sex and specific genetic pathways influence the progression of these conditions. Her publications detail the effects of interventions, such as exercise and targeted antioxidant therapies, on muscle function and contractility in preclinical models. She also studies the molecular underpinnings of neurodegenerative diseases, including the impact of microRNA suppression on functional decline in mouse models of TDP-43 proteinopathy. Tsitkanou has collaborated with researchers at the University of Arkansas at Fayetteville, including Tyrone A. Washington, Eleanor R. Schrems, Francielly Morena da Silva, and Ana Regina Cabrera, co-authoring numerous publications with them.
Metrics
- h-index: 10
- Publications: 28
- Citations: 498
Selected Publications
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Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females (2025)
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Mitochondrial-targeted plastoquinone therapy prevents early onset muscle weakness that occurs before atrophy during ovarian cancer (2025)
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Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice (2025)
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Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice (2025)
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Mitochondrial-targeted plastoquinone therapy ameliorates early onset muscle weakness that precedes ovarian cancer cachexia in mice (2024)
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Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice (2024)
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Exercise training induces mild skeletal muscle adaptations without altering disease progression in a TDP-43 mouse model (2024)
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Proteomic analysis of the TDP-43-associated insoluble fraction from NEFH-TDP-43 mouse brain suggests sustained stress granule formation, CLUH granule recruitment and impaired mitochondrial metabolism (2024)
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Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy (2023)
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Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia (2023)
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Mitochondria-Targeted Antioxidant SkQ1 Improves Muscle Contractility in Female C26 Tumor-Bearing Mice (2023)
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Vastus Lateralis and Vastus Intermedius as Predictors of Quadriceps Femoris Muscle Hypertrophy after Strength Training (2022)
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Exercise Counteracts the Deleterious Effects of Cancer Cachexia (2022)
Collaboration Network
Top Collaborators
Nicholas P. Greene
17 shared publications
In database
- Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress
- Exercise Counteracts the Deleterious Effects of Cancer Cachexia
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy
Showing 5 of 17 shared publications
Tyrone A. Washington
University of Arkansas at Fayetteville
14 shared publications
In database
- Exercise Counteracts the Deleterious Effects of Cancer Cachexia
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
Showing 5 of 14 shared publications
Kevin A. Murach
University of Arkansas at Fayetteville
13 shared publications
In database
- Exercise Counteracts the Deleterious Effects of Cancer Cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
Showing 5 of 13 shared publications
Francielly Morena da Silva
University of Arkansas at Fayetteville
13 shared publications
In database
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
Showing 5 of 13 shared publications
Ana Regina Cabrera
University of Arkansas at Fayetteville
12 shared publications
In database
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
Showing 5 of 12 shared publications
Eleanor R. Schrems
University of Arkansas at Fayetteville
11 shared publications
In database
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Supplemental table 5
Showing 5 of 11 shared publications
Megan E. Rosa‐Caldwell
University of Arkansas at Fayetteville
9 shared publications
In database
- Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy
- Supplemental table 5
- Supplemental table 4
Showing 5 of 9 shared publications
Sabin Khadgi
University of Arkansas at Fayetteville
5 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Mitochondria-Targeted Antioxidant SkQ1 Improves Muscle Contractility in Female C26 Tumor-Bearing Mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Seongkyun Lim
University of Arkansas at Fayetteville
5 shared publications
In database
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Mitochondria-Targeted Antioxidant SkQ1 Improves Muscle Contractility in Female C26 Tumor-Bearing Mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Luca J. Delfinis
York University (CA)
4 shared publications
- Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Mitochondrial-targeted plastoquinone therapy prevents early onset muscle weakness that occurs before atrophy during ovarian cancer
- Mitochondrial-targeted plastoquinone therapy ameliorates early onset muscle weakness that precedes ovarian cancer cachexia in mice
Ruqaiza Muhyudin
University of Arkansas at Fayetteville
4 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Shivam Gandhi
York University (CA)
3 shared publications
- Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress
- Mitochondrial-targeted plastoquinone therapy prevents early onset muscle weakness that occurs before atrophy during ovarian cancer
- Mitochondrial-targeted plastoquinone therapy ameliorates early onset muscle weakness that precedes ovarian cancer cachexia in mice
Madison C. Garibotti
York University (CA)
3 shared publications
- Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress
- Mitochondrial-targeted plastoquinone therapy prevents early onset muscle weakness that occurs before atrophy during ovarian cancer
- Mitochondrial-targeted plastoquinone therapy ameliorates early onset muscle weakness that precedes ovarian cancer cachexia in mice
Arthur J. Cheng
York University (CA)
3 shared publications
- Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress
- Mitochondrial-targeted plastoquinone therapy prevents early onset muscle weakness that occurs before atrophy during ovarian cancer
- Mitochondrial-targeted plastoquinone therapy ameliorates early onset muscle weakness that precedes ovarian cancer cachexia in mice
Christopher G. R. Perry
York University (CA)
3 shared publications
- Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress
- Mitochondrial-targeted plastoquinone therapy prevents early onset muscle weakness that occurs before atrophy during ovarian cancer
- Mitochondrial-targeted plastoquinone therapy ameliorates early onset muscle weakness that precedes ovarian cancer cachexia in mice
Similar Researchers
Based on overlapping research topics
Megan E. Rosa‐Caldwell
University of Arkansas at Fayetteville
Muscle Physiology and Disorders
Mitochondrial Function and Pathology
Cancer-related molecular mechanisms research
Eleanor R. Schrems
University of Arkansas at Fayetteville
Muscle Physiology and Disorders
Mitochondrial Function and Pathology
Cancer-related molecular mechanisms research
Sabin Khadgi
University of Arkansas at Fayetteville
Muscle Physiology and Disorders
Mitochondrial Function and Pathology
Cancer-related molecular mechanisms research
Landen W. Saling
University of Arkansas at Fayetteville
Muscle Physiology and Disorders
Mitochondrial Function and Pathology
Cancer-related molecular mechanisms research
Ruqaiza Muhyudin
University of Arkansas at Fayetteville
Muscle Physiology and Disorders
Mitochondrial Function and Pathology
Cancer-related molecular mechanisms research
Michelle Gray
University of Arkansas at Fayetteville
Disease Models, Animal
Mitochondrial Function and Pathology
Neurological disorders and treatments