Azemat Jamshidi‐Parsian
Researcher
University of Arkansas for Medical Sciences
faculty
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Biography and Research Information
OverviewAI-generated summary
Azemat Jamshidi‐Parsian's research focuses on understanding and developing therapeutic strategies for various diseases, with a significant emphasis on cancer and neurological conditions. This work involves investigating the molecular underpinnings of disease progression and exploring novel drug delivery systems. Jamshidi‐Parsian leads a research group at the University of Arkansas for Medical Sciences, collaborating with colleagues including Robert J. Griffin and Samir V. Jenkins, with whom they have co-authored numerous publications.
Their recent publications highlight investigations into photothermal responses for neural stem cell differentiation, characterization of gliomas using nanopore sequencing, and the role of exosomal microRNAs in hepatocellular carcinoma. Further research includes liposome formulations for tumor-targeted drug delivery, particularly in conjunction with radiation therapy, and the proteomic basis of modulating post-ischemic fibrosis. Additionally, their work has explored how pancreatic tumor-derived extracellular vesicles influence schwann cell phenotypes and the induction of reversible dormancy in lung tumor cells.
Jamshidi‐Parsian has received federal funding, including a $348,182 grant from the NIH/National Cancer Institute as Principal Investigator to study the improvement of cellular immunotherapy during dysbiosis. With a career h-index of 17 and over 1,400 citations across 56 publications, their research contributes to advancing biomedical science and therapeutic development.
Metrics
- h-index: 17
- Publications: 56
- Citations: 1,476
Selected Publications
- Gene Expression Profile of Gm20594 and Hapln1 in Mouse Cardiac Muscle Exposed to Microgravity and Space-like Chronic Low-Dose Radiation (2025) DOI
- Musculoskeletal miRNA profiling after exposure to simulated space stressors in mice (2025) DOI
- Pancreatic cancer extracellular vesicles stimulate Schwann cell activation and perineural invasion in vitro via IL-8/CCL2 (2025) DOI
- Abstract B026: Pancreatic tumor extracellular vesicles induce perineural invasion in vitro via IL- 8/CCL2 (2024) DOI
- Pancreatic Cancer Extracellular Vesicles Stimulate Schwann Cell Activation and Perineural Invasion in Vitro Via Il-8/Ccl2 (2024) DOI
- Abstract 186: Tumor exosome-macrophage interactions: The role of tumor-associated glycans and scavenger receptor class A (2024) DOI
- Morphological Effects and In Vitro Biological Mechanisms of Radiation-Induced Cell Killing by Gold Nanomaterials (2023) DOI
- Exosomal MicroRNA and Protein Profiles of Hepatitis B Virus-Related Hepatocellular Carcinoma Cells (2023) DOI
- Exploiting nanopore sequencing for characterization and grading of <i>IDH</i>‐mutant gliomas (2023) DOI
- Pancreatic Tumor-Derived Extracellular Vesicles Stimulate Schwann Cell Phenotype Indicative of Perineural Invasion via IL-8 Signaling (2023) DOI
- Proteomic analysis of transcription factors involved in the alteration of ischemic mouse heart as modulated by MSC exosomes (2023) DOI
- Proteomic Analysis of Transcription Factors Involved in the Alteration of Ischemic Mouse Heart as Modulated by MSC Exosomes (2023) DOI
- Liposome Formulation for Tumor-Targeted Drug Delivery Using Radiation Therapy (2022) DOI
- Liposome Formulation for Tumor-Targeted Drug Delivery Using Radiation Therapy (2022) DOI
- Proteomic basis of modulation of postischemic fibrosis by MSC exosomes (2021) DOI
Federal Grants 1 $348,182 total
Improvement of cellular immunotherapy during dysbiosis- Resubmission
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