Bart Barlogie

High Impact

Researcher

Last publication 2025 Last refreshed 2026-05-16

faculty

138 h-index 1154 pubs 76,293 cited

Biography and Research Information

OverviewAI-generated summary

Bart Barlogie's research focuses on the molecular and temporal evolution of multiple myeloma, investigating the pathways from precursor conditions to advanced disease states. His work delves into the genetic and molecular characteristics that drive myeloma progression, including the identification of gene signatures that predict the risk of monoclonal gammopathy of undetermined significance (MGUS) progressing to multiple myeloma. Barlogie has also examined the spatial architecture of myeloma and its microenvironment at the single-cell level, contributing to a deeper understanding of disease heterogeneity.

Further research areas include the development of prognostic scoring systems, such as the individualized and weighted Myeloma Prognostic Score System (MPSS), to improve patient risk stratification. His investigations extend to the biological mechanisms underlying myeloma, including the role of specific genes like CST6 in suppressing osteolytic bone disease. Barlogie also contributes to research on the feasibility and risk factors associated with outpatient stem cell transplantation for multiple myeloma.

With a distinguished publication record, including 1154 total publications and over 76,000 citations, Barlogie is recognized as a highly cited researcher. He maintains collaborations with colleagues at the University of Arkansas for Medical Sciences, including Maurizio Zangari, Sharmilan Thanendrarajan, John D. Shaughnessy, and Carolina Schinke, with whom he has co-authored numerous publications.

Metrics

  • h-index: 138
  • Publications: 1154
  • Citations: 76,293

Selected Publications

  • Long-Term Follow-Up of Patients With Multiple Myeloma Treated on Earlier Total Therapy Protocols (2025)
    4 citations DOI OpenAlex
  • Long-term follow-up of Total Therapy IV: a phase 3 clinical trial for standard-risk multiple myeloma (2024)
    5 citations DOI OpenAlex
  • Second primary malignancies after tandem autologous hematopoietic stem cell transplantation for multiple myeloma (2024)
    2 citations DOI OpenAlex
  • Development and validation of an individualized and weighted Myeloma Prognostic Score System (<scp>MPSS</scp>) in patients with newly diagnosed multiple myeloma (2024)
    17 citations DOI OpenAlex
  • Three years of maintenance with VRD in multiple myeloma: results of total therapy IIIB with a 15-year follow-up (2023)
    8 citations DOI OpenAlex
  • Impact of Dexamethasone (Dex) Dose Strength on Outcomes in Newly Diagnosed Multiple Myeloma (NDMM): A Secondary Analysis of SWOG Studies S0777 and S1211 (2023)
    2 citations DOI OpenAlex
  • Sequential Imaging with Diffusion-Weighted Whole-Body MRI (DW-MRI) and PET-CT Identifies Patients at High Risk of Relapse in Multiple Myeloma (2023)
    3 citations DOI OpenAlex
  • Resolving the spatial architecture of myeloma and its microenvironment at the single-cell level (2023)
    30 citations DOI OpenAlex
  • A gene signature can predict risk of MGUS progressing to multiple myeloma (2023)
    13 citations DOI OpenAlex
  • Curability of multiple myeloma (MM) based on relative survival rate (RSR) in patients (pts) treated on earlier total therapy (TT) protocols. (2023)
    1 citation DOI OpenAlex
  • The spatio-temporal evolution of multiple myeloma from baseline to relapse-refractory states (2022)
    65 citations DOI OpenAlex
  • CST6 suppresses osteolytic bone disease in multiple myeloma by blocking osteoclast differentiation (2022)
    23 citations DOI OpenAlex
  • Feasibility of Outpatient Stem Cell Transplantation in Multiple Myeloma and Risk Factors Predictive of Hospital Admission (2022)
    13 citations DOI OpenAlex
  • N-Cadherin Stabilizes β-Catenin and Promotes β-Catenin/TCF Transcriptional Activation and Cell Adhesion-Mediated Drug Resistance in Multiple Myeloma (2021)
    2 citations DOI OpenAlex
  • High‐risk transcriptional profiles in multiple myeloma are an acquired feature that can occur in any subtype and more frequently with each subsequent relapse (2021)
    9 citations DOI OpenAlex

View all publications on OpenAlex →

Collaboration Network

147 Collaborators 46 Institutions 6 Countries

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